Social Security Ruling, SSR 17-3p; Titles II and XVI: Evaluating Cases Involving Sickle Cell Disease (SCD), 43442-43445 [2017-19551]
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43442
Federal Register / Vol. 82, No. 178 / Friday, September 15, 2017 / Notices
SOCIAL SECURITY ADMINISTRATION
Social Security—Survivors Insurance;
96.006—Supplemental Security Income.)
[Docket No. SSA–2017–0007]
Nancy A. Berryhill,
Acting Commissioner of Social Security.
Social Security Ruling, SSR 17–3p;
Titles II and XVI: Evaluating Cases
Involving Sickle Cell Disease (SCD)
AGENCY:
ACTION:
Policy Interpretation Ruling
Social Security Administration.
Notice of Social Security Ruling
(SSR).
We are providing notice of
SSR 17–3p. This SSR provides guidance
on SCD and how we evaluate SCD in
disability claims under titles II and XVI
of the Social Security Act.
SUMMARY:
This SSR is applicable on
September 15, 2017.
DATES:
FOR FURTHER INFORMATION CONTACT:
Cheryl A. Williams, Office of Disability
Policy, Social Security Administration,
6401 Security Boulevard, Baltimore,
Maryland 21235–6401, (410) 965–1020.
For information on eligibility or filing
for benefits, call our national toll-free
number, 1–800–772–1213 or TTY 1–
800–325–0778, or visit our Internet site,
Social Security Online, at https://
www.socialsecurity.gov.
Although
5 U.S.C. 552(a)(1) and (a)(2) do not
require us to publish this SSR, we are
doing so in accordance with 20 CFR
402.35(b)(1).
Through SSRs, we make available to
the public precedential decisions
relating to the Federal old-age,
survivors, disability, supplemental
security income, and special veterans’
benefits programs. We may base SSRs
on determinations or decisions made at
all levels of administrative adjudication,
Federal court decisions, Commissioner’s
decisions, opinions of the Office of the
General Counsel, or other
interpretations of the law and
regulations.
Although SSRs do not have the same
force and effect as statutes or
regulations, they are binding on all
components of the Social Security
Administration. 20 CFR 402.35(b)(1).
This SSR will remain in effect until
we publish a notice in the Federal
Register that rescinds it, or until we
publish a new SSR that replaces or
modifies it.
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SUPPLEMENTARY INFORMATION:
(Catalog of Federal Domestic Assistance,
Programs Nos. 96.001, Social Security—
Disability Insurance; 96.002, Social
Security—Retirement Insurance; 96.004,
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Titles II and XVI: Evaluating Cases
Involving Sickle Cell Disease (SCD)
Purpose: This Social Security Ruling
(SSR) provides background information
on SCD and how we evaluate SCD
during our adjudication process. We
provide this guidance to help
adjudicators consistently apply our
policies in disability claims involving
SCD.
Citations: Sections 216(i), 223(d),
223(f), 1614(a)(3) and 1614(a)(4) of the
Social Security Act, as amended;
Regulations No. 4, subpart P, sections
404.1502, 404.1505, 404.1509, 404.1512,
404.1513, 404.1520, 404.1520a,
404.1520b, 404.1521, 404.1522,
404.1523, 404.1525, 404.1526, 404.1529,
404.1545, 404.1560–404.1569a,
404.1593, 404.1594, appendices 1 and 2;
and Regulations No. 16, subpart I,
sections 416.902, 416.905, 416.906,
416.909, 416.911, 416.912, 416.913,
416.920, 416.920a, 416.920b, 416.921,
416.922, 416.923, 416.924, 416.924a,
416.925, 416.926, 416.926a, 416.929,
416.945, 416.960–416.969a, 416.987,
416.993, 416.994, and 416.994a.
Introduction
SCD is the most common inherited
blood disease in the United States,
affecting an estimated 100,000
Americans.1 SCD is not always easy to
evaluate due to its varying nature and
complications. In this SSR, we provide
basic information about SCD and its
variants and clarify that sickle cell trait
is not a variant of SCD. We also provide
guidance for assessing SCD under the
hematological disorder listings and
determining how this impairment may
affect the residual functional capacity
finding for adults and the functional
equivalence finding for children.
Policy Interpretation
We consider all medical evidence
when we evaluate a claim for disability
benefits. The following information is in
a question and answer format that
provides guidance about SCD and how
to consider evidence regarding this
impairment. Questions 1 and 2 provide
basic background information about
SCD and its variants. Question 3
clarifies that sickle cell trait is not a
variant of SCD. Question 4 discusses the
complications and symptoms of SCD.
1 See Centers for Disease Control and Prevention,
‘‘Sickle Cell Disease.’’ (https://www.cdc.gov/
ncbddd/sicklecell/data.html.)
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Questions 5 through 7 explain how
adjudicators should evaluate SCD at
various points of the adjudication
process, including the adult and child
hematological listings we consider.
List of Questions
1. What is SCD?
2. What are the different variants of
SCD?
3. Is sickle cell trait a variant of SCD?
4. What are the common
complications and symptoms of SCD?
5. How do we evaluate the
complications of SCD under the
hematological disorder listings?
6. How do we evaluate the
complications of SCD when assessing
residual functional capacity (RFC) for
adults?
7. How do we evaluate the
complications of SCD under functional
equivalence for children?
Answers
1. What is SCD?
SCD is a type of hemolytic anemia
and an inherited hematological disorder
that affects the hemoglobin within a
person’s red blood cells (RBC).
Hemoglobin is the protein within RBC
that carries oxygen. The abnormal
hemoglobin makes the RBC more prone
to distortion (‘‘sickling’’), which results
in blocked blood vessels and a
shortened RBC lifespan. Hemolytic
anemia results when the abnormal RBC
are destroyed faster than the body can
produce them.
When hemoglobin is normal, a
person’s RBC are round and easily travel
through blood vessels, bringing oxygen
to the body’s organs and tissues. SCD
causes sickle-shaped RBC that are not
flexible and can stick to vessel walls,
causing blockages (vaso-occlusion) that
slow or stop the flow of blood and
oxygen. This blockage may in turn cause
pain. Persons with SCD are predisposed
to pain, infection, and other
complications. Because people inherit
SCD, the disease is present at birth, but
the age when children display
symptoms varies.2
2. What are the different variants of
SCD?
The different variants of SCD may
indicate the severity of complications
and the resulting functional limitations
caused by SCD. Laboratory blood tests
such as hemoglobin electrophoresis
establish the existence and the variants
2 See National Heart, Lung, and Blood Institute,
‘‘What Are the Signs and Symptoms of Sickle Cell
Disease?’’ (https://www.nhlbi.nih.gov/health/
health-topics/topics/sca). Health problems usually
do not appear until an infant is around 5 to 6
months of age.
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Federal Register / Vol. 82, No. 178 / Friday, September 15, 2017 / Notices
of SCD. The following are the most
common variants of SCD: 3
• Hemoglobin (Hb) SS (HbSS)—a
person with this form of SCD inherits
one sickle cell gene from each parent.
HbSS is the most common and usually
most severe form of SCD.
• HbSC—a person inherits one sickle
cell gene from one parent, and another
gene for an abnormal hemoglobin called
‘‘C’’ from the other parent. HbSC is
usually a milder type of SCD.
• Hb S-beta (Sb) thalassemia— a
person inherits one sickle cell gene from
one parent, and a gene for beta
thalassemia from the other parent. There
are two forms of beta thalassemia, sickle
beta zero thalassemia (Hb Sb0
thalassemia) and sickle beta plus
thalassemia (Hb Sb+ thalassemia). Sickle
beta zero thalassemia is usually a more
severe form of SCD. People with sickle
beta plus thalassemia tend to have a
milder form of SCD.
• HbSD, HbSE, and HbSO—people
with these variants of SCD have one
sickle cell gene plus another abnormal
hemoglobin gene, ‘‘D,’’ ‘‘E,’’ or ‘‘O.’’
These are rarer types of SCD with
varying severity.
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3. Is sickle cell trait a variant of SCD?
No. Sickle cell trait is not a variant of
SCD. Sickle cell trait occurs when a
person inherits one sickle hemoglobin
gene from one parent and a normal gene
from the other parent. People with
sickle cell trait rarely have signs and
symptoms associated with SCD and
usually do not need treatment.
However, in rare cases and under
extreme conditions such as intense
exercise, people with sickle cell trait
have a higher risk of severe breakdown
of muscle tissue (exertional
rhabdomyolysis) that can lead to serious
complications.4 In spite of this higher
risk, recent evidence indicates that
sickle cell trait is not associated with an
increased probability of death.5
Sickle cell trait alone is not an
impairment. As defined by the Social
Security Act, an impairment must result
from anatomical, physiological, or
psychological abnormalities that can be
shown by medically acceptable clinical
and laboratory diagnostic techniques.
3 See Centers for Disease Control and Prevention,
‘‘Sickle Cell Disease.’’ (https://www.cdc.gov/
ncbddd/sicklecell/facts.html).
4 Other conditions that could be harmful for
people with sickle cell trait include high altitudes,
dehydration, low oxygen levels in the air, and
increased pressure in the atmosphere. We evaluate
impairments that result from sickle cell trait under
the affected body system.
5 See Nelson D.A., et al. Sickle Cell Trait,
Rhabdomyolysis, and Mortality among U.S. Army
Soldiers. New England Journal of Medicine, Aug;
375(17), 1695–6 (2016).
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To establish an impairment in this
context, we require objective medical
evidence (medical signs and laboratory
findings) from an acceptable medical
source of complications from sickle cell
trait. In addition, a person’s
complications from sickle cell trait must
meet the statutory duration requirement,
i.e., be expected to result in death or last
or be expected to last for a continuous
period of not less than 12 months.
Therefore, we cannot find a person
disabled due to sickle cell trait if there
are no medical signs or laboratory
findings of complications from sickle
cell trait and the complications from
sickle cell trait do not meet the duration
requirement.
4. What are the common complications
and symptoms of SCD?
Complications of SCD may include,
but are not limited to pain crises,
anemia, osteomyelitis, leg ulcers,
pulmonary infections or infarctions,
acute chest syndrome, pulmonary
hypertension, chronic heart failure,
gallbladder disease, liver failure, kidney
failure, nephritic syndrome, aplastic
crisis, stroke, and mental impairments
such as depression. Examples of
symptoms that may stem from these
complications include pain, fatigue,
malaise, shortness of breath, and
difficulty feeding in infants. The
symptoms of SCD vary from person to
person and can change over time.
A. Pain (vaso-occlusive) crisis is a
common complication of SCD. Pain
crises are either acute or chronic. Acute
pain crises occur suddenly when
sickled RBC stop blood flow and reduce
oxygen delivery. This pain can be
intense, stabbing, or throbbing. Pain can
strike almost anywhere in the body and
in more than one spot at a time. The
pain often occurs in the lower back,
legs, arms, abdomen, and chest.6
Chronic pain in SCD is more than a
continuation of acute pain crisis. It
usually occurs when lack of oxygen to
the bone due to vaso-occlusion results
in the death of bone tissue (avascular
necrosis) at various joints such as the
hips, shoulders and ankles.7
B. Anemia is another complication of
SCD. It occurs when sickled RBC die
prematurely, which reduces the amount
of oxygen-carrying hemoglobin in the
blood. Symptoms from anemia can
6 See National Heart, Lung, and Blood Institute,
‘‘What Are the Signs and Symptoms of Sickle Cell
Disease?’’ (https://www.nhlbi.nih.gov/health/healthtopics/topics/sca/signs).
7 See Okpala I, Tawil A. Management of Pain in
Sickle-Cell Disease. Journal of the Royal Society of
Medicine, Sep; 95(9), 456–458, 2002 (available at:
https://www.ncbi.nlm.nih.gov/pmc/articles/
PMC1279994/).
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include fatigue, weakness, shortness of
breath, and dizziness. Chronic
deprivation of oxygen-rich blood can
damage nerves and organs in the body,
including the spleen, brain, eyes, joints,
bones, lungs, liver, heart, kidneys, and
other organs.
C. Pulmonary complications such as
acute chest syndrome (ACS) and
pulmonary hypertension are the leading
cause of death for SCD patients.8 ACS is
a vaso-occlusion of the pulmonary
vessels. Symptoms of ACS include but
are not limited to chest pain, fever,
tachypnea (abnormally rapid breathing),
wheezing, or coughing. Pulmonary
hypertension can occur when sickled
RBC cause pulmonary arteries to
become narrow and blocked. The result
of this damage to the pulmonary arteries
is high blood pressure in the lungs.
Symptoms of pulmonary hypertension
include shortness of breath, fatigue, and
chest pain.9
D. Strokes and silent strokes affect
people with SCD at a higher rate
because sickled RBC clump along the
walls of larger arteries going to the
brain. Strokes can result in full or
partial paralysis on one side of the body,
problems with balance, or difficulty
speaking or understanding. Silent
strokes can occur without outward
symptoms and are only detectable by
brain imaging. However, silent strokes
can impair intellectual ability, attention,
visual-spatial skills, language, and longterm memory.10
E. Bacterial infections are often severe
complications in people with SCD.
Anemia from SCD and vaso-occlusions
can damage the spleen, which
ultimately increases risk of infection
and damages other organs. Infection
frequently leads to hospitalization and
is the primary cause of death in young
children with SCD.11
F. Mental disorders in people with
SCD are often secondary to the impact
of treatment, pain, and other symptoms.
For example, depression from
reoccurring pain is especially common
8 See Gladwin MT, Miller, A. Pulmonary
Complications of Sickle Cell Disease. American
Journal of Respiratory and Critical Care Medicine,
Jun; 185(11), 1154–1165, 2012 (available at: https://
www.ncbi.nlm.nih.gov/pmc/articles/PMC3373067/).
9 See National Institutes of Health. MedlinePlus.
‘‘Pulmonary Hypertension.’’ (https://
medlineplus.gov/pulmonaryhypertension.html).
10 See, The Internet Stroke Center, ‘‘Stroke as a
Complication of Sickle Cell Disease.’’ (https://
www.strokecenter.org/patients/about-stroke/
pediatric-stroke/stroke-as-a-complication-of-sicklecell-disease/).
11 See Booth, C., et al. Infection in Sickle Cell
Disease: A Review. International Journal of
Infectious Diseases, Jan; 14(1), e2–e12, 2010
(available at: https://www.sciencedirect.com/science/
article/pii/S1201971209001453).
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Federal Register / Vol. 82, No. 178 / Friday, September 15, 2017 / Notices
in people with SCD.12 Other mental
disorders that may occur include, but
are not limited to, anxiety and cognitive
disorders from stroke.13
5. How do we evaluate the
complications of SCD under the
hematological disorder listings?
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We may evaluate SCD under the
following hematological disorder
listings:
• Listing 7.05 and 107.05, Hemolytic
anemias; or
• Listing 7.17 and 107.17,
Hematological disorders treated by bone
marrow or stem cell transplantation; or
• Listing 7.18, Repeated
complications of hematological
disorders.
Under listing 7.05 and 107.05, we
assess hemolytic anemias, including
sickle cell disease, thalassemia, and
their variants. We evaluate pain crises
caused by SCD under listings 7.05A and
107.05A. We assess complications of
SCD requiring hospitalizations under
listings 7.05B and 107.05B. Listings
7.05C and 107.05C describes the criteria
we use to evaluate SCD that results in
anemia with low hemoglobin levels.
Under listings 7.17 and 107.17, we
consider people who receive bone
marrow or stem cell transplantation to
treat their SCD, to be disabled for at
least 12 months after the date of
transplant.
We evaluate adults who have repeated
complications from SCD, but do not
have the requisite findings for listing
7.05 or 7.17, under listing 7.18.14 To
meet listing 7.18, SCD must cause
repeated complications, resulting in
significant, documented symptoms or
signs and a ‘‘marked’’ level of limitation
in one of the general areas of
functioning: Activities of daily living,
social functioning, or completing tasks
because of deficiencies in concentration,
persistence, or pace. We use listing 7.18
to evaluate only hematological
disorders.15
12 See Jonassaint CR, Jones VL, Leong S, Frierson
GM. A Systematic Review of the Association
between Depression and Health Care Utilization in
Children and Adults with Sickle Cell Disease.
British Journal of Hematology, Jul; 174(1), 136–47,
2016.
13 Becker M, Axelrod DJ. Hematologic Problems
in Psychosomatic Medicine. Psychiatric Clinics of
North America, Dec; 30(4), 739–759, 2007 (available
at: https://www.sciencedirect.com/science/article/
pii/S0193953X07000767).
14 We evaluate a child’s functioning under the
rules for functional equivalence. See 20 CFR
416.926a.
15 We use listing 7.18 to evaluate hematological
disorders and complications caused by
hematological disorders. We can only evaluate
anemia under 7.18 if it results from an underlying
hematological disorder. If the person’s anemia
results from a condition that is not a hematological
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If a person’s SCD does not meet a
hematological listing, we will compare
the specific findings in each case to any
appropriate hematological listings to
determine whether medical equivalence
may exist. We may also find medical
equivalence if the person has multiple
impairments, including SCD, none of
which meet or medically equal the
requirements of a listing alone, but the
combination of impairments is
medically equivalent in severity to a
listed impairment.
If the person’s SCD does not meet or
equal the criteria in a listing, we will
consider whether he or she has an
impairment that satisfies the criteria in
a listing in another body system. For
example, we may evaluate the effects of
intracranial bleeding or stroke under
11.00 or 12.00.
6. How do we evaluate the
complications of SCD when assessing
residual functional capacity (RFC) for
adults?
For adults, we assess RFC when the
effects of a person’s SCD, either alone or
in combination with another
impairment(s), do not meet or medically
equal a listing. We base the RFC
assessment on all the relevant evidence
in the record, including the effects of
treatment.16 In assessing RFC, we must
consider all of a person’s work-related
limitations, whether due to SCD, other
impairment(s), or a combination of
impairments. For example, adults with
SCD may have pain, fatigue, and
shortness of breath that may affect their
ability to stand and walk. In addition, a
person experiencing repeated acute pain
crises may have difficulty maintaining
concentration to complete tasks and
have frequent absences from work.
7. How do we evaluate the
complications of SCD under functional
equivalence? 17
Children with SCD that does not meet
or medically equal a listing may
nevertheless have an impairment(s) that
functionally equals the listings under
our rules for evaluating disability in
children.18 When we determine whether
disorder, we would evaluate the anemia under the
listing for that impairment.
16 See 20 CFR 404.1545 and 416.945, and SSR 96–
8p.
17 Functional equivalence applies only to claims
for children under title XVI. All claims for title II,
even if the claimant is under age 18, are decided
under the adult rules.
18 See 20 CFR 416.926a, SSR 09–1p, 74 FR 7527
(2009) also available at https://www.ssa.gov/OP_
Home/rulings/ssi/02/SSR2009-01-ssi-02.html, and
SSR 09–2p, 74 FR 7525 (2009) also available at
https://www.ssa.gov/OP_Home/rulings/ssi/02/
SSR2009-02-ssi-02.html. For the complete titles of
all SSRs cited in this footnote and those following,
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a child’s impairment(s) functionally
equal the listings, we use the six
domains of functioning.
When we evaluate a child’s
functioning in these six domains, we
consider how the child functions
compared to children the same age who
do not have impairments. We must
explain any limitation in a child’s
ability to function appropriately for his
or her age based on a medically
determinable impairment(s).19 It is
important to remember that the
cumulative physical effects of SCD and
its treatment can vary in kind and
intensity, affecting each child
differently. The six domains of
functioning are:
Acquiring and using information.
Some children with SCD may have
limitations in acquiring and using
information due to stroke, including
silent stroke.20 A stroke can cause brain
injury that impairs a child’s ability to
learn, concentrate, speak, and
remember.
Attending and completing tasks.
Frequent pain crises can result in
limitations in attending and completing
tasks at school and at home.21 If a child
does not feel well due to pain, it may
be difficult for him or her to stay
focused on activities long enough to
complete them in an age-appropriate
manner. A child with SCD who is
experiencing pain may also have
difficulty paying attention to details and
may make mistakes on schoolwork due
to an inability to concentrate.
Interacting and relating with others.
SCD can also cause limitations
interacting and relating with others.22
The unpredictable nature of pain in SCD
may cause anxiety and difficulty
maintaining relationships. Children
suffering from complications of SCD
may become withdrawn, uncooperative,
or unresponsive.
Moving about and manipulating
objects. If SCD limits a child’s ability to
move and manipulate objects, we
evaluate those effects in the domain of
‘‘Moving about and manipulating
objects.’’ 23 For example, sickling in the
hip bones, knees, and ankles due to SCD
may cause joint pain and problems with
walking, running, and climbing up and
down stairs.
Caring for yourself. Caring for yourself
involves a child’s basic understanding
of his or her body’s normal functioning
see the CROSS-REFERENCES section at the end of
this SSR.
19 See 20 CFR 416.924a(b) and 416.926a.
20 See 20 CFR 416.926a(g) and SSR 09–3p.
21 See 20 CFR 416.926a(h) and SSR 09–4p.
22 See 20 CFR 416.926a(i) and SSR 09–5p.
23 See 20 CFR 416.926a(j) and SSR 09–6p.
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and the adequate emotional health for
carrying out self-care tasks.24 A child
with SCD may avoid taking medication
or ignore complications of the disease
out of frustration with the limitations of
SCD.
Health and physical well-being. The
ongoing effects of SCD and its treatment
may affect a child’s health and physical
well-being.25 In this domain, we
evaluate the effects of periodic
exacerbations of pain crises due to
sickle cell anemia. We consider the
frequency and duration of the
exacerbations as well as the extent to
which they affect a child’s ability to
function physically.
This SSR is applicable on September
15, 2017. 26
Cross References: SSR 86–8: Titles II
and XVI: The Sequential Evaluation
Process; SSR 96–3p: Titles II and XVI:
Considering Allegations of Pain and
Other Symptoms in Determining
Whether a Medically Determinable
Impairment is Severe; SSR 96–8p: Titles
II and XVI: Assessing Residual
Functional Capacity in Initial Claims;
SSR 09–1p: Title XVI: Determining
Childhood Disability Under the
Functional Equivalence Rule—The
‘‘Whole Child’’ Approach; SSR 09–2p:
Title XVI: Determining Childhood
Disability—Documenting a Child’s
Impairment-Related Limitations; SSR
09–3p: Title XVI: Determining
Childhood Disability—The Functional
Equivalence Domain of ‘‘Acquiring and
Using Information’’; SSR 09–4p: Title
XVI: Determining Childhood
Disability—The Functional Equivalence
Domain of ‘‘Attending and Completing
Tasks’’; SSR 09–5p: Title XVI:
Determining Childhood Disability—The
Functional Equivalence Domain of
‘‘Interacting and Relating with Others’’;
SSR 09–6p: Title XVI: Determining
Childhood Disability—The Functional
Equivalence Domain of ‘‘Moving About
and Manipulating Objects’’; SSR 09–7p:
Title XVI: Determining Childhood
Disability—The Functional Equivalence
Domain of ‘‘Caring for Yourself’’; SSR
09–8p: Title XVI: Determining
24 See
20 CFR 416.926a(k) and SSR 09–7p.
20 CFR 416.926a(l) and SSR 09–8p.
26 We will use this SSR beginning on its
applicable date. We will apply this SSR to new
applications filed on or after the applicable date of
the SSR and to claims that are pending on and after
the applicable date. This means that we will use
this ruling on and after its applicable date in any
case in which we make a determination or decision.
We expect that Federal courts will review our final
decisions using the rules that were in effect at the
time we issued the decisions. If a court reverses our
final decision and remands a case for further
administrative proceedings after the applicable date
of this SSR, we will apply this SSR to the entire
period at issue in the decision we make after the
court’s remand.
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25 See
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17:07 Sep 14, 2017
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Childhood Disability—The Functional
Equivalence Domain of ‘‘Health and
Physical Well-Being’’; SSR 16–3p: Titles
II and XVI: Evaluation of Symptoms in
Disability Claims; and Program
Operations Manual System (POMS) DI
22001.001, DI 22505.001, DI 22505.003,
DI 24501.021, DI 24510.005, DI
25201.005, DI 25220.010, DI 25505.025,
and DI 25505.030.
[FR Doc. 2017–19551 Filed 9–14–17; 8:45 am]
BILLING CODE 4191–02–P
DEPARTMENT OF STATE
[Public Notice: 10119]
Cultural Property Advisory Committee;
Notice of Meeting
Department of State.
Notice of a meeting.
AGENCY:
ACTION:
The Department of State is
issuing this notice to announce the
location, date, time and agenda for the
next meeting of the Cultural Property
Advisory Committee.
DATES: October 23 through 25, 2017,
9:00 a.m. to 5:00 p.m. (EDT). The open
session of the Cultural Property
Advisory Committee will be held on
October 23, 2017 at 10:00 a.m. (EDT). It
will last approximately one hour.
Participants will participate
electronically. Those who wish to
participate in the open session should
visit https://culturalheritage.state.gov
where information will be provided on
how to access the meeting no later than
October 16, 2017.
Written Comments: must be received
no later than October 15, 2017, at 11:59
p.m. (EDT).
ADDRESSES: The meeting will be held at
the U.S. Department of State, Annex 5,
2200 C St. NW. and the Harry S Truman
Building, 2201 C St. NW., Washington,
DC.
Comments: Methods of written
comment submission are as follows:
• Electronic Comments: Use https://
www.regulations.gov, enter the docket
[DOS–2017–0036] and follow the
prompts to submit comments.
• Paper Comments: Only send paper
comments if comments contain
privileged or confidential information
(within the meaning of 19 U.S.C.
2605(i)(1)) to: U.S. Department of State,
Bureau of Educational and Cultural
Affairs—Cultural Heritage Center, SA–5
Floor 5, 2200 C St. NW., Washington,
DC 20522–0505.
FOR FURTHER INFORMATION CONTACT: For
general questions concerning the
meeting, contact Catherine Foster,
Bureau of Educational and Cultural
SUMMARY:
PO 00000
Frm 00121
Fmt 4703
Sfmt 4703
43445
Affairs—Cultural Heritage Center by
phone, (202) 632–6301, or mail:
CulProp@state.gov.
Pursuant
to section 306(e)(2) of the Convention
on Cultural Property Implementation
Act (5 U.S.C. 2601 et seq.) (‘‘the Act’’),
the Department is announcing a meeting
of the Cultural Property Advisory
Committee (‘‘the Committee’’). The
Committee’s responsibilities are carried
out in accordance with provisions of the
Act. A portion of this meeting will be
closed to the public pursuant to 5 U.S.C.
552b(c)(9)(B) and 19 U.S.C. 2605.
Meeting Agenda: The Committee will
review a proposal to extend the
Memorandum of Understanding
Between the Government of United
States of America and the Government
of the Kingdom of Cambodia
Concerning the Imposition of Import
Restrictions on Archaeological Material
from Cambodia from the Bronze Age
through the Khmer Era (‘‘the Cambodia
MOU’’).
Open Session Participation: An open
session of the meeting to receive oral
public comments on the proposed
extension of the Cambodia MOU will be
held Monday, October 23, 2017, from
10:00 a.m. to 11:00 a.m. (EDT). The text
of the Act and a copy of the Cambodia
MOU may be found at https://
culturalheritage.state.gov.
If you wish to make an oral
presentation at the meeting, you must
request to be scheduled by October 15,
2017 via email (culprop@state.gov), and
you must submit a written summary of
your oral presentation, ensuring that it
is received no later than 11:59 p.m.
(EDT) on October 15, 2017, via the
Regulations.gov Web site listed in the
‘‘COMMENTS’’ section above. Oral
comments will be limited to five (5)
minutes to allow time for questions
from members of the Committee. All
oral comments must relate specifically
to matters referred to in 19 U.S.C.
2602(a)(1), with respect to which the
Committee makes its findings and
recommendations. Oral presentation to
the Committee may be requested but,
due to time constraints, is not
guaranteed.
Written Comments: If you do not wish
to make oral comments but still wish to
make your views known, you may
submit written comments for the
Committee to consider. Written
comments from outside interested
parties regarding the proposed
extension of the Cambodia MOU must
be received no later than October 15,
2017, at 11:59 p.m. (EDT). Your written
comments should relate specifically to
SUPPLEMENTARY INFORMATION:
E:\FR\FM\15SEN1.SGM
15SEN1
Agencies
[Federal Register Volume 82, Number 178 (Friday, September 15, 2017)]
[Notices]
[Pages 43442-43445]
From the Federal Register Online via the Government Publishing Office [www.gpo.gov]
[FR Doc No: 2017-19551]
[[Page 43442]]
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SOCIAL SECURITY ADMINISTRATION
[Docket No. SSA-2017-0007]
Social Security Ruling, SSR 17-3p; Titles II and XVI: Evaluating
Cases Involving Sickle Cell Disease (SCD)
AGENCY: Social Security Administration.
ACTION: Notice of Social Security Ruling (SSR).
-----------------------------------------------------------------------
SUMMARY: We are providing notice of SSR 17-3p. This SSR provides
guidance on SCD and how we evaluate SCD in disability claims under
titles II and XVI of the Social Security Act.
DATES: This SSR is applicable on September 15, 2017.
FOR FURTHER INFORMATION CONTACT: Cheryl A. Williams, Office of
Disability Policy, Social Security Administration, 6401 Security
Boulevard, Baltimore, Maryland 21235-6401, (410) 965-1020. For
information on eligibility or filing for benefits, call our national
toll-free number, 1-800-772-1213 or TTY 1-800-325-0778, or visit our
Internet site, Social Security Online, at https://www.socialsecurity.gov.
SUPPLEMENTARY INFORMATION: Although 5 U.S.C. 552(a)(1) and (a)(2) do
not require us to publish this SSR, we are doing so in accordance with
20 CFR 402.35(b)(1).
Through SSRs, we make available to the public precedential
decisions relating to the Federal old-age, survivors, disability,
supplemental security income, and special veterans' benefits programs.
We may base SSRs on determinations or decisions made at all levels of
administrative adjudication, Federal court decisions, Commissioner's
decisions, opinions of the Office of the General Counsel, or other
interpretations of the law and regulations.
Although SSRs do not have the same force and effect as statutes or
regulations, they are binding on all components of the Social Security
Administration. 20 CFR 402.35(b)(1).
This SSR will remain in effect until we publish a notice in the
Federal Register that rescinds it, or until we publish a new SSR that
replaces or modifies it.
(Catalog of Federal Domestic Assistance, Programs Nos. 96.001,
Social Security--Disability Insurance; 96.002, Social Security--
Retirement Insurance; 96.004, Social Security--Survivors Insurance;
96.006--Supplemental Security Income.)
Nancy A. Berryhill,
Acting Commissioner of Social Security.
Policy Interpretation Ruling
Titles II and XVI: Evaluating Cases Involving Sickle Cell Disease (SCD)
Purpose: This Social Security Ruling (SSR) provides background
information on SCD and how we evaluate SCD during our adjudication
process. We provide this guidance to help adjudicators consistently
apply our policies in disability claims involving SCD.
Citations: Sections 216(i), 223(d), 223(f), 1614(a)(3) and
1614(a)(4) of the Social Security Act, as amended; Regulations No. 4,
subpart P, sections 404.1502, 404.1505, 404.1509, 404.1512, 404.1513,
404.1520, 404.1520a, 404.1520b, 404.1521, 404.1522, 404.1523, 404.1525,
404.1526, 404.1529, 404.1545, 404.1560-404.1569a, 404.1593, 404.1594,
appendices 1 and 2; and Regulations No. 16, subpart I, sections
416.902, 416.905, 416.906, 416.909, 416.911, 416.912, 416.913, 416.920,
416.920a, 416.920b, 416.921, 416.922, 416.923, 416.924, 416.924a,
416.925, 416.926, 416.926a, 416.929, 416.945, 416.960-416.969a,
416.987, 416.993, 416.994, and 416.994a.
Introduction
SCD is the most common inherited blood disease in the United
States, affecting an estimated 100,000 Americans.\1\ SCD is not always
easy to evaluate due to its varying nature and complications. In this
SSR, we provide basic information about SCD and its variants and
clarify that sickle cell trait is not a variant of SCD. We also provide
guidance for assessing SCD under the hematological disorder listings
and determining how this impairment may affect the residual functional
capacity finding for adults and the functional equivalence finding for
children.
---------------------------------------------------------------------------
\1\ See Centers for Disease Control and Prevention, ``Sickle
Cell Disease.'' (https://www.cdc.gov/ncbddd/sicklecell/data.html.)
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Policy Interpretation
We consider all medical evidence when we evaluate a claim for
disability benefits. The following information is in a question and
answer format that provides guidance about SCD and how to consider
evidence regarding this impairment. Questions 1 and 2 provide basic
background information about SCD and its variants. Question 3 clarifies
that sickle cell trait is not a variant of SCD. Question 4 discusses
the complications and symptoms of SCD. Questions 5 through 7 explain
how adjudicators should evaluate SCD at various points of the
adjudication process, including the adult and child hematological
listings we consider.
List of Questions
1. What is SCD?
2. What are the different variants of SCD?
3. Is sickle cell trait a variant of SCD?
4. What are the common complications and symptoms of SCD?
5. How do we evaluate the complications of SCD under the
hematological disorder listings?
6. How do we evaluate the complications of SCD when assessing
residual functional capacity (RFC) for adults?
7. How do we evaluate the complications of SCD under functional
equivalence for children?
Answers
1. What is SCD?
SCD is a type of hemolytic anemia and an inherited hematological
disorder that affects the hemoglobin within a person's red blood cells
(RBC). Hemoglobin is the protein within RBC that carries oxygen. The
abnormal hemoglobin makes the RBC more prone to distortion
(``sickling''), which results in blocked blood vessels and a shortened
RBC lifespan. Hemolytic anemia results when the abnormal RBC are
destroyed faster than the body can produce them.
When hemoglobin is normal, a person's RBC are round and easily
travel through blood vessels, bringing oxygen to the body's organs and
tissues. SCD causes sickle-shaped RBC that are not flexible and can
stick to vessel walls, causing blockages (vaso-occlusion) that slow or
stop the flow of blood and oxygen. This blockage may in turn cause
pain. Persons with SCD are predisposed to pain, infection, and other
complications. Because people inherit SCD, the disease is present at
birth, but the age when children display symptoms varies.\2\
---------------------------------------------------------------------------
\2\ See National Heart, Lung, and Blood Institute, ``What Are
the Signs and Symptoms of Sickle Cell Disease?'' (https://www.nhlbi.nih.gov/health/health-topics/topics/sca). Health problems
usually do not appear until an infant is around 5 to 6 months of
age.
---------------------------------------------------------------------------
2. What are the different variants of SCD?
The different variants of SCD may indicate the severity of
complications and the resulting functional limitations caused by SCD.
Laboratory blood tests such as hemoglobin electrophoresis establish the
existence and the variants
[[Page 43443]]
of SCD. The following are the most common variants of SCD: \3\
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\3\ See Centers for Disease Control and Prevention, ``Sickle
Cell Disease.'' (https://www.cdc.gov/ncbddd/sicklecell/facts.html).
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Hemoglobin (Hb) SS (HbSS)--a person with this form of SCD
inherits one sickle cell gene from each parent. HbSS is the most common
and usually most severe form of SCD.
HbSC--a person inherits one sickle cell gene from one
parent, and another gene for an abnormal hemoglobin called ``C'' from
the other parent. HbSC is usually a milder type of SCD.
Hb S-beta (S[beta]) thalassemia-- a person inherits one
sickle cell gene from one parent, and a gene for beta thalassemia from
the other parent. There are two forms of beta thalassemia, sickle beta
zero thalassemia (Hb S[beta]\0\ thalassemia) and sickle beta plus
thalassemia (Hb S[beta]\+\ thalassemia). Sickle beta zero thalassemia
is usually a more severe form of SCD. People with sickle beta plus
thalassemia tend to have a milder form of SCD.
HbSD, HbSE, and HbSO--people with these variants of SCD
have one sickle cell gene plus another abnormal hemoglobin gene, ``D,''
``E,'' or ``O.'' These are rarer types of SCD with varying severity.
3. Is sickle cell trait a variant of SCD?
No. Sickle cell trait is not a variant of SCD. Sickle cell trait
occurs when a person inherits one sickle hemoglobin gene from one
parent and a normal gene from the other parent. People with sickle cell
trait rarely have signs and symptoms associated with SCD and usually do
not need treatment. However, in rare cases and under extreme conditions
such as intense exercise, people with sickle cell trait have a higher
risk of severe breakdown of muscle tissue (exertional rhabdomyolysis)
that can lead to serious complications.\4\ In spite of this higher
risk, recent evidence indicates that sickle cell trait is not
associated with an increased probability of death.\5\
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\4\ Other conditions that could be harmful for people with
sickle cell trait include high altitudes, dehydration, low oxygen
levels in the air, and increased pressure in the atmosphere. We
evaluate impairments that result from sickle cell trait under the
affected body system.
\5\ See Nelson D.A., et al. Sickle Cell Trait, Rhabdomyolysis,
and Mortality among U.S. Army Soldiers. New England Journal of
Medicine, Aug; 375(17), 1695-6 (2016).
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Sickle cell trait alone is not an impairment. As defined by the
Social Security Act, an impairment must result from anatomical,
physiological, or psychological abnormalities that can be shown by
medically acceptable clinical and laboratory diagnostic techniques. To
establish an impairment in this context, we require objective medical
evidence (medical signs and laboratory findings) from an acceptable
medical source of complications from sickle cell trait. In addition, a
person's complications from sickle cell trait must meet the statutory
duration requirement, i.e., be expected to result in death or last or
be expected to last for a continuous period of not less than 12 months.
Therefore, we cannot find a person disabled due to sickle cell trait if
there are no medical signs or laboratory findings of complications from
sickle cell trait and the complications from sickle cell trait do not
meet the duration requirement.
4. What are the common complications and symptoms of SCD?
Complications of SCD may include, but are not limited to pain
crises, anemia, osteomyelitis, leg ulcers, pulmonary infections or
infarctions, acute chest syndrome, pulmonary hypertension, chronic
heart failure, gallbladder disease, liver failure, kidney failure,
nephritic syndrome, aplastic crisis, stroke, and mental impairments
such as depression. Examples of symptoms that may stem from these
complications include pain, fatigue, malaise, shortness of breath, and
difficulty feeding in infants. The symptoms of SCD vary from person to
person and can change over time.
A. Pain (vaso-occlusive) crisis is a common complication of SCD.
Pain crises are either acute or chronic. Acute pain crises occur
suddenly when sickled RBC stop blood flow and reduce oxygen delivery.
This pain can be intense, stabbing, or throbbing. Pain can strike
almost anywhere in the body and in more than one spot at a time. The
pain often occurs in the lower back, legs, arms, abdomen, and chest.\6\
Chronic pain in SCD is more than a continuation of acute pain crisis.
It usually occurs when lack of oxygen to the bone due to vaso-occlusion
results in the death of bone tissue (avascular necrosis) at various
joints such as the hips, shoulders and ankles.\7\
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\6\ See National Heart, Lung, and Blood Institute, ``What Are
the Signs and Symptoms of Sickle Cell Disease?'' (https://www.nhlbi.nih.gov/health/health-topics/topics/sca/signs).
\7\ See Okpala I, Tawil A. Management of Pain in Sickle-Cell
Disease. Journal of the Royal Society of Medicine, Sep; 95(9), 456-
458, 2002 (available at: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1279994/).
---------------------------------------------------------------------------
B. Anemia is another complication of SCD. It occurs when sickled
RBC die prematurely, which reduces the amount of oxygen-carrying
hemoglobin in the blood. Symptoms from anemia can include fatigue,
weakness, shortness of breath, and dizziness. Chronic deprivation of
oxygen-rich blood can damage nerves and organs in the body, including
the spleen, brain, eyes, joints, bones, lungs, liver, heart, kidneys,
and other organs.
C. Pulmonary complications such as acute chest syndrome (ACS) and
pulmonary hypertension are the leading cause of death for SCD
patients.\8\ ACS is a vaso-occlusion of the pulmonary vessels. Symptoms
of ACS include but are not limited to chest pain, fever, tachypnea
(abnormally rapid breathing), wheezing, or coughing. Pulmonary
hypertension can occur when sickled RBC cause pulmonary arteries to
become narrow and blocked. The result of this damage to the pulmonary
arteries is high blood pressure in the lungs. Symptoms of pulmonary
hypertension include shortness of breath, fatigue, and chest pain.\9\
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\8\ See Gladwin MT, Miller, A. Pulmonary Complications of Sickle
Cell Disease. American Journal of Respiratory and Critical Care
Medicine, Jun; 185(11), 1154-1165, 2012 (available at: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3373067/).
\9\ See National Institutes of Health. MedlinePlus. ``Pulmonary
Hypertension.'' (https://medlineplus.gov/pulmonaryhypertension.html).
---------------------------------------------------------------------------
D. Strokes and silent strokes affect people with SCD at a higher
rate because sickled RBC clump along the walls of larger arteries going
to the brain. Strokes can result in full or partial paralysis on one
side of the body, problems with balance, or difficulty speaking or
understanding. Silent strokes can occur without outward symptoms and
are only detectable by brain imaging. However, silent strokes can
impair intellectual ability, attention, visual-spatial skills,
language, and long-term memory.\10\
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\10\ See, The Internet Stroke Center, ``Stroke as a Complication
of Sickle Cell Disease.'' (https://www.strokecenter.org/patients/about-stroke/pediatric-stroke/stroke-as-a-complication-of-sickle-cell-disease/).
---------------------------------------------------------------------------
E. Bacterial infections are often severe complications in people
with SCD. Anemia from SCD and vaso-occlusions can damage the spleen,
which ultimately increases risk of infection and damages other organs.
Infection frequently leads to hospitalization and is the primary cause
of death in young children with SCD.\11\
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\11\ See Booth, C., et al. Infection in Sickle Cell Disease: A
Review. International Journal of Infectious Diseases, Jan; 14(1),
e2-e12, 2010 (available at: https://www.sciencedirect.com/science/article/pii/S1201971209001453).
---------------------------------------------------------------------------
F. Mental disorders in people with SCD are often secondary to the
impact of treatment, pain, and other symptoms. For example, depression
from reoccurring pain is especially common
[[Page 43444]]
in people with SCD.\12\ Other mental disorders that may occur include,
but are not limited to, anxiety and cognitive disorders from
stroke.\13\
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\12\ See Jonassaint CR, Jones VL, Leong S, Frierson GM. A
Systematic Review of the Association between Depression and Health
Care Utilization in Children and Adults with Sickle Cell Disease.
British Journal of Hematology, Jul; 174(1), 136-47, 2016.
\13\ Becker M, Axelrod DJ. Hematologic Problems in Psychosomatic
Medicine. Psychiatric Clinics of North America, Dec; 30(4), 739-759,
2007 (available at: https://www.sciencedirect.com/science/article/pii/S0193953X07000767).
---------------------------------------------------------------------------
5. How do we evaluate the complications of SCD under the hematological
disorder listings?
We may evaluate SCD under the following hematological disorder
listings:
Listing 7.05 and 107.05, Hemolytic anemias; or
Listing 7.17 and 107.17, Hematological disorders treated
by bone marrow or stem cell transplantation; or
Listing 7.18, Repeated complications of hematological
disorders.
Under listing 7.05 and 107.05, we assess hemolytic anemias,
including sickle cell disease, thalassemia, and their variants. We
evaluate pain crises caused by SCD under listings 7.05A and 107.05A. We
assess complications of SCD requiring hospitalizations under listings
7.05B and 107.05B. Listings 7.05C and 107.05C describes the criteria we
use to evaluate SCD that results in anemia with low hemoglobin levels.
Under listings 7.17 and 107.17, we consider people who receive bone
marrow or stem cell transplantation to treat their SCD, to be disabled
for at least 12 months after the date of transplant.
We evaluate adults who have repeated complications from SCD, but do
not have the requisite findings for listing 7.05 or 7.17, under listing
7.18.\14\ To meet listing 7.18, SCD must cause repeated complications,
resulting in significant, documented symptoms or signs and a ``marked''
level of limitation in one of the general areas of functioning:
Activities of daily living, social functioning, or completing tasks
because of deficiencies in concentration, persistence, or pace. We use
listing 7.18 to evaluate only hematological disorders.\15\
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\14\ We evaluate a child's functioning under the rules for
functional equivalence. See 20 CFR 416.926a.
\15\ We use listing 7.18 to evaluate hematological disorders and
complications caused by hematological disorders. We can only
evaluate anemia under 7.18 if it results from an underlying
hematological disorder. If the person's anemia results from a
condition that is not a hematological disorder, we would evaluate
the anemia under the listing for that impairment.
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If a person's SCD does not meet a hematological listing, we will
compare the specific findings in each case to any appropriate
hematological listings to determine whether medical equivalence may
exist. We may also find medical equivalence if the person has multiple
impairments, including SCD, none of which meet or medically equal the
requirements of a listing alone, but the combination of impairments is
medically equivalent in severity to a listed impairment.
If the person's SCD does not meet or equal the criteria in a
listing, we will consider whether he or she has an impairment that
satisfies the criteria in a listing in another body system. For
example, we may evaluate the effects of intracranial bleeding or stroke
under 11.00 or 12.00.
6. How do we evaluate the complications of SCD when assessing residual
functional capacity (RFC) for adults?
For adults, we assess RFC when the effects of a person's SCD,
either alone or in combination with another impairment(s), do not meet
or medically equal a listing. We base the RFC assessment on all the
relevant evidence in the record, including the effects of
treatment.\16\ In assessing RFC, we must consider all of a person's
work-related limitations, whether due to SCD, other impairment(s), or a
combination of impairments. For example, adults with SCD may have pain,
fatigue, and shortness of breath that may affect their ability to stand
and walk. In addition, a person experiencing repeated acute pain crises
may have difficulty maintaining concentration to complete tasks and
have frequent absences from work.
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\16\ See 20 CFR 404.1545 and 416.945, and SSR 96-8p.
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7. How do we evaluate the complications of SCD under functional
equivalence? \17\
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\17\ Functional equivalence applies only to claims for children
under title XVI. All claims for title II, even if the claimant is
under age 18, are decided under the adult rules.
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Children with SCD that does not meet or medically equal a listing
may nevertheless have an impairment(s) that functionally equals the
listings under our rules for evaluating disability in children.\18\
When we determine whether a child's impairment(s) functionally equal
the listings, we use the six domains of functioning.
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\18\ See 20 CFR 416.926a, SSR 09-1p, 74 FR 7527 (2009) also
available at https://www.ssa.gov/OP_Home/rulings/ssi/02/SSR2009-01-ssi-02.html, and SSR 09-2p, 74 FR 7525 (2009) also available at
https://www.ssa.gov/OP_Home/rulings/ssi/02/SSR2009-02-ssi-02.html.
For the complete titles of all SSRs cited in this footnote and those
following, see the CROSS-REFERENCES section at the end of this SSR.
---------------------------------------------------------------------------
When we evaluate a child's functioning in these six domains, we
consider how the child functions compared to children the same age who
do not have impairments. We must explain any limitation in a child's
ability to function appropriately for his or her age based on a
medically determinable impairment(s).\19\ It is important to remember
that the cumulative physical effects of SCD and its treatment can vary
in kind and intensity, affecting each child differently. The six
domains of functioning are:
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\19\ See 20 CFR 416.924a(b) and 416.926a.
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Acquiring and using information. Some children with SCD may have
limitations in acquiring and using information due to stroke, including
silent stroke.\20\ A stroke can cause brain injury that impairs a
child's ability to learn, concentrate, speak, and remember.
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\20\ See 20 CFR 416.926a(g) and SSR 09-3p.
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Attending and completing tasks. Frequent pain crises can result in
limitations in attending and completing tasks at school and at
home.\21\ If a child does not feel well due to pain, it may be
difficult for him or her to stay focused on activities long enough to
complete them in an age-appropriate manner. A child with SCD who is
experiencing pain may also have difficulty paying attention to details
and may make mistakes on schoolwork due to an inability to concentrate.
---------------------------------------------------------------------------
\21\ See 20 CFR 416.926a(h) and SSR 09-4p.
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Interacting and relating with others. SCD can also cause
limitations interacting and relating with others.\22\ The unpredictable
nature of pain in SCD may cause anxiety and difficulty maintaining
relationships. Children suffering from complications of SCD may become
withdrawn, uncooperative, or unresponsive.
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\22\ See 20 CFR 416.926a(i) and SSR 09-5p.
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Moving about and manipulating objects. If SCD limits a child's
ability to move and manipulate objects, we evaluate those effects in
the domain of ``Moving about and manipulating objects.'' \23\ For
example, sickling in the hip bones, knees, and ankles due to SCD may
cause joint pain and problems with walking, running, and climbing up
and down stairs.
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\23\ See 20 CFR 416.926a(j) and SSR 09-6p.
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Caring for yourself. Caring for yourself involves a child's basic
understanding of his or her body's normal functioning
[[Page 43445]]
and the adequate emotional health for carrying out self-care tasks.\24\
A child with SCD may avoid taking medication or ignore complications of
the disease out of frustration with the limitations of SCD.
---------------------------------------------------------------------------
\24\ See 20 CFR 416.926a(k) and SSR 09-7p.
---------------------------------------------------------------------------
Health and physical well-being. The ongoing effects of SCD and its
treatment may affect a child's health and physical well-being.\25\ In
this domain, we evaluate the effects of periodic exacerbations of pain
crises due to sickle cell anemia. We consider the frequency and
duration of the exacerbations as well as the extent to which they
affect a child's ability to function physically.
---------------------------------------------------------------------------
\25\ See 20 CFR 416.926a(l) and SSR 09-8p.
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This SSR is applicable on September 15, 2017. \26\
---------------------------------------------------------------------------
\26\ We will use this SSR beginning on its applicable date. We
will apply this SSR to new applications filed on or after the
applicable date of the SSR and to claims that are pending on and
after the applicable date. This means that we will use this ruling
on and after its applicable date in any case in which we make a
determination or decision. We expect that Federal courts will review
our final decisions using the rules that were in effect at the time
we issued the decisions. If a court reverses our final decision and
remands a case for further administrative proceedings after the
applicable date of this SSR, we will apply this SSR to the entire
period at issue in the decision we make after the court's remand.
---------------------------------------------------------------------------
Cross References: SSR 86-8: Titles II and XVI: The Sequential
Evaluation Process; SSR 96-3p: Titles II and XVI: Considering
Allegations of Pain and Other Symptoms in Determining Whether a
Medically Determinable Impairment is Severe; SSR 96-8p: Titles II and
XVI: Assessing Residual Functional Capacity in Initial Claims; SSR 09-
1p: Title XVI: Determining Childhood Disability Under the Functional
Equivalence Rule--The ``Whole Child'' Approach; SSR 09-2p: Title XVI:
Determining Childhood Disability--Documenting a Child's Impairment-
Related Limitations; SSR 09-3p: Title XVI: Determining Childhood
Disability--The Functional Equivalence Domain of ``Acquiring and Using
Information''; SSR 09-4p: Title XVI: Determining Childhood Disability--
The Functional Equivalence Domain of ``Attending and Completing
Tasks''; SSR 09-5p: Title XVI: Determining Childhood Disability--The
Functional Equivalence Domain of ``Interacting and Relating with
Others''; SSR 09-6p: Title XVI: Determining Childhood Disability--The
Functional Equivalence Domain of ``Moving About and Manipulating
Objects''; SSR 09-7p: Title XVI: Determining Childhood Disability--The
Functional Equivalence Domain of ``Caring for Yourself''; SSR 09-8p:
Title XVI: Determining Childhood Disability--The Functional Equivalence
Domain of ``Health and Physical Well-Being''; SSR 16-3p: Titles II and
XVI: Evaluation of Symptoms in Disability Claims; and Program
Operations Manual System (POMS) DI 22001.001, DI 22505.001, DI
22505.003, DI 24501.021, DI 24510.005, DI 25201.005, DI 25220.010, DI
25505.025, and DI 25505.030.
[FR Doc. 2017-19551 Filed 9-14-17; 8:45 am]
BILLING CODE 4191-02-P