Ipconazole; Pesticide Tolerances, 15235-15240 [2014-06059]
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Federal Register / Vol. 79, No. 53 / Wednesday, March 19, 2014 / Rules and Regulations
Accordingly, this action merely
approves a State plan as meeting
Federal requirements and does not
impose additional requirements beyond
those imposed by State law. For that
reason, this action:
• Is not a ‘‘significant regulatory
action’’ subject to review by the Office
of Management and Budget under
Executive Order 12866 (58 FR 51735,
October 4, 1993);
• does not impose an information
collection burden under the provisions
of the Paperwork Reduction Act (44
U.S.C. 3501 et seq.);
• is certified as not having a
significant economic impact on a
substantial number of small entities
under the Regulatory Flexibility Act (5
U.S.C. 601 et seq.);
• does not contain any unfunded
mandate or significantly or uniquely
affect small governments, as described
in the Unfunded Mandates Reform Act
of 1995 (Pub. L. 104–4);
• does not have Federalism
implications as specified in Executive
Order 13132 (64 FR 43255, August 10,
1999);
• is not an economically significant
regulatory action based on health or
safety risks subject to Executive Order
13045 (62 FR 19885, April 23, 1997);
• is not a significant regulatory action
subject to Executive Order 13211 (66 FR
28355, May 22, 2001);
• is not subject to requirements of
Section 12(d) of the National
Technology Transfer and Advancement
Act of 1995 (15 U.S.C. 272 note) because
application of those requirements would
be inconsistent with the Clean Air Act;
and
• does not provide EPA with the
discretionary authority to address
disproportionate human health or
environmental effects with practical,
appropriate, and legally permissible
methods under Executive Order 12898
(59 FR 7629, February 16, 1994).
In addition, this rule does not have
tribal implications as specified by
Executive Order 13175 (65 FR 67249,
November 9, 2000), because the SIP is
not approved to apply in Indian country
located in the State, and EPA notes that
it will not impose substantial direct
costs on tribal governments or preempt
tribal law.
The Congressional Review Act, 5
U.S.C. 801 et seq., as added by the Small
Business Regulatory Enforcement
Fairness Act of 1996, generally provides
that before a rule may take effect, the
agency promulgating the rule must
submit a rule report, which includes a
copy of the rule, to each House of the
Congress and to the Comptroller General
of the United States. EPA will submit a
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report containing this action and other
required information to the U.S. Senate,
the U.S. House of Representatives, and
the Comptroller General of the United
States prior to publication of the rule in
the Federal Register. A major rule
cannot take effect until 60 days after it
is published in the Federal Register.
This action is not a ‘‘major rule’’ as
defined by 5 U.S.C. 804(2).
Under section 307(b)(1) of the Clean
Air Act, petitions for judicial review of
this action must be filed in the United
States Court of Appeals for the
appropriate circuit by May 19, 2014.
Filing a petition for reconsideration by
the Administrator of this final rule does
not affect the finality of this action for
the purposes of judicial review nor does
it extend the time within which a
petition for judicial review may be filed,
and shall not postpone the effectiveness
of such rule or action. Parties with
objections to this direct final rule are
encouraged to file a comment in
response to the parallel notice of
proposed rulemaking for this action
published in the Proposed Rules section
of today’s Federal Register, rather than
file an immediate petition for judicial
review of this direct final rule, so that
EPA can withdraw this direct final rule
and address the comment in the
proposed rulemaking. This action may
not be challenged later in proceedings to
enforce its requirements (see section
307(b)(2)).
List of Subjects in 40 CFR Part 52
Environmental protection, Air
pollution control, Incorporation by
reference, Intergovernmental relations,
Particulate matter, Reporting and
recordkeeping requirements.
Dated: March 5, 2014.
Jared Blumenfeld,
Regional Administrator, Region IX.
Part 52, Chapter I, Title 40 of the Code
of Federal Regulations is amended as
follows:
PART 52 [AMENDED]
1. The authority citation for Part 52
continues to read as follows:
■
Authority: 42 U.S.C. 7401 et seq.
Subpart D—Arizona
2. Section 52.120 is amended by
adding paragraph (c)(159) to read as
follows:
■
§ 52.120
*
Identification of plan.
*
*
(c) * * *
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*
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15235
(159) The following plan was
submitted on January 23, 2012 by the
Governor’s Designee.
(i) [Reserved]
(ii) Additional Materials.
(A) Arizona Department of
Environmental Quality
(1) Final Update of the Limited
Maintenance Plan for the Payson PM10
Maintenance Area (December 2011),
adopted by the Arizona Department of
Environmental Quality on January 23,
2012.
[FR Doc. 2014–05669 Filed 3–18–14; 8:45 am]
BILLING CODE 6560–50–P
ENVIRONMENTAL PROTECTION
AGENCY
40 CFR Part 180
[EPA–HQ–OPP–2012–0796; FRL–9907–25]
Ipconazole; Pesticide Tolerances
Environmental Protection
Agency (EPA).
ACTION: Final rule.
AGENCY:
This regulation establishes
tolerances for residues of ipconazole in
or on vegetable, legume, group 6.
Chemtura Corporation requested these
tolerances under the Federal Food,
Drug, and Cosmetic Act (FFDCA).
DATES: This regulation is effective
March 19, 2014. Objections and requests
for hearings must be received on or
before May 19, 2014, and must be filed
in accordance with the instructions
provided in 40 CFR part 178 (see also
Unit I.C. of the SUPPLEMENTARY
INFORMATION).
SUMMARY:
The docket for this action,
identified by docket identification (ID)
number EPA–HQ–OPP–2012–0796, is
available at https://www.regulations.gov
or at the Office of Pesticide Programs
Regulatory Public Docket (OPP Docket)
in the Environmental Protection Agency
Docket Center (EPA/DC), EPA West
Bldg., Rm. 3334, 1301 Constitution Ave.
NW., Washington, DC 20460–0001. The
Public Reading Room is open from 8:30
a.m. to 4:30 p.m., Monday through
Friday, excluding legal holidays. The
telephone number for the Public
Reading Room is (202) 566–1744, and
the telephone number for the OPP
Docket is (703) 305–5805. Please review
the visitor instructions and additional
information about the docket available
at https://www.epa.gov/dockets.
FOR FURTHER INFORMATION CONTACT: Lois
Rossi, Registration Division (7505P),
Office of Pesticide Programs,
Environmental Protection Agency, 1200
Pennsylvania Ave. NW., Washington,
ADDRESSES:
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Federal Register / Vol. 79, No. 53 / Wednesday, March 19, 2014 / Rules and Regulations
DC 20460–0001; telephone number:
(703) 305–7090; email address:
RDFRNotices@epa.gov.
SUPPLEMENTARY INFORMATION:
I. General Information
A. Does this action apply to me?
You may be potentially affected by
this action if you are an agricultural
producer, food manufacturer, or
pesticide manufacturer. The following
list of North American Industrial
Classification System (NAICS) codes is
not intended to be exhaustive, but rather
provides a guide to help readers
determine whether this document
applies to them. Potentially affected
entities may include:
• Crop production (NAICS code 111).
• Animal production (NAICS code
112).
• Food manufacturing (NAICS code
311).
• Pesticide manufacturing (NAICS
code 32532).
B. How can I get electronic access to
other related information?
You may access a frequently updated
electronic version of EPA’s tolerance
regulations at 40 CFR part 180 through
the Government Printing Office’s e-CFR
site at https://www.ecfr.gov/cgi-bin/textidx?&c=ecfr&tpl=/ecfrbrowse/Title40/
40tab_02.tpl.
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C. How can I file an objection or hearing
request?
Under FFDCA section 408(g), 21
U.S.C. 346a, any person may file an
objection to any aspect of this regulation
and may also request a hearing on those
objections. You must file your objection
or request a hearing on this regulation
in accordance with the instructions
provided in 40 CFR part 178. To ensure
proper receipt by EPA, you must
identify docket ID number EPA–HQ–
OPP–2012–0796 in the subject line on
the first page of your submission. All
objections and requests for a hearing
must be in writing, and must be
received by the Hearing Clerk on or
before May 19, 2014. Addresses for mail
and hand delivery of objections and
hearing requests are provided in 40 CFR
178.25(b).
In addition to filing an objection or
hearing request with the Hearing Clerk
as described in 40 CFR part 178, please
submit a copy of the filing (excluding
any Confidential Business Information
(CBI)) for inclusion in the public docket.
Information not marked confidential
pursuant to 40 CFR part 2 may be
disclosed publicly by EPA without prior
notice. Submit the non-CBI copy of your
objection or hearing request, identified
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by docket ID number EPA–HQ–OPP–
2012–0796, by one of the following
methods:
• Federal eRulemaking Portal: https://
www.regulations.gov. Follow the online
instructions for submitting comments.
Do not submit electronically any
information you consider to be CBI or
other information whose disclosure is
restricted by statute.
• Mail: OPP Docket, Environmental
Protection Agency Docket Center (EPA/
DC), (28221T), 1200 Pennsylvania Ave.
NW., Washington, DC 20460–0001.
• Hand Delivery: To make special
arrangements for hand delivery or
delivery of boxed information, please
follow the instructions at https://
www.epa.gov/dockets/contacts.html.
Additional instructions on commenting
or visiting the docket, along with more
information about dockets generally, is
available at https://www.epa.gov/
dockets.
II. Summary of Petitioned-for Tolerance
In the Federal Register of December
19, 2012 (77 FR 75082) (FRL–9372–6),
EPA issued a document pursuant to
FFDCA section 408(d)(3), 21 U.S.C.
346a(d)(3), announcing the filing of a
pesticide petition (PP 2F8076) by
Chemtura Corporation, 199 Benson Rd.,
Middlebury, CT 06749. The petition
requested that 40 CFR 180.646 be
amended by establishing tolerances for
residues of the fungicide ipconazole (2[(4-chlorophenyl)methyl]-5-(1methylethyl)-1-(1H-1,2,4-triazol-1ylmethyl)cyclopentanol) in or on
legume vegetables, succulent or dried,
crop group 6 at 0.01 parts per million
(ppm). That document referenced a
summary of the petition prepared by
Chemtura Corporation, the registrant,
which is available in the docket,
https://www.regulations.gov. There were
no comments received in response to
the notice of filing.
III. Aggregate Risk Assessment and
Determination of Safety
Section 408(b)(2)(A)(i) of FFDCA
allows EPA to establish a tolerance (the
legal limit for a pesticide chemical
residue in or on a food) only if EPA
determines that the tolerance is ‘‘safe.’’
Section 408(b)(2)(A)(ii) of FFDCA
defines ‘‘safe’’ to mean that ‘‘there is a
reasonable certainty that no harm will
result from aggregate exposure to the
pesticide chemical residue, including
all anticipated dietary exposures and all
other exposures for which there is
reliable information.’’ This includes
exposure through drinking water and in
residential settings, but does not include
occupational exposure. Section
408(b)(2)(C) of FFDCA requires EPA to
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give special consideration to exposure
of infants and children to the pesticide
chemical residue in establishing a
tolerance and to ‘‘ensure that there is a
reasonable certainty that no harm will
result to infants and children from
aggregate exposure to the pesticide
chemical residue. . . .’’
Consistent with FFDCA section
408(b)(2)(D), and the factors specified in
FFDCA section 408(b)(2)(D), EPA has
reviewed the available scientific data
and other relevant information in
support of this action. EPA has
sufficient data to assess the hazards of
and to make a determination on
aggregate exposure for ipconazole
including exposure resulting from the
tolerances established by this action.
EPA’s assessment of exposures and risks
associated with ipconazole follows.
A. Toxicological Profile
EPA has evaluated the available
toxicity data and considered its validity,
completeness, and reliability as well as
the relationship of the results of the
studies to human risk. EPA has also
considered available information
concerning the variability of the
sensitivities of major identifiable
subgroups of consumers, including
infants and children. Ipconazole has
low acute toxicity via the oral, dermal,
and inhalation routes of exposure. It
causes low to mild irritation to the eyes
and skin; it is not a dermal sensitizer.
Ipconazole may cause local, portal-ofentry irritation via all routes following
repeated exposure. Systemic effects that
were noted in dogs, mice, rabbits and/
or rats following exposure to ipconazole
were generally limited to decreased
body weight, body weight gain, and
food consumption; and liver and kidney
effects. Developmental effects were
observed only at the maternally-toxic
dose. No consistent evidence of
neurotoxicity was observed following
acute, subchronic, or chronic dosing in
multiple species in the available
ipconazole database and the triazole
fungicides as a group typically show
either no evidence of neurotoxicity or
neurotoxicity at doses significantly
higher than the regulatory points of
departure. Ipconazole is classified as not
likely to be a human carcinogen and
there is no concern for mutagenicity.
Specific information on the studies
received and the nature of the adverse
effects caused by ipconazole as well as
the no-observed-adverse-effect-level
(NOAEL) and the lowest-observedadverse-effect-level (LOAEL) from the
toxicity studies can be found at https://
www.regulations.gov in document
‘‘Revised Ipconazole Human Health
Risk Assessment of the Proposed Use on
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Legume Vegetables (Crop Group 6)’’ on
page 23 in docket ID number EPA–HQ–
OPP–2012–0796.
B. Toxicological Points of Departure
(POD)/Levels of Concern (LOC)
Once a pesticide’s toxicological
profile is determined, EPA identifies
toxicological POD and LOC to use in
evaluating the risk posed by human
exposure to the pesticide. For hazards
that have a threshold below which there
is no appreciable risk, the toxicological
POD is used as the basis for derivation
of reference values for risk assessment.
PODs are developed based on a careful
analysis of the doses in each
toxicological study to determine the
dose at which no adverse effects are
observed (the NOAEL) and the lowest
dose at which adverse effects of concern
are identified (the LOAEL). Uncertainty/
safety factors (SF) are used in
conjunction with the POD to calculate a
safe exposure level—generally referred
to as a population-adjusted dose (PAD)
or a reference dose (RfD)—and a safe
margin of exposure (MOE). For nonthreshold risks, the Agency assumes
that any amount of exposure will lead
15237
to some degree of risk. Thus, the Agency
estimates risk in terms of the probability
of an occurrence of the adverse effect
expected in a lifetime. For more
information on the general principles
EPA uses in risk characterization and a
complete description of the risk
assessment process, see https://
www.epa.gov/pesticides/factsheets/
riskassess.htm.
A summary of the toxicological
endpoints for ipconazole used for
human risk assessment is shown in the
following Table of this unit.
TABLE—SUMMARY OF TOXICOLOGICAL DOSES AND ENDPOINTS FOR IPCONAZOLE FOR USE IN HUMAN HEALTH RISK
ASSESSMENT
Exposure/scenario
Acute dietary (females 13–50
years of age).
Acute dietary (general population including infants and
children).
Chronic dietary (all populations)
Cancer (oral, dermal, inhalation).
Point of departure
and uncertainty/
safety factors
RfD, PAD, LOC for
risk assessment
Study and toxicological effects
NOAEL = 10 mg/kg/
day
UFA = 10x
UFH = 10x
FQPA SF = 1x
No appropriate endpoint attributable
to a single dose of
ipconazole was
identified for this
population.
NOAEL= 1.5 mg/kg/
day
UFA = 10x
UFH = 10x
FQPA SF = 1x
Acute RfD = 0.1 mg/
kg/day
aPAD = 0.1 mg/kg/
day
Co-critical developmental toxicity studies in rats and rabbits.
LOAELrats = 30 mg/kg/day, based on increased visceral and
skeletal variations.
LOAELrabbits = 50 mg/kg/day, based on increased incidence of
skeletal variations and malformations.
Chronic RfD = 0.015
mg/kg/day
cPAD = 0.015 mg/
kg/day
Chronic toxicity study in dogs.
LOAEL = 5 mg/kg/day, based on skin reddening (both sexes)
and decreased body weight gain in females.
No evidence of carcinogenicity. Classification: Not likely to be a human carcinogen, based on two adequate rodent carcinogenicity studies.
FQPA SF = Food Quality Protection Act Safety Factor. LOAEL = lowest-observed-adverse-effect-level. LOC = level of concern. mg/kg/day =
milligram/kilogram/day. MOE = margin of exposure. NOAEL = no-observed-adverse-effect-level. PAD = population adjusted dose (a = acute, c =
chronic). UF = uncertainty factor. UFA = extrapolation from animal to human (interspecies). UFH = potential variation in sensitivity among members of the human population (intraspecies).
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C. Exposure Assessment
1. Dietary exposure from food and
feed uses. In evaluating dietary
exposure to ipconazole, EPA considered
exposure under the petitioned-for
tolerances as well as all existing
ipconazole tolerances in 40 CFR
180.646. EPA assessed dietary
exposures from ipconazole in food as
follows:
i. Acute exposure. Quantitative acute
dietary exposure and risk assessments
are performed for a food-use pesticide,
if a toxicological study has indicated the
possibility of an effect of concern
occurring as a result of a 1-day or single
exposure.
No acute endpoint attributable to a
single exposure and relevant for the
general population was identified in the
toxicity database for ipconazole. A
developmental endpoint suitable for
acute assessment was identified;
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therefore an acute dietary assessment
was performed only for women of childbearing age (females 13–49 years old). In
estimating acute dietary exposure, EPA
used the Dietary Exposure Evaluation
Model software with the Food
Commodity Intake Database, Version
3.16 (DEEM–FCID), which uses food
consumption information from the U.S.
Department of Agriculture (USDA)
2003–2008 National Health and
Nutrition Examination Surveys of What
We Eat in America (NHANES/WWEIA).
As to residue levels in food, EPA
assumed tolerance level residues and
100% crop treated.
ii. Chronic exposure. In conducting
the chronic dietary exposure assessment
EPA used the food consumption data
from the USDA 2003–2008 National
Health and Nutrition Examination
Surveys of What We Eat in America
(NHANES/WWEIA). As to residue levels
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in food, EPA used tolerance level
residues and assumed 100% crop
treated.
iii. Cancer. Based on the data
summarized in Unit III.A., EPA has
concluded that ipconazole is not a likely
carcinogen. Therefore, a dietary
exposure assessment for the purpose of
assessing cancer risk is unnecessary.
iv. Anticipated residue and percent
crop treated (PCT) information. EPA did
not use anticipated residue and/or PCT
information in the dietary assessment
for ipconazole. Tolerance level residues
and/or 100% crop treated were assumed
for all food commodities.
2. Dietary exposure from drinking
water. The Agency used screening level
water exposure models in the dietary
exposure analysis and risk assessment
for ipconazole in drinking water. These
simulation models take into account
data on the physical, chemical, and fate/
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transport characteristics of ipconazole.
Further information regarding EPA
drinking water models used in pesticide
exposure assessment can be found at
https://www.epa.gov/oppefed1/models/
water/index.htm.
Based on the Pesticide Root Zone
Model/Exposure Analysis Modeling
System (PRZM/EXAMS), and Pesticide
Root Zone Model Ground Water (PRZM
GW) models, the estimated drinking
water concentrations (EDWCs) of
ipconazole for acute exposures are
estimated to be 0.173 parts per billion
(ppb) for surface water and 1.01 ppb for
ground water.
The EDWCs of ipconazole for chronic
exposures for non-cancer assessments
are estimated to be 0.105 ppb for surface
water and 0.822 ppb for ground water.
Modeled estimates of drinking water
concentrations were directly entered
into the dietary exposure model. For
acute dietary risk assessment, the water
concentration value of 1.01 ppb was
used to assess the contribution from
drinking water. For chronic dietary risk
assessment, the water concentration of
value 0.822 ppb was used to assess the
contribution from drinking water.
3. From non-dietary exposure. The
term ‘‘residential exposure’’ is used in
this document to refer to nonoccupational, non-dietary exposure
(e.g., for lawn and garden pest control,
indoor pest control, termiticides, and
flea and tick control on pets).
Ipconazole is not registered for any
specific use patterns that would result
in residential exposure.
4. Cumulative effects from substances
with a common mechanism of toxicity.
Section 408(b)(2)(D)(v) of FFDCA
requires that, when considering whether
to establish, modify, or revoke a
tolerance, the Agency consider
‘‘available information’’ concerning the
cumulative effects of a particular
pesticide’s residues and ‘‘other
substances that have a common
mechanism of toxicity.’’
Ipconazole is a member of the
conazole class of pesticides. Although
conazoles act similarly in plants by
inhibiting ergosterol biosynthesis, there
is not necessarily a relationship between
their pesticidal activity and their
mechanism of toxicity in mammals.
Structural similarities do not constitute
a common mechanism of toxicity.
Evidence is needed to establish that the
chemicals operate by the same, or
essentially the same, sequence of major
biochemical events (EPA, 2002). In
conazoles, however, a variable pattern
of toxicological responses is found;
some are hepatotoxic and
hepatocarcinogenic in mice. Some
induce thyroid tumors in rats. Some
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induce developmental, reproductive,
and neurological effects in rodents.
Furthermore, the conazoles produce a
diverse range of biochemical events,
including altered cholesterol levels,
stress responses, and altered DNA
methylation. It is not clearly understood
whether these biochemical events are
directly connected to their toxicological
outcomes. Thus, there is currently no
conclusive data to indicate that
conazoles share common mechanisms of
toxicity and EPA is not following a
cumulative risk approach based on a
common mechanism of toxicity for the
conazoles. For information regarding
EPA’s procedures for cumulating effects
from substances found to have a
common mechanism of toxicity, see
EPA’s Web site at https://www.epa.gov/
pesticides/cumulative.
Ipconazole is a triazole-derived
pesticide. This class of compounds can
form the common metabolite 1,2,4triazole and three triazole conjugates
(triazolylalanine, triazolylacetic acid,
and triazolylpyrivic acid). To support
existing tolerances and to establish new
tolerances for triazole-derivative
pesticides, including ipconazole, U.S.
EPA conducted a human health risk
assessment for exposure to 1,2,4triazole, triazolylalanine, and
triazolylacetic acid resulting from the
use of all current and pending uses of
any triazole-derived fungicide. The risk
assessment is a highly conservative,
screening-level evaluation in terms of
hazards associated with common
metabolites (e.g., use of a maximum
combination of uncertainty factors) and
potential dietary and non-dietary
exposures (i.e., high end estimates of
both dietary and non-dietary exposures).
In addition, the Agency retained the
additional 10x FQPA SF for the
protection of infants and children. The
assessment includes evaluations of risks
for various subgroups, including those
comprised of infants and children. The
Agency’s complete risk assessment is
found in the propiconazole
reregistration docket at https://
www.regulations.gov, Docket
Identification (ID) Number EPA–HQ–
OPP–2005–0497.
An updated dietary exposure and risk
analysis for the common triazole
metabolites 1,2,4-triazole (T),
triazolylalanine (TA), triazolylacetic
acid (TAA), and triazolylpyruvic acid
(TP) was conducted completed in May
2013, in association with a registration
request for several other triazole
fungicides. That analysis concluded that
risk estimates were below the Agency’s
level of concern for all population
groups. After inclusion of ipconazole
uses covered by this action, aggregate
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risk estimates for T, TA, TAA, and TP
for all durations of exposure and for all
population subgroups are below the
Agency’s level of concern. This updated
assessment may be found on https://
www.regulations.gov by searching for
the following title and docket number:
‘‘Common Triazole Metabolites:
Updated Aggregate Human Health Risk
Assessment to Address The New
Section 3 Registrations For Use of
Prothioconazole on Bushberry Crop
Subgroup 13–07B, Low Growing Berry,
Except Strawberry, Crop Subgroup 13–
07H, and Cucurbit Vegetables Crop
Group 9; Use of Flutriafol on Coffee; and
Ipconazole on Crop Group 6’’ (located in
docket ID number EPA–HQ–OPP–2012–
0876).
D. Safety Factor for Infants and
Children
1. In general. Section 408(b)(2)(C) of
FFDCA provides that EPA shall apply
an additional tenfold (10x) margin of
safety for infants and children in the
case of threshold effects to account for
prenatal and postnatal toxicity and the
completeness of the database on toxicity
and exposure unless EPA determines
based on reliable data that a different
margin of safety will be safe for infants
and children. This additional margin of
safety is commonly referred to as the
FQPA SF. In applying this provision,
EPA either retains the default value of
10x, or uses a different additional safety
factor when reliable data available to
EPA support the choice of a different
factor.
2. Prenatal and postnatal sensitivity.
Offspring effects only occurred in the
presence of maternal toxicity and were
not considered more severe than the
parental effects. Therefore, EPA
concluded that there is no quantitative
or qualitative evidence of increased
susceptibility to rat or rabbit fetuses
exposed in utero and/or post-natally to
ipconazole.
3. Conclusion. EPA has determined
that reliable data show the safety of
infants and children would be
adequately protected if the FQPA SF
were reduced to 1x. That decision is
based on the following findings:
i. The toxicity database for ipconazole
is complete. The Agency waived the
requirement for an Immunotoxicity
study for ipconazole as there is minimal
evidence that ipconazole targets the
immune system, nor are the conazoles
of a chemical class expected to have an
adverse effect on the immune system.
An increase in leukocytes was observed
in females at 78.3 mg/kg/day in the 28day inhalation study, however this was
not of concern because it was the only
evidence of potential immunotoxicity in
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the entire ipconazole database, the effect
occurred at a dose 10-fold higher than
the dose (7.8 mg/kg/day) that caused
portal-of-entry effects in the same study,
and the effect occurred at a dose much
greater than the PODs chosen for risk
assessment. The overall weight of
evidence suggests that ipconazole does
not target the immune system.
ii. There is no consistent evidence of
neurotoxicity in the available databases.
Clinical signs suggestive of
neurotoxicity were observed in the in
vivo mammalian cytogenetics study;
however, they were seen at relatively
high doses, which far exceed the
anticipated dietary exposure, and no
other signs were observed in any of the
other studies, including studies with
neurotoxicity assessments. Based on the
lack of evidence in the database, EPA
waived the requirement for the acute
neurotoxicity study. Also, the
subchronic neurotoxicity study
requirement is considered to be satisfied
by the neurotoxicity assessments
performed in both the rat subchronic
and chronic toxicity/carcinogenicity
studies in which no signs of
neurotoxicity were observed. This is
consistent with what is known about the
triazole fungicides as a class, which
typically show either no evidence of
neurotoxicity or neurotoxicity at doses
significantly higher than the regulatory
points of departure. Therefore,
ipconazole is not considered to be
neurotoxic and there is no need for a
developmental neurotoxicity study or
additional UFs to account for
neurotoxicity.
iii. There is no evidence that
ipconazole results in increased
susceptibility following in utero
exposure for rats or rabbits in the
prenatal developmental studies or in
young rats in the 2-generation
reproduction study, and there are no
residual uncertainties with respect to
pre- or postnatal exposure.
iv. There are no residual uncertainties
identified in the exposure databases.
EPA made conservative (protective)
assumptions in the ground and surface
water modeling used to assess exposure
to ipconazole in drinking water. These
assessments will not underestimate the
exposure and risks posed by ipconazole.
E. Aggregate Risks and Determination of
Safety
EPA determines whether acute and
chronic dietary pesticide exposures are
safe by comparing aggregate exposure
estimates to the acute PAD (aPAD) and
chronic PAD (cPAD). For linear cancer
risks, EPA calculates the lifetime
probability of acquiring cancer given the
estimated aggregate exposure. Short-,
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intermediate-, and chronic-term risks
are evaluated by comparing the
estimated aggregate food, water, and
residential exposure to the appropriate
PODs to ensure that an adequate MOE
exists.
1. Acute risk. An acute aggregate risk
assessment takes into account exposure
estimates from acute dietary
consumption of food and drinking
water. Using the exposure assumptions
discussed in this unit for acute
exposure, the acute dietary exposure
from food and water to ipconazole will
occupy <1% of the aPAD for females
13–49 years old, the population group
receiving the greatest exposure.
2. Chronic risk. Using the exposure
assumptions described in this unit for
chronic exposure, EPA has concluded
that chronic exposure to ipconazole
from food and water will utilize <1% of
the cPAD for children 1–2 years old the
population group receiving the greatest
exposure. There are no residential uses
for ipconazole.
3. Short-term risk. Short-term
aggregate exposure takes into account
short-term residential exposure plus
chronic exposure to food and water
(considered to be a background
exposure level).
A short-term adverse effect was
identified; however, ipconazole is not
registered for any use patterns that
would result in short-term residential
exposure. Because there is no short-term
residential exposure and chronic dietary
exposure has already been assessed
under the appropriately protective
cPAD (which is at least as protective as
the POD used to assess short-term risk),
no further assessment of short-term risk
is necessary, and EPA relies on the
chronic dietary risk assessment for
evaluating short-term risk for
ipconazole.
4. Intermediate-term risk.
Intermediate-term aggregate exposure
takes into account intermediate-term
residential exposure plus chronic
exposure to food and water (considered
to be a background exposure level).
An intermediate-term adverse effect
was identified; however, ipconazole is
not registered for any use patterns that
would result in intermediate-term
residential exposure. Because there is
no intermediate-term residential
exposure and chronic dietary exposure
has already been assessed under the
appropriately protective cPAD (which is
at least as protective as the POD used to
assess intermediate-term risk), no
further assessment of intermediate-term
risk is necessary, and EPA relies on the
chronic dietary risk assessment for
evaluating intermediate-term risk for
ipconazole.
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15239
5. Aggregate cancer risk for U.S.
population. Ipconazole has been
classified as not likely to be
carcinogenic, and is not expected to
pose a cancer risk to humans.
6. Determination of safety. Based on
these risk assessments, EPA concludes
that there is a reasonable certainty that
no harm will result to the general
population, or to infants and children
from aggregate exposure to ipconazole
residues.
IV. Other Considerations
A. Analytical Enforcement Methodology
Adequate enforcement methodology
(liquid chromatography/mass
spectrometry/mass spectrometry (LC/
MS/MS) (AC/3020)) is available to
enforce the tolerance expression. The
method may be requested from: Chief,
Analytical Chemistry Branch,
Environmental Science Center, 701
Mapes Rd., Ft. Meade, MD 20755–5350;
telephone number: (410) 305–2905;
email address: residuemethods@
epa.gov.
B. International Residue Limits
In making its tolerance decisions, EPA
seeks to harmonize U.S. tolerances with
international standards whenever
possible, consistent with U.S. food
safety standards and agricultural
practices. EPA considers the
international maximum residue limits
(MRLs) established by the Codex
Alimentarius Commission (Codex), as
required by FFDCA section 408(b)(4).
The Codex Alimentarius is a joint
United Nations Food and Agriculture
Organization/World Health
Organization food standards program,
and it is recognized as an international
food safety standards-setting
organization in trade agreements to
which the United States is a party. EPA
may establish a tolerance that is
different from a Codex MRL; however,
FFDCA section 408(b)(4) requires that
EPA explain the reasons for departing
from the Codex level.
The Codex has not established a MRL
for ipconazole on vegetable, legume,
group 6.
C. Revisions to Petitioned-for Tolerances
The petitioner requested a tolerance
for residues of ipconazole in or on
‘‘legume vegetables succulent or dried,
crop group 6’’. EPA is correcting the
commodity term and establishing a
tolerance for ‘‘vegetable, legume, group
6’’.
V. Conclusion
Therefore, tolerances are established
for residues of ipconazole (2-[(4chlorophenyl)methyl]-5-(1-
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methylethyl)-1-(1H-1,2,4-triazol-1ylmethyl)cyclopentanol) in or on
vegetable, legume, group 6 at 0.01 ppm.
EPA is revising the tolerance expression
for ipconazole to clarify that metabolites
and degradates are covered by the
tolerances and to specify how
compliance with the tolerances is to be
measured. The existing tolerances for
pea and bean, dried shelled, except
soybean, subgroup 6C at 0.01 ppm and
soybean, seed at 0.01 ppm will be
removed from paragraph (a) of § 180.646
as these tolerances are encompassed
within vegetable, legume, group 6.
VI. Statutory and Executive Order
Reviews
This final rule establishes tolerances
under FFDCA section 408(d) in
response to a petition submitted to the
Agency. The Office of Management and
Budget (OMB) has exempted these types
of actions from review under Executive
Order 12866, entitled ‘‘Regulatory
Planning and Review’’ (58 FR 51735,
October 4, 1993). Because this final rule
has been exempted from review under
Executive Order 12866, this final rule is
not subject to Executive Order 13211,
entitled ‘‘Actions Concerning
Regulations That Significantly Affect
Energy Supply, Distribution, or Use’’ (66
FR 28355, May 22, 2001) or Executive
Order 13045, entitled ‘‘Protection of
Children from Environmental Health
Risks and Safety Risks’’ (62 FR 19885,
April 23, 1997). This final rule does not
contain any information collections
subject to OMB approval under the
Paperwork Reduction Act (PRA) (44
U.S.C. 3501 et seq.), nor does it require
any special considerations under
Executive Order 12898, entitled
‘‘Federal Actions to Address
Environmental Justice in Minority
Populations and Low-Income
Populations’’ (59 FR 7629, February 16,
1994).
Since tolerances and exemptions that
are established on the basis of a petition
under FFDCA section 408(d), such as
the tolerances in this final rule, do not
require the issuance of a proposed rule,
the requirements of the Regulatory
Flexibility Act (RFA) (5 U.S.C. 601 et
seq.), do not apply.
This final rule directly regulates
growers, food processors, food handlers,
and food retailers, not States or tribes,
nor does this action alter the
relationships or distribution of power
and responsibilities established by
Congress in the preemption provisions
of FFDCA section 408(n)(4). As such,
the Agency has determined that this
action will not have a substantial direct
effect on States or tribal governments,
on the relationship between the national
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government and the States or tribal
governments, or on the distribution of
power and responsibilities among the
various levels of government or between
the Federal Government and Indian
Tribes. Thus, the Agency has
determined that Executive Order 13132,
entitled ‘‘Federalism’’ (64 FR 43255,
August 10, 1999) and Executive Order
13175, entitled ‘‘Consultation and
Coordination with Indian Tribal
Governments’’ (65 FR 67249, November
9, 2000) do not apply to this final rule.
In addition, this final rule does not
impose any enforceable duty or contain
any unfunded mandate as described
under Title II of the Unfunded Mandates
Reform Act of 1995 (UMRA) (2 U.S.C.
1501 et seq.).
This action does not involve any
technical standards that would require
Agency consideration of voluntary
consensus standards pursuant to section
12(d) of the National Technology
Transfer and Advancement Act of 1995
(NTTAA) (15 U.S.C. 272 note).
§ 180.646 Ipconazole; tolerances for
residues.
VII. Congressional Review Act
DEPARTMENT OF HEALTH AND
HUMAN SERVICES
Pursuant to the Congressional Review
Act (5 U.S.C. 801 et seq.), EPA will
submit a report containing this rule and
other required information to the U.S.
Senate, the U.S. House of
Representatives, and the Comptroller
General of the United States prior to
publication of the rule in the Federal
Register. This action is not a ‘‘major
rule’’ as defined by 5 U.S.C. 804(2).
List of Subjects in 40 CFR Part 180
Environmental protection,
Administrative practice and procedure,
Agricultural commodities, Pesticides
and pests, Reporting and recordkeeping
requirements.
Dated: March 12, 2014.
Lois Rossi,
Director, Registration Division, Office of
Pesticide Programs.
Therefore, 40 CFR chapter I is
amended as follows:
PART 180—[AMENDED]
1. The authority citation for part 180
continues to read as follows:
■
Authority: 21 U.S.C. 321(q), 346a and 371.
2. In § 180.646:
a. Revise the introductory text in
paragraph (a).
■ b. Remove ‘‘Pea and bean, dried
shelled, except soybean, subgroup 6C’’,
and ‘‘Soybean, seed’’ from the table in
paragraph (a).
■ c. Add alphabetically ‘‘Vegetable,
legume, group 6’’ to the table in
paragraph (a).
The amendments read as follows:
■
■
PO 00000
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(a) General. Tolerances are
established for residues of ipconazole,
including its metabolites and
degradates, in or on the commodities
listed in the table below. Compliance
with the tolerance levels specified
below is to be determined by measuring
only ipconazole (2-[(4chlorophenyl)methyl]-5-(1methylethyl)-1-(1H-1,2,4-triazol-1ylmethyl)cyclopentanol) in or on the
commodity.
Parts per
million
Commodity
*
*
*
Vegetable, legume, group 6
*
*
*
*
*
*
0.01
*
[FR Doc. 2014–06059 Filed 3–18–14; 8:45 am]
BILLING CODE 6560–50–P
45 CFR Part 156
[CMS–9943–IFC]
RIN 0938–AS28
Patient Protection and Affordable Care
Act; Third Party Payment of Qualified
Health Plan Premiums
Centers for Medicare and
Medicaid Services, Department of
Health and Human Services (HHS).
ACTION: Interim final rule with comment
period.
AGENCY:
This interim final rule
requires issuers of qualified health plans
(QHPs), including stand-alone dental
plans (SADPs), to accept premium and
cost-sharing payments made on behalf
of enrollees by the Ryan White HIV/
AIDS Program, other Federal and State
government programs that provide
premium and cost sharing support for
specific individuals, and Indian tribes,
tribal organizations, and urban Indian
organizations.
SUMMARY:
Effective Date: This interim final
rule is effective on March 14, 2014.
Comment date: To be assured
consideration, comments must be
received at one of the addresses
provided below, no later than 5 p.m. on
May 13, 2014.
ADDRESSES: In commenting, please refer
to file code CMS–9943–IFC. Because of
staff and resource limitations, we cannot
accept comments by facsimile (FAX)
transmission. You may submit
DATES:
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[Federal Register Volume 79, Number 53 (Wednesday, March 19, 2014)]
[Rules and Regulations]
[Pages 15235-15240]
From the Federal Register Online via the Government Printing Office [www.gpo.gov]
[FR Doc No: 2014-06059]
-----------------------------------------------------------------------
ENVIRONMENTAL PROTECTION AGENCY
40 CFR Part 180
[EPA-HQ-OPP-2012-0796; FRL-9907-25]
Ipconazole; Pesticide Tolerances
AGENCY: Environmental Protection Agency (EPA).
ACTION: Final rule.
-----------------------------------------------------------------------
SUMMARY: This regulation establishes tolerances for residues of
ipconazole in or on vegetable, legume, group 6. Chemtura Corporation
requested these tolerances under the Federal Food, Drug, and Cosmetic
Act (FFDCA).
DATES: This regulation is effective March 19, 2014. Objections and
requests for hearings must be received on or before May 19, 2014, and
must be filed in accordance with the instructions provided in 40 CFR
part 178 (see also Unit I.C. of the SUPPLEMENTARY INFORMATION).
ADDRESSES: The docket for this action, identified by docket
identification (ID) number EPA-HQ-OPP-2012-0796, is available at https://www.regulations.gov or at the Office of Pesticide Programs Regulatory
Public Docket (OPP Docket) in the Environmental Protection Agency
Docket Center (EPA/DC), EPA West Bldg., Rm. 3334, 1301 Constitution
Ave. NW., Washington, DC 20460-0001. The Public Reading Room is open
from 8:30 a.m. to 4:30 p.m., Monday through Friday, excluding legal
holidays. The telephone number for the Public Reading Room is (202)
566-1744, and the telephone number for the OPP Docket is (703) 305-
5805. Please review the visitor instructions and additional information
about the docket available at https://www.epa.gov/dockets.
FOR FURTHER INFORMATION CONTACT: Lois Rossi, Registration Division
(7505P), Office of Pesticide Programs, Environmental Protection Agency,
1200 Pennsylvania Ave. NW., Washington,
[[Page 15236]]
DC 20460-0001; telephone number: (703) 305-7090; email address:
RDFRNotices@epa.gov.
SUPPLEMENTARY INFORMATION:
I. General Information
A. Does this action apply to me?
You may be potentially affected by this action if you are an
agricultural producer, food manufacturer, or pesticide manufacturer.
The following list of North American Industrial Classification System
(NAICS) codes is not intended to be exhaustive, but rather provides a
guide to help readers determine whether this document applies to them.
Potentially affected entities may include:
Crop production (NAICS code 111).
Animal production (NAICS code 112).
Food manufacturing (NAICS code 311).
Pesticide manufacturing (NAICS code 32532).
B. How can I get electronic access to other related information?
You may access a frequently updated electronic version of EPA's
tolerance regulations at 40 CFR part 180 through the Government
Printing Office's e-CFR site at https://www.ecfr.gov/cgi-bin/text-idx?&c=ecfr&tpl=/ecfrbrowse/Title40/40tab_02.tpl.
C. How can I file an objection or hearing request?
Under FFDCA section 408(g), 21 U.S.C. 346a, any person may file an
objection to any aspect of this regulation and may also request a
hearing on those objections. You must file your objection or request a
hearing on this regulation in accordance with the instructions provided
in 40 CFR part 178. To ensure proper receipt by EPA, you must identify
docket ID number EPA-HQ-OPP-2012-0796 in the subject line on the first
page of your submission. All objections and requests for a hearing must
be in writing, and must be received by the Hearing Clerk on or before
May 19, 2014. Addresses for mail and hand delivery of objections and
hearing requests are provided in 40 CFR 178.25(b).
In addition to filing an objection or hearing request with the
Hearing Clerk as described in 40 CFR part 178, please submit a copy of
the filing (excluding any Confidential Business Information (CBI)) for
inclusion in the public docket. Information not marked confidential
pursuant to 40 CFR part 2 may be disclosed publicly by EPA without
prior notice. Submit the non-CBI copy of your objection or hearing
request, identified by docket ID number EPA-HQ-OPP-2012-0796, by one of
the following methods:
Federal eRulemaking Portal: https://www.regulations.gov.
Follow the online instructions for submitting comments. Do not submit
electronically any information you consider to be CBI or other
information whose disclosure is restricted by statute.
Mail: OPP Docket, Environmental Protection Agency Docket
Center (EPA/DC), (28221T), 1200 Pennsylvania Ave. NW., Washington, DC
20460-0001.
Hand Delivery: To make special arrangements for hand
delivery or delivery of boxed information, please follow the
instructions at https://www.epa.gov/dockets/contacts.html.
Additional instructions on commenting or visiting the docket, along
with more information about dockets generally, is available at https://www.epa.gov/dockets.
II. Summary of Petitioned-for Tolerance
In the Federal Register of December 19, 2012 (77 FR 75082) (FRL-
9372-6), EPA issued a document pursuant to FFDCA section 408(d)(3), 21
U.S.C. 346a(d)(3), announcing the filing of a pesticide petition (PP
2F8076) by Chemtura Corporation, 199 Benson Rd., Middlebury, CT 06749.
The petition requested that 40 CFR 180.646 be amended by establishing
tolerances for residues of the fungicide ipconazole (2-[(4-
chlorophenyl)methyl]-5-(1-methylethyl)-1-(1H-1,2,4-triazol-1-
ylmethyl)cyclopentanol) in or on legume vegetables, succulent or dried,
crop group 6 at 0.01 parts per million (ppm). That document referenced
a summary of the petition prepared by Chemtura Corporation, the
registrant, which is available in the docket, https://www.regulations.gov. There were no comments received in response to the
notice of filing.
III. Aggregate Risk Assessment and Determination of Safety
Section 408(b)(2)(A)(i) of FFDCA allows EPA to establish a
tolerance (the legal limit for a pesticide chemical residue in or on a
food) only if EPA determines that the tolerance is ``safe.'' Section
408(b)(2)(A)(ii) of FFDCA defines ``safe'' to mean that ``there is a
reasonable certainty that no harm will result from aggregate exposure
to the pesticide chemical residue, including all anticipated dietary
exposures and all other exposures for which there is reliable
information.'' This includes exposure through drinking water and in
residential settings, but does not include occupational exposure.
Section 408(b)(2)(C) of FFDCA requires EPA to give special
consideration to exposure of infants and children to the pesticide
chemical residue in establishing a tolerance and to ``ensure that there
is a reasonable certainty that no harm will result to infants and
children from aggregate exposure to the pesticide chemical residue. . .
.''
Consistent with FFDCA section 408(b)(2)(D), and the factors
specified in FFDCA section 408(b)(2)(D), EPA has reviewed the available
scientific data and other relevant information in support of this
action. EPA has sufficient data to assess the hazards of and to make a
determination on aggregate exposure for ipconazole including exposure
resulting from the tolerances established by this action. EPA's
assessment of exposures and risks associated with ipconazole follows.
A. Toxicological Profile
EPA has evaluated the available toxicity data and considered its
validity, completeness, and reliability as well as the relationship of
the results of the studies to human risk. EPA has also considered
available information concerning the variability of the sensitivities
of major identifiable subgroups of consumers, including infants and
children. Ipconazole has low acute toxicity via the oral, dermal, and
inhalation routes of exposure. It causes low to mild irritation to the
eyes and skin; it is not a dermal sensitizer. Ipconazole may cause
local, portal-of-entry irritation via all routes following repeated
exposure. Systemic effects that were noted in dogs, mice, rabbits and/
or rats following exposure to ipconazole were generally limited to
decreased body weight, body weight gain, and food consumption; and
liver and kidney effects. Developmental effects were observed only at
the maternally-toxic dose. No consistent evidence of neurotoxicity was
observed following acute, subchronic, or chronic dosing in multiple
species in the available ipconazole database and the triazole
fungicides as a group typically show either no evidence of
neurotoxicity or neurotoxicity at doses significantly higher than the
regulatory points of departure. Ipconazole is classified as not likely
to be a human carcinogen and there is no concern for mutagenicity.
Specific information on the studies received and the nature of the
adverse effects caused by ipconazole as well as the no-observed-
adverse-effect-level (NOAEL) and the lowest-observed-adverse-effect-
level (LOAEL) from the toxicity studies can be found at https://www.regulations.gov in document ``Revised Ipconazole Human Health Risk
Assessment of the Proposed Use on
[[Page 15237]]
Legume Vegetables (Crop Group 6)'' on page 23 in docket ID number EPA-
HQ-OPP-2012-0796.
B. Toxicological Points of Departure (POD)/Levels of Concern (LOC)
Once a pesticide's toxicological profile is determined, EPA
identifies toxicological POD and LOC to use in evaluating the risk
posed by human exposure to the pesticide. For hazards that have a
threshold below which there is no appreciable risk, the toxicological
POD is used as the basis for derivation of reference values for risk
assessment. PODs are developed based on a careful analysis of the doses
in each toxicological study to determine the dose at which no adverse
effects are observed (the NOAEL) and the lowest dose at which adverse
effects of concern are identified (the LOAEL). Uncertainty/safety
factors (SF) are used in conjunction with the POD to calculate a safe
exposure level--generally referred to as a population-adjusted dose
(PAD) or a reference dose (RfD)--and a safe margin of exposure (MOE).
For non-threshold risks, the Agency assumes that any amount of exposure
will lead to some degree of risk. Thus, the Agency estimates risk in
terms of the probability of an occurrence of the adverse effect
expected in a lifetime. For more information on the general principles
EPA uses in risk characterization and a complete description of the
risk assessment process, see https://www.epa.gov/pesticides/factsheets/riskassess.htm.
A summary of the toxicological endpoints for ipconazole used for
human risk assessment is shown in the following Table of this unit.
Table--Summary of Toxicological Doses and Endpoints for Ipconazole for Use in Human Health Risk Assessment
----------------------------------------------------------------------------------------------------------------
Point of departure
Exposure/scenario and uncertainty/ RfD, PAD, LOC for Study and toxicological effects
safety factors risk assessment
----------------------------------------------------------------------------------------------------------------
Acute dietary (females 13-50 NOAEL = 10 mg/kg/day Acute RfD = 0.1 mg/ Co-critical developmental toxicity
years of age). UFA = 10x........... kg/day studies in rats and rabbits.
UFH = 10x........... aPAD = 0.1 mg/kg/ LOAELrats = 30 mg/kg/day, based on
FQPA SF = 1x........ day. increased visceral and skeletal
variations.
LOAELrabbits = 50 mg/kg/day, based
on increased incidence of
skeletal variations and
malformations.
Acute dietary (general population No appropriate
including infants and children). endpoint
attributable to a
single dose of
ipconazole was
identified for this
population.
Chronic dietary (all populations) NOAEL= 1.5 mg/kg/day Chronic RfD = 0.015 Chronic toxicity study in dogs.
UFA = 10x........... mg/kg/day LOAEL = 5 mg/kg/day, based on skin
UFH = 10x........... cPAD = 0.015 mg/kg/ reddening (both sexes) and
FQPA SF = 1x........ day. decreased body weight gain in
females.
------------------------------------------------------------------------------
Cancer (oral, dermal, inhalation) No evidence of carcinogenicity. Classification: Not likely to be a human
carcinogen, based on two adequate rodent carcinogenicity studies.
----------------------------------------------------------------------------------------------------------------
FQPA SF = Food Quality Protection Act Safety Factor. LOAEL = lowest-observed-adverse-effect-level. LOC = level
of concern. mg/kg/day = milligram/kilogram/day. MOE = margin of exposure. NOAEL = no-observed-adverse-effect-
level. PAD = population adjusted dose (a = acute, c = chronic). UF = uncertainty factor. UFA = extrapolation
from animal to human (interspecies). UFH = potential variation in sensitivity among members of the human
population (intraspecies).
C. Exposure Assessment
1. Dietary exposure from food and feed uses. In evaluating dietary
exposure to ipconazole, EPA considered exposure under the petitioned-
for tolerances as well as all existing ipconazole tolerances in 40 CFR
180.646. EPA assessed dietary exposures from ipconazole in food as
follows:
i. Acute exposure. Quantitative acute dietary exposure and risk
assessments are performed for a food-use pesticide, if a toxicological
study has indicated the possibility of an effect of concern occurring
as a result of a 1-day or single exposure.
No acute endpoint attributable to a single exposure and relevant
for the general population was identified in the toxicity database for
ipconazole. A developmental endpoint suitable for acute assessment was
identified; therefore an acute dietary assessment was performed only
for women of child-bearing age (females 13-49 years old). In estimating
acute dietary exposure, EPA used the Dietary Exposure Evaluation Model
software with the Food Commodity Intake Database, Version 3.16 (DEEM-
FCID), which uses food consumption information from the U.S. Department
of Agriculture (USDA) 2003-2008 National Health and Nutrition
Examination Surveys of What We Eat in America (NHANES/WWEIA). As to
residue levels in food, EPA assumed tolerance level residues and 100%
crop treated.
ii. Chronic exposure. In conducting the chronic dietary exposure
assessment EPA used the food consumption data from the USDA 2003-2008
National Health and Nutrition Examination Surveys of What We Eat in
America (NHANES/WWEIA). As to residue levels in food, EPA used
tolerance level residues and assumed 100% crop treated.
iii. Cancer. Based on the data summarized in Unit III.A., EPA has
concluded that ipconazole is not a likely carcinogen. Therefore, a
dietary exposure assessment for the purpose of assessing cancer risk is
unnecessary.
iv. Anticipated residue and percent crop treated (PCT) information.
EPA did not use anticipated residue and/or PCT information in the
dietary assessment for ipconazole. Tolerance level residues and/or 100%
crop treated were assumed for all food commodities.
2. Dietary exposure from drinking water. The Agency used screening
level water exposure models in the dietary exposure analysis and risk
assessment for ipconazole in drinking water. These simulation models
take into account data on the physical, chemical, and fate/
[[Page 15238]]
transport characteristics of ipconazole. Further information regarding
EPA drinking water models used in pesticide exposure assessment can be
found at https://www.epa.gov/oppefed1/models/water/index.htm.
Based on the Pesticide Root Zone Model/Exposure Analysis Modeling
System (PRZM/EXAMS), and Pesticide Root Zone Model Ground Water (PRZM
GW) models, the estimated drinking water concentrations (EDWCs) of
ipconazole for acute exposures are estimated to be 0.173 parts per
billion (ppb) for surface water and 1.01 ppb for ground water.
The EDWCs of ipconazole for chronic exposures for non-cancer
assessments are estimated to be 0.105 ppb for surface water and 0.822
ppb for ground water.
Modeled estimates of drinking water concentrations were directly
entered into the dietary exposure model. For acute dietary risk
assessment, the water concentration value of 1.01 ppb was used to
assess the contribution from drinking water. For chronic dietary risk
assessment, the water concentration of value 0.822 ppb was used to
assess the contribution from drinking water.
3. From non-dietary exposure. The term ``residential exposure'' is
used in this document to refer to non-occupational, non-dietary
exposure (e.g., for lawn and garden pest control, indoor pest control,
termiticides, and flea and tick control on pets). Ipconazole is not
registered for any specific use patterns that would result in
residential exposure.
4. Cumulative effects from substances with a common mechanism of
toxicity. Section 408(b)(2)(D)(v) of FFDCA requires that, when
considering whether to establish, modify, or revoke a tolerance, the
Agency consider ``available information'' concerning the cumulative
effects of a particular pesticide's residues and ``other substances
that have a common mechanism of toxicity.''
Ipconazole is a member of the conazole class of pesticides.
Although conazoles act similarly in plants by inhibiting ergosterol
biosynthesis, there is not necessarily a relationship between their
pesticidal activity and their mechanism of toxicity in mammals.
Structural similarities do not constitute a common mechanism of
toxicity. Evidence is needed to establish that the chemicals operate by
the same, or essentially the same, sequence of major biochemical events
(EPA, 2002). In conazoles, however, a variable pattern of toxicological
responses is found; some are hepatotoxic and hepatocarcinogenic in
mice. Some induce thyroid tumors in rats. Some induce developmental,
reproductive, and neurological effects in rodents. Furthermore, the
conazoles produce a diverse range of biochemical events, including
altered cholesterol levels, stress responses, and altered DNA
methylation. It is not clearly understood whether these biochemical
events are directly connected to their toxicological outcomes. Thus,
there is currently no conclusive data to indicate that conazoles share
common mechanisms of toxicity and EPA is not following a cumulative
risk approach based on a common mechanism of toxicity for the
conazoles. For information regarding EPA's procedures for cumulating
effects from substances found to have a common mechanism of toxicity,
see EPA's Web site at https://www.epa.gov/pesticides/cumulative.
Ipconazole is a triazole-derived pesticide. This class of compounds
can form the common metabolite 1,2,4-triazole and three triazole
conjugates (triazolylalanine, triazolylacetic acid, and
triazolylpyrivic acid). To support existing tolerances and to establish
new tolerances for triazole-derivative pesticides, including
ipconazole, U.S. EPA conducted a human health risk assessment for
exposure to 1,2,4-triazole, triazolylalanine, and triazolylacetic acid
resulting from the use of all current and pending uses of any triazole-
derived fungicide. The risk assessment is a highly conservative,
screening-level evaluation in terms of hazards associated with common
metabolites (e.g., use of a maximum combination of uncertainty factors)
and potential dietary and non-dietary exposures (i.e., high end
estimates of both dietary and non-dietary exposures). In addition, the
Agency retained the additional 10x FQPA SF for the protection of
infants and children. The assessment includes evaluations of risks for
various subgroups, including those comprised of infants and children.
The Agency's complete risk assessment is found in the propiconazole
reregistration docket at https://www.regulations.gov, Docket
Identification (ID) Number EPA-HQ-OPP-2005-0497.
An updated dietary exposure and risk analysis for the common
triazole metabolites 1,2,4-triazole (T), triazolylalanine (TA),
triazolylacetic acid (TAA), and triazolylpyruvic acid (TP) was
conducted completed in May 2013, in association with a registration
request for several other triazole fungicides. That analysis concluded
that risk estimates were below the Agency's level of concern for all
population groups. After inclusion of ipconazole uses covered by this
action, aggregate risk estimates for T, TA, TAA, and TP for all
durations of exposure and for all population subgroups are below the
Agency's level of concern. This updated assessment may be found on
https://www.regulations.gov by searching for the following title and
docket number: ``Common Triazole Metabolites: Updated Aggregate Human
Health Risk Assessment to Address The New Section 3 Registrations For
Use of Prothioconazole on Bushberry Crop Subgroup 13-07B, Low Growing
Berry, Except Strawberry, Crop Subgroup 13-07H, and Cucurbit Vegetables
Crop Group 9; Use of Flutriafol on Coffee; and Ipconazole on Crop Group
6'' (located in docket ID number EPA-HQ-OPP-2012-0876).
D. Safety Factor for Infants and Children
1. In general. Section 408(b)(2)(C) of FFDCA provides that EPA
shall apply an additional tenfold (10x) margin of safety for infants
and children in the case of threshold effects to account for prenatal
and postnatal toxicity and the completeness of the database on toxicity
and exposure unless EPA determines based on reliable data that a
different margin of safety will be safe for infants and children. This
additional margin of safety is commonly referred to as the FQPA SF. In
applying this provision, EPA either retains the default value of 10x,
or uses a different additional safety factor when reliable data
available to EPA support the choice of a different factor.
2. Prenatal and postnatal sensitivity. Offspring effects only
occurred in the presence of maternal toxicity and were not considered
more severe than the parental effects. Therefore, EPA concluded that
there is no quantitative or qualitative evidence of increased
susceptibility to rat or rabbit fetuses exposed in utero and/or post-
natally to ipconazole.
3. Conclusion. EPA has determined that reliable data show the
safety of infants and children would be adequately protected if the
FQPA SF were reduced to 1x. That decision is based on the following
findings:
i. The toxicity database for ipconazole is complete. The Agency
waived the requirement for an Immunotoxicity study for ipconazole as
there is minimal evidence that ipconazole targets the immune system,
nor are the conazoles of a chemical class expected to have an adverse
effect on the immune system. An increase in leukocytes was observed in
females at 78.3 mg/kg/day in the 28-day inhalation study, however this
was not of concern because it was the only evidence of potential
immunotoxicity in
[[Page 15239]]
the entire ipconazole database, the effect occurred at a dose 10-fold
higher than the dose (7.8 mg/kg/day) that caused portal-of-entry
effects in the same study, and the effect occurred at a dose much
greater than the PODs chosen for risk assessment. The overall weight of
evidence suggests that ipconazole does not target the immune system.
ii. There is no consistent evidence of neurotoxicity in the
available databases. Clinical signs suggestive of neurotoxicity were
observed in the in vivo mammalian cytogenetics study; however, they
were seen at relatively high doses, which far exceed the anticipated
dietary exposure, and no other signs were observed in any of the other
studies, including studies with neurotoxicity assessments. Based on the
lack of evidence in the database, EPA waived the requirement for the
acute neurotoxicity study. Also, the subchronic neurotoxicity study
requirement is considered to be satisfied by the neurotoxicity
assessments performed in both the rat subchronic and chronic toxicity/
carcinogenicity studies in which no signs of neurotoxicity were
observed. This is consistent with what is known about the triazole
fungicides as a class, which typically show either no evidence of
neurotoxicity or neurotoxicity at doses significantly higher than the
regulatory points of departure. Therefore, ipconazole is not considered
to be neurotoxic and there is no need for a developmental neurotoxicity
study or additional UFs to account for neurotoxicity.
iii. There is no evidence that ipconazole results in increased
susceptibility following in utero exposure for rats or rabbits in the
prenatal developmental studies or in young rats in the 2-generation
reproduction study, and there are no residual uncertainties with
respect to pre- or postnatal exposure.
iv. There are no residual uncertainties identified in the exposure
databases. EPA made conservative (protective) assumptions in the ground
and surface water modeling used to assess exposure to ipconazole in
drinking water. These assessments will not underestimate the exposure
and risks posed by ipconazole.
E. Aggregate Risks and Determination of Safety
EPA determines whether acute and chronic dietary pesticide
exposures are safe by comparing aggregate exposure estimates to the
acute PAD (aPAD) and chronic PAD (cPAD). For linear cancer risks, EPA
calculates the lifetime probability of acquiring cancer given the
estimated aggregate exposure. Short-, intermediate-, and chronic-term
risks are evaluated by comparing the estimated aggregate food, water,
and residential exposure to the appropriate PODs to ensure that an
adequate MOE exists.
1. Acute risk. An acute aggregate risk assessment takes into
account exposure estimates from acute dietary consumption of food and
drinking water. Using the exposure assumptions discussed in this unit
for acute exposure, the acute dietary exposure from food and water to
ipconazole will occupy <1% of the aPAD for females 13-49 years old, the
population group receiving the greatest exposure.
2. Chronic risk. Using the exposure assumptions described in this
unit for chronic exposure, EPA has concluded that chronic exposure to
ipconazole from food and water will utilize <1% of the cPAD for
children 1-2 years old the population group receiving the greatest
exposure. There are no residential uses for ipconazole.
3. Short-term risk. Short-term aggregate exposure takes into
account short-term residential exposure plus chronic exposure to food
and water (considered to be a background exposure level).
A short-term adverse effect was identified; however, ipconazole is
not registered for any use patterns that would result in short-term
residential exposure. Because there is no short-term residential
exposure and chronic dietary exposure has already been assessed under
the appropriately protective cPAD (which is at least as protective as
the POD used to assess short-term risk), no further assessment of
short-term risk is necessary, and EPA relies on the chronic dietary
risk assessment for evaluating short-term risk for ipconazole.
4. Intermediate-term risk. Intermediate-term aggregate exposure
takes into account intermediate-term residential exposure plus chronic
exposure to food and water (considered to be a background exposure
level).
An intermediate-term adverse effect was identified; however,
ipconazole is not registered for any use patterns that would result in
intermediate-term residential exposure. Because there is no
intermediate-term residential exposure and chronic dietary exposure has
already been assessed under the appropriately protective cPAD (which is
at least as protective as the POD used to assess intermediate-term
risk), no further assessment of intermediate-term risk is necessary,
and EPA relies on the chronic dietary risk assessment for evaluating
intermediate-term risk for ipconazole.
5. Aggregate cancer risk for U.S. population. Ipconazole has been
classified as not likely to be carcinogenic, and is not expected to
pose a cancer risk to humans.
6. Determination of safety. Based on these risk assessments, EPA
concludes that there is a reasonable certainty that no harm will result
to the general population, or to infants and children from aggregate
exposure to ipconazole residues.
IV. Other Considerations
A. Analytical Enforcement Methodology
Adequate enforcement methodology (liquid chromatography/mass
spectrometry/mass spectrometry (LC/MS/MS) (AC/3020)) is available to
enforce the tolerance expression. The method may be requested from:
Chief, Analytical Chemistry Branch, Environmental Science Center, 701
Mapes Rd., Ft. Meade, MD 20755-5350; telephone number: (410) 305-2905;
email address: residuemethods@epa.gov.
B. International Residue Limits
In making its tolerance decisions, EPA seeks to harmonize U.S.
tolerances with international standards whenever possible, consistent
with U.S. food safety standards and agricultural practices. EPA
considers the international maximum residue limits (MRLs) established
by the Codex Alimentarius Commission (Codex), as required by FFDCA
section 408(b)(4). The Codex Alimentarius is a joint United Nations
Food and Agriculture Organization/World Health Organization food
standards program, and it is recognized as an international food safety
standards-setting organization in trade agreements to which the United
States is a party. EPA may establish a tolerance that is different from
a Codex MRL; however, FFDCA section 408(b)(4) requires that EPA explain
the reasons for departing from the Codex level.
The Codex has not established a MRL for ipconazole on vegetable,
legume, group 6.
C. Revisions to Petitioned-for Tolerances
The petitioner requested a tolerance for residues of ipconazole in
or on ``legume vegetables succulent or dried, crop group 6''. EPA is
correcting the commodity term and establishing a tolerance for
``vegetable, legume, group 6''.
V. Conclusion
Therefore, tolerances are established for residues of ipconazole
(2-[(4-chlorophenyl)methyl]-5-(1-
[[Page 15240]]
methylethyl)-1-(1H-1,2,4-triazol-1-ylmethyl)cyclopentanol) in or on
vegetable, legume, group 6 at 0.01 ppm. EPA is revising the tolerance
expression for ipconazole to clarify that metabolites and degradates
are covered by the tolerances and to specify how compliance with the
tolerances is to be measured. The existing tolerances for pea and bean,
dried shelled, except soybean, subgroup 6C at 0.01 ppm and soybean,
seed at 0.01 ppm will be removed from paragraph (a) of Sec. 180.646 as
these tolerances are encompassed within vegetable, legume, group 6.
VI. Statutory and Executive Order Reviews
This final rule establishes tolerances under FFDCA section 408(d)
in response to a petition submitted to the Agency. The Office of
Management and Budget (OMB) has exempted these types of actions from
review under Executive Order 12866, entitled ``Regulatory Planning and
Review'' (58 FR 51735, October 4, 1993). Because this final rule has
been exempted from review under Executive Order 12866, this final rule
is not subject to Executive Order 13211, entitled ``Actions Concerning
Regulations That Significantly Affect Energy Supply, Distribution, or
Use'' (66 FR 28355, May 22, 2001) or Executive Order 13045, entitled
``Protection of Children from Environmental Health Risks and Safety
Risks'' (62 FR 19885, April 23, 1997). This final rule does not contain
any information collections subject to OMB approval under the Paperwork
Reduction Act (PRA) (44 U.S.C. 3501 et seq.), nor does it require any
special considerations under Executive Order 12898, entitled ``Federal
Actions to Address Environmental Justice in Minority Populations and
Low-Income Populations'' (59 FR 7629, February 16, 1994).
Since tolerances and exemptions that are established on the basis
of a petition under FFDCA section 408(d), such as the tolerances in
this final rule, do not require the issuance of a proposed rule, the
requirements of the Regulatory Flexibility Act (RFA) (5 U.S.C. 601 et
seq.), do not apply.
This final rule directly regulates growers, food processors, food
handlers, and food retailers, not States or tribes, nor does this
action alter the relationships or distribution of power and
responsibilities established by Congress in the preemption provisions
of FFDCA section 408(n)(4). As such, the Agency has determined that
this action will not have a substantial direct effect on States or
tribal governments, on the relationship between the national government
and the States or tribal governments, or on the distribution of power
and responsibilities among the various levels of government or between
the Federal Government and Indian Tribes. Thus, the Agency has
determined that Executive Order 13132, entitled ``Federalism'' (64 FR
43255, August 10, 1999) and Executive Order 13175, entitled
``Consultation and Coordination with Indian Tribal Governments'' (65 FR
67249, November 9, 2000) do not apply to this final rule. In addition,
this final rule does not impose any enforceable duty or contain any
unfunded mandate as described under Title II of the Unfunded Mandates
Reform Act of 1995 (UMRA) (2 U.S.C. 1501 et seq.).
This action does not involve any technical standards that would
require Agency consideration of voluntary consensus standards pursuant
to section 12(d) of the National Technology Transfer and Advancement
Act of 1995 (NTTAA) (15 U.S.C. 272 note).
VII. Congressional Review Act
Pursuant to the Congressional Review Act (5 U.S.C. 801 et seq.),
EPA will submit a report containing this rule and other required
information to the U.S. Senate, the U.S. House of Representatives, and
the Comptroller General of the United States prior to publication of
the rule in the Federal Register. This action is not a ``major rule''
as defined by 5 U.S.C. 804(2).
List of Subjects in 40 CFR Part 180
Environmental protection, Administrative practice and procedure,
Agricultural commodities, Pesticides and pests, Reporting and
recordkeeping requirements.
Dated: March 12, 2014.
Lois Rossi,
Director, Registration Division, Office of Pesticide Programs.
Therefore, 40 CFR chapter I is amended as follows:
PART 180--[AMENDED]
0
1. The authority citation for part 180 continues to read as follows:
Authority: 21 U.S.C. 321(q), 346a and 371.
0
2. In Sec. 180.646:
0
a. Revise the introductory text in paragraph (a).
0
b. Remove ``Pea and bean, dried shelled, except soybean, subgroup 6C'',
and ``Soybean, seed'' from the table in paragraph (a).
0
c. Add alphabetically ``Vegetable, legume, group 6'' to the table in
paragraph (a).
The amendments read as follows:
Sec. 180.646 Ipconazole; tolerances for residues.
(a) General. Tolerances are established for residues of ipconazole,
including its metabolites and degradates, in or on the commodities
listed in the table below. Compliance with the tolerance levels
specified below is to be determined by measuring only ipconazole (2-
[(4-chlorophenyl)methyl]-5-(1-methylethyl)-1-(1H-1,2,4-triazol-1-
ylmethyl)cyclopentanol) in or on the commodity.
------------------------------------------------------------------------
Parts per
Commodity million
------------------------------------------------------------------------
* * * * *
Vegetable, legume, group 6.............................. 0.01
------------------------------------------------------------------------
* * * * *
[FR Doc. 2014-06059 Filed 3-18-14; 8:45 am]
BILLING CODE 6560-50-P