Findings of Research Misconduct, 42087-42088 [2023-13847]
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Dated: June 12, 2023.
Julia G. Gorey,
Executive Director, SACHRP, Office for
Human Research Protections.
[FR Doc. 2023–13833 Filed 6–28–23; 8:45 am]
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DEPARTMENT OF HEALTH AND
HUMAN SERVICES
Office of the Secretary
Findings of Research Misconduct
Office of the Secretary, HHS.
Notice.
AGENCY:
ACTION:
Findings of research
misconduct have been made against
Yiorgos (Georgios) I. Laliotis, M.D.
(Respondent), who was a Postdoctoral
Fellow, Department of Cancer Biology
and Genetics, College of Medicine, The
Ohio State University (OSU), and
Postdoctoral Fellow, Department of
Oncology, Johns Hopkins University
(JHU). Respondent engaged in research
misconduct in research supported by
U.S. Public Health Service (PHS) funds,
specifically National Cancer Institute
(NCI), National Institutes of Health
(NIH), grants R01 CA186729, R01
CA198117, P30 CA016058, K22
CA245487, and R21 CA252530 and
included in grant applications
submitted for PHS funds, specifically
R01 CA186729–07 and R01 CA198117–
05 submitted to NCI, NIH. The
administrative actions, including
supervision for a period of three (3)
years, were implemented beginning on
June 12, 2023, and are detailed below.
FOR FURTHER INFORMATION CONTACT:
Sheila Garrity, JD, MPH, MBA, Director,
Office of Research Integrity, 1101
Wootton Parkway, Suite 240, Rockville,
MD 20852, (240) 453–8200.
SUPPLEMENTARY INFORMATION: Notice is
hereby given that the Office of Research
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SUMMARY:
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Integrity (ORI) has taken final action in
the following case:
Yiorgos (Georgios) I. Laliotis, M.D.,
The Ohio State University and Johns
Hopkins University: Based on the
reports of inquiries conducted by OSU
and JHU, admissions by Respondent,
and analysis conducted by ORI in its
oversight review, ORI found that
Yiorgos (Georgios) I. Laliotis, M.D.,
former Postdoctoral Fellow, Department
of Cancer Biology and Genetics, College
of Medicine, OSU, and former
Postdoctoral Fellow, Department of
Oncology, JHU, engaged in research
misconduct in research supported by
U.S. Public Health Service (PHS) funds,
specifically NCI, NIH, grants R01
CA186729, R01 CA198117, P30
CA016058, K22 CA245487, and R21
CA252530 and included in grant
applications submitted for PHS funds,
specifically R01 CA186729–07 and R01
CA198117–05 submitted to NCI, NIH.
ORI found that Respondent engaged
in research misconduct by intentionally
and knowingly falsifying and/or
fabricating data, methods, results, and
conclusions by representing a fabricated
Exon 2 splice variant of U2AF2, which
would translate as a Serine-ArginineRich deficient U2AF65 isoform, leading
to the repression of lung
adenocarcinomas and by enhancing the
role of splicing in mutant PIK3CA breast
cancer cell lines in the following three
(3) published papers, two (2) NIH grant
applications, and two (2) unpublished
manuscripts:
• AKT3-mediated IWS1
phosphorylation promotes the
proliferation of EGFR-mutant lung
adenocarcinomas through cell cycleregulated U2AF2 RNA splicing. Nat.
Commun. 2021 Jul 30; 12(1):4624. doi:
10.1038/s41467–021–24795–1 (hereafter
referred to as ‘‘Nat. Commun. 2021’’).
Retraction in: Nat. Commun. 2022 Jun
28;13(1):3711. doi: 10.1038/s41467–
022–31445–7.
• Phosphor-IWS1-dependent U2AF2
splicing regulates trafficking of CAR–Epositive intronless gene mRNAs and
sensitivity to viral infection. Commun.
Biol. 2021 Oct 11; 4(1):1179. doi:
10.1038/s42003–021–02668-z (hereafter
referred to as ‘‘Commun. Biol. 2021’’).
Retraction in: Commun. Biol. 2021 Dec
15;4(1):1419. doi: 10.1038/s42003–021–
02941–1.
• Overexpression of the SETD2 WW
domain inhibits the phosphor-IWS1/
SETD2 interaction and the oncogenic
AKT/IWS1 RNA splicing program.
bioRxiv 2021.08.12.454141.
doi: 10.1101/2021.08.12.454141
(hereafter referred to as ‘‘bioRxiv 2021’’).
Withdrawn. The manuscript also was
submitted to Commun. Biol. in 2021 but
PO 00000
Frm 00047
Fmt 4703
Sfmt 4703
42087
was withdrawn prior to completion of
peer review.
• R01 CA186729–07, ‘‘The role of
IWS1-dependent alternative RNA
splicing in lung cancer,’’ submitted to
NCI, NIH, on November 5, 2020.
• R01 CA198117–05, ‘‘The role of
IWS1 in development and
tumorigenesis,’’ submitted to NCI, NIH,
on June 3, 2019.
• The transcriptomic landscape of
oncogenic P13K reveals key functions in
splicing and gene expression regulation.
Manuscript submitted to Cancer Res.
(hereafter referred to as the ‘‘Cancer Res.
manuscript’’).
• Interpretable deep learning for
chromatin-informed inference of
transcriptional programs driven by
somatic alterations across cancers.
Manuscript in preparation (hereafter
referred to as ‘‘Manuscript 2021’’).
Specifically, ORI finds that
Respondent knowingly and
intentionally:
• falsified the sequencing data in Figure
1g of Nat. Commun. 2021 by splicing
two sequencing chromatograms
together to falsely represent a novel
identification of a previously
undescribed U2AF2 RNA transcript
lacking Exon 2
• falsified conclusions about the
fabricated U2AF2 splice variant in
RT–PCR results in Figures 1f, 2a, 2b,
2c, 3d, 4a, 4b, 4c, 4e, 5h, 6f, 6i, and
7c of Nat. Commun. 2021
• falsified conclusions about the
fabricated U2AF2 splice variant as the
source of two endogenous protein
isoforms in immunoblot panels in
Figures 5c and 5g of Nat. Commun.
2021 and Figure 2 of R01 CA186729–
07
• falsified the experimental conditions
of p-ERK1/2 (Y202/T204), p-CDK1
(Y15), CDK1, and Cyclin B1
immunoblot panels in Figure 5g of
Nat. Commun. 2021 and Figure 2 of
R01 CA186729–07 by using shControl
or shIWS1 instead of the samples as
reported in the figure labels to falsely
represent the immunoblots as the
result of U2AF2 containing spliced
Exon 2
• falsified the experimental conditions
of the a-actinin immunoblot panel in
Figure 1e of Commun. Biol. 2021 by
using shIWS1 instead of shISWS1/
U2AF65b-V5 as reported in the figure
label
• in Commun. Biol. 2021, bioRxiv 2021,
R01 CA186729–07, and R01
CA198117–05, reported falsified
conclusions highlighting the role of
the fabricated U2AF2 RNA transcript
lacking Exon 2 from Nat. Commun.
2021
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42088
Federal Register / Vol. 88, No. 124 / Thursday, June 29, 2023 / Notices
• fabricated and/or falsified the dose
response curves in Figures 3k and
S5N of the Cancer Res. manuscript by
treating the MCF7 and T47D cells
lines with DMSO or Alpelisib instead
of treating with the presence or
absence of splicing inhibitors H3B–
8800 or E7070 as reported in the
figure legend
• fabricated and/or falsified the
quantitative RNA
immunoprecipitation qPCR data in
Figures S4c and S4d of the Cancer
Res. Manuscript
• fabricated and/or falsified the qPCR
data in Figure 6 of Manuscript 2021
to show changes in gene expression
between control and inhibitor
treatment
• fabricated and/or falsified the
experimental methods described in
the legend of Figure 6 of Manuscript
2021 by using CREB1 as a control
gene instead of ACTIN as reported in
the figure legend
Respondent entered into a Voluntary
Settlement Agreement (Agreement) and
voluntarily agreed to the following:
(1) Respondent will have his research
supervised for a period of three (3) years
beginning on June 12, 2023 (the
‘‘Supervision Period’’). Prior to the
submission of an application for PHS
support for a research project on which
Respondent’s participation is proposed
and prior to Respondent’s participation
in any capacity in PHS-supported
research, Respondent will submit a plan
for supervision of Respondent’s duties
to ORI for approval. The supervision
plan must be designed to ensure the
integrity of Respondent’s research.
Respondent will not participate in any
PHS-supported research until such a
supervision plan is approved by ORI.
Respondent will comply with the
agreed-upon supervision plan.
(2) The requirements for Respondent’s
supervision plan are as follows:
i. A committee of 2–3 senior faculty
members at the institution who are
familiar with Respondent’s field of
research, but not including
Respondent’s supervisor or
collaborators, will provide oversight and
guidance for a period of three (3) years
from the effective date of the
Agreement. The committee will review
primary data from Respondent’s
laboratory on a quarterly basis and
submit a report to ORI at six (6)-month
intervals setting forth the committee
meeting dates and Respondent’s
compliance with appropriate research
standards and confirming the integrity
of Respondent’s research.
ii. The committee will conduct an
advance review of each application for
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17:21 Jun 28, 2023
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PHS funds, or report, manuscript, or
abstract involving PHS-supported
research in which Respondent is
involved. The review will include a
discussion with Respondent of the
primary data represented in those
documents and will include a
certification to ORI that the data
presented in the proposed application,
report, manuscript, or abstract are
supported by the research record.
(3) During the Supervision Period,
Respondent will ensure that any
institution employing him submits, in
conjunction with each application for
PHS funds, or report, manuscript, or
abstract involving PHS-supported
research in which Respondent is
involved, a certification to ORI that the
data provided by Respondent are based
on actual experiments or are otherwise
legitimately derived and that the data,
procedures, and methodology are
accurately reported and not plagiarized
in the application, report, manuscript,
or abstract.
(4) If no supervision plan is provided
to ORI, Respondent will provide
certification to ORI at the conclusion of
the Supervision Period that his
participation was not proposed on a
research project for which an
application for PHS support was
submitted and that he has not
participated in any capacity in PHSsupported research.
(5) During the Supervision Period,
Respondent will exclude himself
voluntarily from serving in any advisory
or consultant capacity to PHS including,
but not limited to, service on any PHS
advisory committee, board, and/or peer
review committee.
Dated: June 26, 2023.
Sheila Garrity,
Director, Office of Research Integrity, Office
of the Assistant Secretary for Health.
[FR Doc. 2023–13847 Filed 6–28–23; 8:45 am]
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DEPARTMENT OF HEALTH AND
HUMAN SERVICES
Office of Global Affairs: Stakeholder
Listening Session in Preparation for
the G20 Health Working Group
Ministers Meeting
Notice of public listening
session; request for comments.
ACTION:
Time and Date: The listening session
will be held on Wednesday, August 9,
2023, from 12 to 2:00 p.m., Eastern
Daylight Time.
Place: The session will be held
virtually, with online and dial-in
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Status: This meeting is open to the
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hhs.gov by August 4, 2023. See RSVP
section below for details.
SUPPLEMENTARY INFORMATION:
Purpose: The U.S. Department of
Health and Human Services (HHS), with
support from relevant health-related
U.S. Government offices, is charged
with leading the U.S. delegation to the
Group of 20 (G20) Health Working
Group Ministers’ Meeting and will
convene an informal Stakeholder
Listening Session.
The Stakeholder Listening Session is
designed to seek input from
stakeholders and subject matter experts
to help inform and prepare for U.S.
government engagement with the G20
Health Ministers. The G20 comprises 19
countries (Argentina, Australia, Brazil,
Canada, China, France, Germany, India,
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Korea, Mexico, Russia, Saudi Arabia,
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and United States) and the European
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strengthening global architecture and
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Each year, a different member country
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hosts the meetings. The presidency
proposes the group’s priorities for the
year and hosts discussions to work
towards consensus positions and
actions on those priorities. This year’s
G20 presidency is India, which will be
hosting the Health Working Group
Ministers’ Meeting on August 18 and 19,
2023.
Matters to be Discussed: The
Stakeholder Listening Session will
cover priority areas expected to be
addressed at the G20 Health Working
Group Ministers Meeting. The following
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Priority I: Health emergencies’
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Priority II: Strengthening cooperation
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Priority III: Digital health innovations
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Agencies
[Federal Register Volume 88, Number 124 (Thursday, June 29, 2023)]
[Notices]
[Pages 42087-42088]
From the Federal Register Online via the Government Publishing Office [www.gpo.gov]
[FR Doc No: 2023-13847]
-----------------------------------------------------------------------
DEPARTMENT OF HEALTH AND HUMAN SERVICES
Office of the Secretary
Findings of Research Misconduct
AGENCY: Office of the Secretary, HHS.
ACTION: Notice.
-----------------------------------------------------------------------
SUMMARY: Findings of research misconduct have been made against Yiorgos
(Georgios) I. Laliotis, M.D. (Respondent), who was a Postdoctoral
Fellow, Department of Cancer Biology and Genetics, College of Medicine,
The Ohio State University (OSU), and Postdoctoral Fellow, Department of
Oncology, Johns Hopkins University (JHU). Respondent engaged in
research misconduct in research supported by U.S. Public Health Service
(PHS) funds, specifically National Cancer Institute (NCI), National
Institutes of Health (NIH), grants R01 CA186729, R01 CA198117, P30
CA016058, K22 CA245487, and R21 CA252530 and included in grant
applications submitted for PHS funds, specifically R01 CA186729-07 and
R01 CA198117-05 submitted to NCI, NIH. The administrative actions,
including supervision for a period of three (3) years, were implemented
beginning on June 12, 2023, and are detailed below.
FOR FURTHER INFORMATION CONTACT: Sheila Garrity, JD, MPH, MBA,
Director, Office of Research Integrity, 1101 Wootton Parkway, Suite
240, Rockville, MD 20852, (240) 453-8200.
SUPPLEMENTARY INFORMATION: Notice is hereby given that the Office of
Research Integrity (ORI) has taken final action in the following case:
Yiorgos (Georgios) I. Laliotis, M.D., The Ohio State University and
Johns Hopkins University: Based on the reports of inquiries conducted
by OSU and JHU, admissions by Respondent, and analysis conducted by ORI
in its oversight review, ORI found that Yiorgos (Georgios) I. Laliotis,
M.D., former Postdoctoral Fellow, Department of Cancer Biology and
Genetics, College of Medicine, OSU, and former Postdoctoral Fellow,
Department of Oncology, JHU, engaged in research misconduct in research
supported by U.S. Public Health Service (PHS) funds, specifically NCI,
NIH, grants R01 CA186729, R01 CA198117, P30 CA016058, K22 CA245487, and
R21 CA252530 and included in grant applications submitted for PHS
funds, specifically R01 CA186729-07 and R01 CA198117-05 submitted to
NCI, NIH.
ORI found that Respondent engaged in research misconduct by
intentionally and knowingly falsifying and/or fabricating data,
methods, results, and conclusions by representing a fabricated Exon 2
splice variant of U2AF2, which would translate as a Serine-Arginine-
Rich deficient U2AF65 isoform, leading to the repression of lung
adenocarcinomas and by enhancing the role of splicing in mutant PIK3CA
breast cancer cell lines in the following three (3) published papers,
two (2) NIH grant applications, and two (2) unpublished manuscripts:
AKT3-mediated IWS1 phosphorylation promotes the
proliferation of EGFR-mutant lung adenocarcinomas through cell cycle-
regulated U2AF2 RNA splicing. Nat. Commun. 2021 Jul 30; 12(1):4624.
doi: 10.1038/s41467-021-24795-1 (hereafter referred to as ``Nat.
Commun. 2021''). Retraction in: Nat. Commun. 2022 Jun 28;13(1):3711.
doi: 10.1038/s41467-022-31445-7.
Phosphor-IWS1-dependent U2AF2 splicing regulates
trafficking of CAR-E-positive intronless gene mRNAs and sensitivity to
viral infection. Commun. Biol. 2021 Oct 11; 4(1):1179. doi: 10.1038/
s42003-021-02668-z (hereafter referred to as ``Commun. Biol. 2021'').
Retraction in: Commun. Biol. 2021 Dec 15;4(1):1419. doi: 10.1038/
s42003-021-02941-1.
Overexpression of the SETD2 WW domain inhibits the
phosphor-IWS1/SETD2 interaction and the oncogenic AKT/IWS1 RNA splicing
program. bioRxiv 2021.08.12.454141.
doi: 10.1101/2021.08.12.454141 (hereafter referred to as ``bioRxiv
2021''). Withdrawn. The manuscript also was submitted to Commun. Biol.
in 2021 but was withdrawn prior to completion of peer review.
R01 CA186729-07, ``The role of IWS1-dependent alternative
RNA splicing in lung cancer,'' submitted to NCI, NIH, on November 5,
2020.
R01 CA198117-05, ``The role of IWS1 in development and
tumorigenesis,'' submitted to NCI, NIH, on June 3, 2019.
The transcriptomic landscape of oncogenic P13K reveals key
functions in splicing and gene expression regulation. Manuscript
submitted to Cancer Res. (hereafter referred to as the ``Cancer Res.
manuscript'').
Interpretable deep learning for chromatin-informed
inference of transcriptional programs driven by somatic alterations
across cancers. Manuscript in preparation (hereafter referred to as
``Manuscript 2021'').
Specifically, ORI finds that Respondent knowingly and
intentionally:
falsified the sequencing data in Figure 1g of Nat. Commun.
2021 by splicing two sequencing chromatograms together to falsely
represent a novel identification of a previously undescribed U2AF2 RNA
transcript lacking Exon 2
falsified conclusions about the fabricated U2AF2 splice
variant in RT-PCR results in Figures 1f, 2a, 2b, 2c, 3d, 4a, 4b, 4c,
4e, 5h, 6f, 6i, and 7c of Nat. Commun. 2021
falsified conclusions about the fabricated U2AF2 splice
variant as the source of two endogenous protein isoforms in immunoblot
panels in Figures 5c and 5g of Nat. Commun. 2021 and Figure 2 of R01
CA186729-07
falsified the experimental conditions of p-ERK1/2 (Y202/T204),
p-CDK1 (Y15), CDK1, and Cyclin B1 immunoblot panels in Figure 5g of
Nat. Commun. 2021 and Figure 2 of R01 CA186729-07 by using shControl or
shIWS1 instead of the samples as reported in the figure labels to
falsely represent the immunoblots as the result of U2AF2 containing
spliced Exon 2
falsified the experimental conditions of the [alpha]-actinin
immunoblot panel in Figure 1e of Commun. Biol. 2021 by using shIWS1
instead of shISWS1/U2AF65[beta]-V5 as reported in the figure label
in Commun. Biol. 2021, bioRxiv 2021, R01 CA186729-07, and R01
CA198117-05, reported falsified conclusions highlighting the role of
the fabricated U2AF2 RNA transcript lacking Exon 2 from Nat. Commun.
2021
[[Page 42088]]
fabricated and/or falsified the dose response curves in
Figures 3k and S5N of the Cancer Res. manuscript by treating the MCF7
and T47D cells lines with DMSO or Alpelisib instead of treating with
the presence or absence of splicing inhibitors H3B-8800 or E7070 as
reported in the figure legend
fabricated and/or falsified the quantitative RNA
immunoprecipitation qPCR data in Figures S4c and S4d of the Cancer Res.
Manuscript
fabricated and/or falsified the qPCR data in Figure 6 of
Manuscript 2021 to show changes in gene expression between control and
inhibitor treatment
fabricated and/or falsified the experimental methods described
in the legend of Figure 6 of Manuscript 2021 by using CREB1 as a
control gene instead of ACTIN as reported in the figure legend
Respondent entered into a Voluntary Settlement Agreement
(Agreement) and voluntarily agreed to the following:
(1) Respondent will have his research supervised for a period of
three (3) years beginning on June 12, 2023 (the ``Supervision
Period''). Prior to the submission of an application for PHS support
for a research project on which Respondent's participation is proposed
and prior to Respondent's participation in any capacity in PHS-
supported research, Respondent will submit a plan for supervision of
Respondent's duties to ORI for approval. The supervision plan must be
designed to ensure the integrity of Respondent's research. Respondent
will not participate in any PHS-supported research until such a
supervision plan is approved by ORI. Respondent will comply with the
agreed-upon supervision plan.
(2) The requirements for Respondent's supervision plan are as
follows:
i. A committee of 2-3 senior faculty members at the institution who
are familiar with Respondent's field of research, but not including
Respondent's supervisor or collaborators, will provide oversight and
guidance for a period of three (3) years from the effective date of the
Agreement. The committee will review primary data from Respondent's
laboratory on a quarterly basis and submit a report to ORI at six (6)-
month intervals setting forth the committee meeting dates and
Respondent's compliance with appropriate research standards and
confirming the integrity of Respondent's research.
ii. The committee will conduct an advance review of each
application for PHS funds, or report, manuscript, or abstract involving
PHS-supported research in which Respondent is involved. The review will
include a discussion with Respondent of the primary data represented in
those documents and will include a certification to ORI that the data
presented in the proposed application, report, manuscript, or abstract
are supported by the research record.
(3) During the Supervision Period, Respondent will ensure that any
institution employing him submits, in conjunction with each application
for PHS funds, or report, manuscript, or abstract involving PHS-
supported research in which Respondent is involved, a certification to
ORI that the data provided by Respondent are based on actual
experiments or are otherwise legitimately derived and that the data,
procedures, and methodology are accurately reported and not plagiarized
in the application, report, manuscript, or abstract.
(4) If no supervision plan is provided to ORI, Respondent will
provide certification to ORI at the conclusion of the Supervision
Period that his participation was not proposed on a research project
for which an application for PHS support was submitted and that he has
not participated in any capacity in PHS-supported research.
(5) During the Supervision Period, Respondent will exclude himself
voluntarily from serving in any advisory or consultant capacity to PHS
including, but not limited to, service on any PHS advisory committee,
board, and/or peer review committee.
Dated: June 26, 2023.
Sheila Garrity,
Director, Office of Research Integrity, Office of the Assistant
Secretary for Health.
[FR Doc. 2023-13847 Filed 6-28-23; 8:45 am]
BILLING CODE 4150-31-P