Government-Owned Inventions; Availability for Licensing, 31859-31861 [2012-13007]
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Federal Register / Vol. 77, No. 104 / Wednesday, May 30, 2012 / Notices
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IV. Electronic Access
Persons with access to the Internet
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Dated: May 15, 2012.
Leslie Kux,
Assistant Commissioner for Policy.
[FR Doc. 2012–12928 Filed 5–29–12; 8:45 am]
BILLING CODE 4160–01–P
DEPARTMENT OF HEALTH AND
HUMAN SERVICES
National Institutes of Health
Government-Owned Inventions;
Availability for Licensing
National Institutes of Health,
Public Health Service, HHS.
ACTION: Notice.
AGENCY:
The inventions listed below
are owned by an agency of the U.S.
Government and are available for
licensing in the U.S. in accordance with
35 U.S.C. 207 to achieve expeditious
commercialization of results of
federally-funded research and
development. Foreign patent
applications are filed on selected
inventions to extend market coverage
for companies and may also be available
for licensing.
FOR FURTHER INFORMATION CONTACT:
Licensing information and copies of the
U.S. patent applications listed below
may be obtained by writing to the
indicated licensing contact at the Office
of Technology Transfer, National
Institutes of Health, 6011 Executive
Boulevard, Suite 325, Rockville,
Maryland 20852–3804; telephone: 301–
496–7057; fax: 301–402–0220. A signed
Confidential Disclosure Agreement will
be required to receive copies of the
patent applications.
srobinson on DSK4SPTVN1PROD with NOTICES
SUMMARY:
Rabbit Polyclonal Antibody To Detect a
Pro-Peptide Fragment of NSAIDActivated Gene (NAG–1)/GDF15, a
Protein Associated With Cancer
Description of Technology: Chronic
inflammation is clearly associated with
an increase in the risk of cancer. Nonsteroidal anti-inflammatory drugs
(NSAIDs) are well documented as agents
that inhibit tumor growth and with
long-term use can prevent tumor
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17:58 May 29, 2012
Jkt 226001
development. NSAID-activated gene
(NAG–1), a unique member of the TGFbeta superfamily, is highly induced by
NSAIDs and numerous drugs and
chemicals with anti-tumorigenic
activities.
The protein product of NAG–1 is first
formed into an immature peptide dimer
that must be cut at a specific site before
it can be secreted as a mature protein.
Currently available antibodies can only
detect either the immature form of
NAG–1 or the secreted mature protein,
but do not recognize the peptide
fragment that remains when the
immature dimer is cut to form the
mature protein. Now available for the
first time, the present new antibody
recognizes this NAG–1 pro-peptide
fragment.
Potential Commercial Applications:
As a research tool to detect expression
of the NAG–1/GDF15 cleavage fragment
in cells and media from cultured cells.
Competitive Advantages: No other
antibody is currently available to detect
the NAG–1/GDF15 pro-peptide
fragment.
Development Stage: In vitro data
available
Inventor: Thomas Eling (NIEHS)
Intellectual Property: HHS Reference
No. E–177–2012/0—Research Tool.
Patent protection is not being pursued
for this technology.
Related Technology: HHS Reference
No. E–093–2011/0—Transgenic mice
expressing human GDF15/Nag-1/Mic-1
Licensing Contact: Patrick McCue,
Ph.D.; 301–435–5560;
mccuepat@mail.nih.gov.
Collaborative Research Opportunity:
The NIEHS is seeking statements of
capability or interest from parties
interested in collaborative research to
further develop, evaluate or
commercialize this antibody. For
collaboration opportunities, please
contact Elizabeth M. Denholm, Ph.D. at
denholme@niehs.nih.gov.
Software for Automated Determination
of Macromolecular Structure Using
Cryo-Electron Microscopy
Description of Technology: Available
for licensing is software for automated
generation of density maps of
macromolecular structures from series
of 2D digital micrographs of frozen
hydrated specimens collected using an
electron microscope equipped with an
ultra-cooled computerized stage. Series
of images of biological specimens
collected at different tilt angles relative
to the electron beam are aligned to
compensate for mechanical errors of the
stage and combined to obtain 3D images
(tomograms). Sub volumes containing a
single macromolecular complex can be
PO 00000
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Fmt 4703
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31859
extracted from the 3D image of a protein
solution, or suspension of viruses or
cells. These individual sub-volumes of
identical structures are aligned and
averaged together to generate a density
map of the macromolecular complex of
interest.
Potential Commercial Applications:
• Macromolecular imaging
• Molecular interaction
• Molecular structure and reactivity
Competitive Advantages:
• Noise processing
• Algorithmic averaging
Development Stage: Prototype
Inventors: Mario Juan Borgnia,
Alberto Bartesaghi, Sriram
Subramaniam (all of NCI)
Publications:
1. Amat F, et al. Markov random field
based automatic image alignment for
electron tomography. J Struct Biol. 2008
Mar;161(3):260–75. [PMID 17855124]
2. Kremer JR, et al. Computer
visualization of three-dimensional
image data using IMOD. J Struct Biol.
1996 Jan–Feb;116(1):71–76. [PMID
8742726]
3. Mastronarde DN. Dual-axis
tomography: an approach with
alignment methods that preserve
resolution. J Struct Biol. 1997
Dec;120(3):343–52. [PMID 9441937]
4. Bartesaghi A, et al. An energy-based
three-dimensional segmentation
approach for the quantitative
interpretation of electron tomograms.
IEEE Trans Image Process. 2005
Sep;14(9):1314–23. [PMID 16190467]
Intellectual Property: HHS Reference
No. E–162–2012/0—Research Tool.
Patent protection is not being pursued
for this technology.
Licensing Contact: Michael
Shmilovich; 301–435–5019;
mish@codon.nih.gov.
Collaborative Research Opportunity:
The NCI Laboratory of Cell Biology is
seeking statements of capability or
interest from parties interested in
collaborative research to further
develop, evaluate or commercialize this
technology. For collaboration
opportunities, please contact John
Hewes, Ph.D. at hewesj@mail.nih.gov.
Chimeric Antigen Receptors That
Recognize Mesothelin for Cancer
Immunotherapy
Description of Technology: Scientists
at the National Institutes of Health (NIH)
have developed chimeric antigen
receptors (CARs) with high affinity for
mesothelin to use as a promising
immunotherapy to treat cancers, such as
pancreatic cancer, ovarian cancer, and
mesothelioma. Mesothelin is a protein
cancer antigen with limited expression
on normal cells that is overexpressed by
E:\FR\FM\30MYN1.SGM
30MYN1
srobinson on DSK4SPTVN1PROD with NOTICES
31860
Federal Register / Vol. 77, No. 104 / Wednesday, May 30, 2012 / Notices
cancer cells. CARs are hybrid proteins
consisting of an antibody portion that
recognizes a cancer antigen, such as a
mesothelin-specific antibody, fused to
receptor signaling domains that serve to
activate the CAR-expressing cell to kill
tumor cells. Cells that express CARs,
most notably T cells, are highly reactive
against their specific tumor antigen in
an MHC-unrestricted manner to
generate an immune response that
promotes robust tumor cell elimination
when infused into cancer patients. The
instant technology includes CAR
constructs with one of three different
mesothelin-specific antibody portions,
including either the mouse-derived SS
or SS1 antibody fragments or the human
HN1 antibody fragment. Infusion of
cells expressing these mesothelinspecific CARs into patients could prove
to be a powerful new
immunotherapeutic tool for treating
various cancers that express mesothelin.
Potential Commercial Applications:
• Immunotherapeutics to treat and/or
prevent the reoccurrence of cancers that
overexpress mesothelin, including
pancreatic cancer, ovarian cancer, and
mesothelioma and other cancers with
few effective treatment options.
• A personalized cancer treatment
strategy for patients whose tumor cells
express mesothelin whereby the
patient’s own T cells are isolated,
engineered to express a mesothelinspecific CAR, and re-infused into the
body to attack the tumor(s).
• Tools to diagnose the presence of
mesothelin-expressing tumors in
patients.
Competitive Advantages:
• Minimal side effects: Mesothelin is
overexpressed on tumor cells. CARs
specific for the mesothelin antigen they
are expected to primarily target tumor
cells, and thus, generate fewer side
effects than other cancer treatment
approaches.
• Successful track record:
Immunotoxins containing the antibody
portions of some of these CARs have
shown promising results in clinical
studies for cancer treatment.
• Cutting edge: With the advent of
Provenge(R) and Yervoy(R),
immunotherapy is now more widely
accepted as a viable cancer treatment
option.
Development Stage:
• Pre-clinical.
• Clinical.
• In vitro data available.
Inventors: Steven A. Feldman, Steven
A. Rosenberg, Ira Pastan (all of NCI).
Intellectual Property: HHS Reference
No. E–078–2012/0—U.S. Patent
Application No. 61/614,612 filed 23 Mar
2012.
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Related Technologies:
• HHS Reference No. E–002–1996/1.
• HHS Reference No. E–021–1998/0.
• HHS Reference No. E–139–1999/0.
• HHS Reference No. E–091–2009/0.
• HHS Reference Nos. E–093–1995/
1,/2.
Licensing Contact: Samuel E. Bish,
Ph.D.; 301–435–5282;
bishse@mail.nih.gov.
Low-dose Cardiac Computed
Tomography Method for Whole Heart
Extracellular Volume
Description of Technology:
Myocardial infarction and
cardiomyopathies result in myocardial
scar and diffuse fibrosis. Together these
result in poor cardiac function.
Myocardial scar is a specific target for
therapy, but is difficult to identify.
Cardiac Computed Tomography (CCT)
struggles to identify large scars, and
could not previously identify fibrosis.
MRI is often used, but MRI is expensive
and not widely available. We have
developed a method to quantify both
diffuse and focal myocardial scar by
CCT using low radiation dose methods.
Extracellular volume fraction (ECV) is
the distribution of iodine in the scar
relative to blood pool. ECV is
abnormally elevated in scar. The new
CCT technique involves (a) CCT data
about the myocardium and blood pool
is extracted (via a shape constrained
graph cut technique), (b) an algorithm
(Demons deformable registration) is
applied to pre-contrast and low dose
post-contrast image information, (c) the
ECV value is computed. Along with
coronary artery depiction on CCT, the
ECV can be used to quantitatively
measure myocardial scar and diffuse
myocardial fibrosis for a complete
depiction of the patient’s myocardial
status/health.
Potential Commercial Applications:
Medical imaging
Competitive Advantages: Cardiac
Computed Tomography is faster, more
widely available and comparatively
inexpensive versus Cardiac Magnetic
Resonance Imaging.
Development Stage:
• Prototype
• Pre-clinical
• Clinical
Inventors: David Bluemke, Songtao
Liu, Marcelo N. Nacif, Jianhua Yao,
Christopher T. Sibley, Xinjian Chen,
Ronald M. Summers (all of NIHCC)
Intellectual Property: HHS Reference
No. E–267–2011/0
Licensing Contact: Tedd Fenn; 301–
435–5031; Tedd.Fenn@nih.gov
Collaborative Research Opportunity:
The NIH Clinical Center is seeking
statements of capability or interest from
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parties interested in collaborative
research to further develop, evaluate or
commercialize Cardiac CT, Cardiac
CTA, myocardial scar, myocardial
fibrosis, coronary artery disease
imaging. For collaboration
opportunities, please contact Ken Rose,
Ph.D. at rosek@mail.nih.gov.
Quantitative in Vivo Methods To
Estimate the Conduction Time of Nerve
Impulses in the Brain
Description of Technology: The axon
diameter distribution (ADD) is an
important anatomical feature of nerve
fascicles both in normal and abnormal
development. Axon diameter directly
affects nerve function. It is well known
that in myelinated axons, the
conduction velocity is directly
proportional to axon diameter.
Moreover, it is hypothesized that in
amyotrophic lateral sclerosis (ALS) large
diameter axons are damaged selectively,
while in autism, small-diameter axons
are over-expressed. Despite its
importance, the ADD within nerve
fascicles has not been measurable invivo, and currently can only be assessed
by invasive histological means.
Previously, the NICHD inventors
developed magnetic resonance imaging
(MRI) methods to measure the ADD
within nerve fascicles (e.g., by
AxCaliber MRI). This invention extends
from the inventor’s prior work to
AxCaliber MRI along with the noninvasive measurement of the arc-length
of a nerve pathway (e.g., using DTI
tractography), to estimate the mean
conduction time of nerve impulses
along that pathway, as well as other
statistical moments of the conduction
time distribution. This method could be
used to diagnose abnormalities in nerve
conduction in brain regions and
providing a neuroanatomical basis for
many cognitive and behavior disorders.
Potential Commercial Applications:
• Used to diagnose abnormalities in
nerve conduction in brain regions
• Provides a neuroanatomical basis
for many cognitive and behavior
disorders
• A basic tool in neuroscience
research to explore the dynamic
functioning of the brain
Competitive Advantages:
• Diagnose a number of cognitive and
behavioral abnormalities, disease and
disorders [currently only assessed using
psychological or psychiatric testing].
• A new quantitative imaging
biomarker
• Used to understand and follow
brain changes during normal aging and
in Alzheimer’s disease.
• Used to explain motor deficits in
ALS disease.
E:\FR\FM\30MYN1.SGM
30MYN1
Federal Register / Vol. 77, No. 104 / Wednesday, May 30, 2012 / Notices
srobinson on DSK4SPTVN1PROD with NOTICES
• Provides way of classifying and
understanding various neurological and
neuropsychiatric conditions according
to conduction delays.
Development Stage:
• Prototype
• Clinical
• In vivo data available (animal)
• In vivo data available (human)
Inventor: Peter J. Basser (NICHD)
Publications:
1. Assaf Y, et al. Ax-Caliber: a method
for measuring axon diameter
distribution from diffusion MRI. Magn
Reson Med. 2008 Jun;59(6):1347–54.
[PMID 18506799]
2. Barazany D, et al. In vivo
measurement of axon diameter
distribution in the corpus callosum of
rat brain. Brain 2009 May;132(Pt
5):1210–20. [PMID 19403788]
Intellectual Property: HHS Reference
No. E–226–2010/0—U.S. Provisional
Application No. 61/535,851 filed 16 Sep
2011
Related Technology: HHS Reference
No. E–079–2003/1—U.S. Patent
Application No. 12/114,713 filed 02
May 2008
Licensing Contact: John Stansberry,
Ph.D.; 301–435–5236;
stansbej@mail.nih.gov.
Collaborative Research Opportunity:
The NICHD is seeking statements of
capability or interest from parties
interested in collaborative research to
further develop, evaluate or
commercialize methods to estimate
conduction time of nerve impulses in
brain. For collaboration opportunities,
please contact Charlotte McGuinness at
mcguinnc@mail.nih.gov.
technology uses a novel synthetic gene
(VOVO) in a highly sensitive, specific
and high-throughput Luciferase
Immunoprecipitation Systems (LIPS)
format. LIPS screening using VOVO
offers an efficient and qualitative
approach for serological screening of
antibodies in Lyme disease in human
and veterinary applications.
Potential Commercial Applications:
Diagnostic for Lyme disease in human
and veterinary applications.
Competitive Advantages: Higher
efficiencies, High-throughput Format
Qualitative
Development Stage:
• Early-stage
• Pre-clinical
Inventors: Peter D. Burbelo (NIDCR),
Michael J. Iadarola (NIDCR), Adriana
Marques (NIAID)
Publication: Burbelo PD, et al. Simple,
quantitative, and highly sensitive
antibody detection for Lyme disease.
Clin Vaccine Immunol. 2010
Jun;17(6):904–9. [PMID: 20392886]
Intellectual Property: HHS Reference
No. E–036–2010/1—PCT application
PCT/US2011/027888 filed 10 Mar 2011
Licensing Contact: Tedd Fenn; 301–
435–5031; Tedd.Fenn@nih.gov
Collaborative Research Opportunity:
The NIDCR, Laboratory of Sensory
Biology, Neurobiology and Pain
Therapeutics Section, is seeking
statements of capability or interest from
parties interested in collaborative
research to further develop, evaluate or
commercialize this technology. For
collaboration opportunities, please
contact David Bradley, Ph.D. at
bradleyda@nidcr.nih.gov.
Simple, Quantitative Sensitive HighThroughput Antibody Detection for
Lyme Disease
Description of Technology: This
technology is for compositions and
methods for diagnosis of Lyme disease.
Currently, Lyme disease is diagnosed by
clinical exam and a history of exposure
to endemic regions. Although,
laboratory tests may aid diagnosis, the
best tests currently available are slow
and labor intensive and require
understanding of the test, and infection
stage. A two-step antibody based test
process is currently the recommended
laboratory test. The first step is either an
enzyme immunoassay (EIA), or an
indirect immunofluorescence assay
(IFA). If the first step is positive, a
‘‘Western blot’’ test is then performed.
Because early intervention is critical to
prevent neurological, rheumatological
and cardiac damage from advanced
infection, more sensitive, specific,
simpler, high-throughput format
laboratory diagnostics are needed. This
Dated: May 23, 2012.
Richard U. Rodriguez,
Director, Division of Technology Development
and Transfer, Office of Technology Transfer,
National Institutes of Health.
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Jkt 226001
[FR Doc. 2012–13007 Filed 5–29–12; 8:45 am]
BILLING CODE 4140–01–P
DEPARTMENT OF HEALTH AND
HUMAN SERVICES
National Institutes of Health
Center for Scientific Review; Notice of
Closed Meetings
Pursuant to section 10(d) of the
Federal Advisory Committee Act, as
amended (5 U.S.C. App.), notice is
hereby given of the following meetings.
The meetings will be closed to the
public in accordance with the
provisions set forth in sections
552b(c)(4) and 552b(c)(6), Title 5 U.S.C.,
as amended. The grant applications and
the discussions could disclose
PO 00000
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31861
confidential trade secrets or commercial
property such as patentable material,
and personal information concerning
individuals associated with the grant
applications, the disclosure of which
would constitute a clearly unwarranted
invasion of personal privacy.
Name of Committee: Center for Scientific
Review Special Emphasis Panel; Member
Conflict: Neurodevelopment and Metabolism.
Date: June 14, 2012.
Time: 1:00 p.m. to 3:00 p.m.
Agenda: To review and evaluate grant
applications.
Place: National Institutes of Health, 6701
Rockledge Drive, Bethesda, MD 20892
(Telephone Conference Call).
Contact Person: Laurent Taupenot, Ph.D.,
Scientific Review Officer, Center for
Scientific Review, National Institutes of
Health, 6701 Rockledge Drive, Room 4811,
MSC 7850, Bethesda, MD 20892, 301–435–
1203, taupenol@csr.nih.gov.
Name of Committee: Center for Scientific
Review Special Emphasis Panel; Small
Business Grant Applications: Immunology
Date: June 18, 2012.
Time: 8:00 a.m. to 6:00 p.m.
Agenda: To review and evaluate grant
applications.
Place: The River Inn, 924 25th Street NW.,
Washington, DC 20037.
Contact Person: Stephen M. Nigida, Ph.D.,
Scientific Review Officer, Center for
Scientific Review, National Institutes of
Health, 6701 Rockledge Drive, Room 4212,
MSC 7812, Bethesda, MD 20892, 301–435–
1222, nigidas@csr.nih.gov.
Name of Committee: Center for Scientific
Review Special Emphasis Panel; PAR 10–
018: Accelerating the Pace of Drug Abuse
Research Using Existing Epidemiology,
Prevention, and Treatment Research Data.
Date: June 19, 2012.
Time: 2:00 p.m. to 5:00 p.m.
Agenda: To review and evaluate grant
applications.
Place: National Institutes of Health, 6701
Rockledge Drive, Bethesda, MD 20892
(Telephone Conference Call).
Contact Person: George Vogler, Ph.D.,
Scientific Review Officer, PSE IRG, Center for
Scientific Review, National Institutes of
Health, 6701 Rockledge Drive, Room 3140,
Bethesda, MD 20892, 301–435–0694,
voglergp@csr.nih.gov.
Name of Committee: Center for Scientific
Review Special Emphasis Panel; Member
Conflict: Teen Relationship Violence.
Date: June 20, 2012.
Time: 2:00 p.m. to 3:00 p.m.
Agenda: To review and evaluate grant
applications.
Place: National Institutes of Health, 6701
Rockledge Drive, Bethesda, MD 20892,
(Telephone Conference Call).
Contact Person: Monica Basco, Ph.D.,
Scientific Review Officer, Center for
Scientific Review, National Institutes of
Health, 6701 Rockledge Drive, Room 3220,
MSC 7808, Bethesda, MD 20892, 301–496–
7010, bascoma@mail.nih.gov.
Name of Committee: Center for Scientific
Review Special Emphasis Panel; Clinical and
E:\FR\FM\30MYN1.SGM
30MYN1
Agencies
[Federal Register Volume 77, Number 104 (Wednesday, May 30, 2012)]
[Notices]
[Pages 31859-31861]
From the Federal Register Online via the Government Printing Office [www.gpo.gov]
[FR Doc No: 2012-13007]
-----------------------------------------------------------------------
DEPARTMENT OF HEALTH AND HUMAN SERVICES
National Institutes of Health
Government-Owned Inventions; Availability for Licensing
AGENCY: National Institutes of Health, Public Health Service, HHS.
ACTION: Notice.
-----------------------------------------------------------------------
SUMMARY: The inventions listed below are owned by an agency of the U.S.
Government and are available for licensing in the U.S. in accordance
with 35 U.S.C. 207 to achieve expeditious commercialization of results
of federally-funded research and development. Foreign patent
applications are filed on selected inventions to extend market coverage
for companies and may also be available for licensing.
FOR FURTHER INFORMATION CONTACT: Licensing information and copies of
the U.S. patent applications listed below may be obtained by writing to
the indicated licensing contact at the Office of Technology Transfer,
National Institutes of Health, 6011 Executive Boulevard, Suite 325,
Rockville, Maryland 20852-3804; telephone: 301-496-7057; fax: 301-402-
0220. A signed Confidential Disclosure Agreement will be required to
receive copies of the patent applications.
Rabbit Polyclonal Antibody To Detect a Pro-Peptide Fragment of NSAID-
Activated Gene (NAG-1)/GDF15, a Protein Associated With Cancer
Description of Technology: Chronic inflammation is clearly
associated with an increase in the risk of cancer. Non-steroidal anti-
inflammatory drugs (NSAIDs) are well documented as agents that inhibit
tumor growth and with long-term use can prevent tumor development.
NSAID-activated gene (NAG-1), a unique member of the TGF-beta
superfamily, is highly induced by NSAIDs and numerous drugs and
chemicals with anti-tumorigenic activities.
The protein product of NAG-1 is first formed into an immature
peptide dimer that must be cut at a specific site before it can be
secreted as a mature protein. Currently available antibodies can only
detect either the immature form of NAG-1 or the secreted mature
protein, but do not recognize the peptide fragment that remains when
the immature dimer is cut to form the mature protein. Now available for
the first time, the present new antibody recognizes this NAG-1 pro-
peptide fragment.
Potential Commercial Applications: As a research tool to detect
expression of the NAG-1/GDF15 cleavage fragment in cells and media from
cultured cells.
Competitive Advantages: No other antibody is currently available to
detect the NAG-1/GDF15 pro-peptide fragment.
Development Stage: In vitro data available
Inventor: Thomas Eling (NIEHS)
Intellectual Property: HHS Reference No. E-177-2012/0--Research
Tool. Patent protection is not being pursued for this technology.
Related Technology: HHS Reference No. E-093-2011/0--Transgenic mice
expressing human GDF15/Nag-1/Mic-1
Licensing Contact: Patrick McCue, Ph.D.; 301-435-5560;
mccuepat@mail.nih.gov.
Collaborative Research Opportunity: The NIEHS is seeking statements
of capability or interest from parties interested in collaborative
research to further develop, evaluate or commercialize this antibody.
For collaboration opportunities, please contact Elizabeth M. Denholm,
Ph.D. at denholme@niehs.nih.gov.
Software for Automated Determination of Macromolecular Structure Using
Cryo-Electron Microscopy
Description of Technology: Available for licensing is software for
automated generation of density maps of macromolecular structures from
series of 2D digital micrographs of frozen hydrated specimens collected
using an electron microscope equipped with an ultra-cooled computerized
stage. Series of images of biological specimens collected at different
tilt angles relative to the electron beam are aligned to compensate for
mechanical errors of the stage and combined to obtain 3D images
(tomograms). Sub volumes containing a single macromolecular complex can
be extracted from the 3D image of a protein solution, or suspension of
viruses or cells. These individual sub-volumes of identical structures
are aligned and averaged together to generate a density map of the
macromolecular complex of interest.
Potential Commercial Applications:
Macromolecular imaging
Molecular interaction
Molecular structure and reactivity
Competitive Advantages:
Noise processing
Algorithmic averaging
Development Stage: Prototype
Inventors: Mario Juan Borgnia, Alberto Bartesaghi, Sriram
Subramaniam (all of NCI)
Publications:
1. Amat F, et al. Markov random field based automatic image
alignment for electron tomography. J Struct Biol. 2008 Mar;161(3):260-
75. [PMID 17855124]
2. Kremer JR, et al. Computer visualization of three-dimensional
image data using IMOD. J Struct Biol. 1996 Jan-Feb;116(1):71-76. [PMID
8742726]
3. Mastronarde DN. Dual-axis tomography: an approach with alignment
methods that preserve resolution. J Struct Biol. 1997 Dec;120(3):343-
52. [PMID 9441937]
4. Bartesaghi A, et al. An energy-based three-dimensional
segmentation approach for the quantitative interpretation of electron
tomograms. IEEE Trans Image Process. 2005 Sep;14(9):1314-23. [PMID
16190467]
Intellectual Property: HHS Reference No. E-162-2012/0--Research
Tool. Patent protection is not being pursued for this technology.
Licensing Contact: Michael Shmilovich; 301-435-5019;
mish@codon.nih.gov.
Collaborative Research Opportunity: The NCI Laboratory of Cell
Biology is seeking statements of capability or interest from parties
interested in collaborative research to further develop, evaluate or
commercialize this technology. For collaboration opportunities, please
contact John Hewes, Ph.D. at hewesj@mail.nih.gov.
Chimeric Antigen Receptors That Recognize Mesothelin for Cancer
Immunotherapy
Description of Technology: Scientists at the National Institutes of
Health (NIH) have developed chimeric antigen receptors (CARs) with high
affinity for mesothelin to use as a promising immunotherapy to treat
cancers, such as pancreatic cancer, ovarian cancer, and mesothelioma.
Mesothelin is a protein cancer antigen with limited expression on
normal cells that is overexpressed by
[[Page 31860]]
cancer cells. CARs are hybrid proteins consisting of an antibody
portion that recognizes a cancer antigen, such as a mesothelin-specific
antibody, fused to receptor signaling domains that serve to activate
the CAR-expressing cell to kill tumor cells. Cells that express CARs,
most notably T cells, are highly reactive against their specific tumor
antigen in an MHC-unrestricted manner to generate an immune response
that promotes robust tumor cell elimination when infused into cancer
patients. The instant technology includes CAR constructs with one of
three different mesothelin-specific antibody portions, including either
the mouse-derived SS or SS1 antibody fragments or the human HN1
antibody fragment. Infusion of cells expressing these mesothelin-
specific CARs into patients could prove to be a powerful new
immunotherapeutic tool for treating various cancers that express
mesothelin.
Potential Commercial Applications:
Immunotherapeutics to treat and/or prevent the
reoccurrence of cancers that overexpress mesothelin, including
pancreatic cancer, ovarian cancer, and mesothelioma and other cancers
with few effective treatment options.
A personalized cancer treatment strategy for patients
whose tumor cells express mesothelin whereby the patient's own T cells
are isolated, engineered to express a mesothelin-specific CAR, and re-
infused into the body to attack the tumor(s).
Tools to diagnose the presence of mesothelin-expressing
tumors in patients.
Competitive Advantages:
Minimal side effects: Mesothelin is overexpressed on tumor
cells. CARs specific for the mesothelin antigen they are expected to
primarily target tumor cells, and thus, generate fewer side effects
than other cancer treatment approaches.
Successful track record: Immunotoxins containing the
antibody portions of some of these CARs have shown promising results in
clinical studies for cancer treatment.
Cutting edge: With the advent of Provenge(R) and
Yervoy(R), immunotherapy is now more widely accepted as a viable cancer
treatment option.
Development Stage:
Pre-clinical.
Clinical.
In vitro data available.
Inventors: Steven A. Feldman, Steven A. Rosenberg, Ira Pastan (all
of NCI).
Intellectual Property: HHS Reference No. E-078-2012/0--U.S. Patent
Application No. 61/614,612 filed 23 Mar 2012.
Related Technologies:
HHS Reference No. E-002-1996/1.
HHS Reference No. E-021-1998/0.
HHS Reference No. E-139-1999/0.
HHS Reference No. E-091-2009/0.
HHS Reference Nos. E-093-1995/1,/2.
Licensing Contact: Samuel E. Bish, Ph.D.; 301-435-5282;
bishse@mail.nih.gov.
Low-dose Cardiac Computed Tomography Method for Whole Heart
Extracellular Volume
Description of Technology: Myocardial infarction and
cardiomyopathies result in myocardial scar and diffuse fibrosis.
Together these result in poor cardiac function. Myocardial scar is a
specific target for therapy, but is difficult to identify. Cardiac
Computed Tomography (CCT) struggles to identify large scars, and could
not previously identify fibrosis. MRI is often used, but MRI is
expensive and not widely available. We have developed a method to
quantify both diffuse and focal myocardial scar by CCT using low
radiation dose methods. Extracellular volume fraction (ECV) is the
distribution of iodine in the scar relative to blood pool. ECV is
abnormally elevated in scar. The new CCT technique involves (a) CCT
data about the myocardium and blood pool is extracted (via a shape
constrained graph cut technique), (b) an algorithm (Demons deformable
registration) is applied to pre-contrast and low dose post-contrast
image information, (c) the ECV value is computed. Along with coronary
artery depiction on CCT, the ECV can be used to quantitatively measure
myocardial scar and diffuse myocardial fibrosis for a complete
depiction of the patient's myocardial status/health.
Potential Commercial Applications: Medical imaging
Competitive Advantages: Cardiac Computed Tomography is faster, more
widely available and comparatively inexpensive versus Cardiac Magnetic
Resonance Imaging.
Development Stage:
Prototype
Pre-clinical
Clinical
Inventors: David Bluemke, Songtao Liu, Marcelo N. Nacif, Jianhua
Yao, Christopher T. Sibley, Xinjian Chen, Ronald M. Summers (all of
NIHCC)
Intellectual Property: HHS Reference No. E-267-2011/0
Licensing Contact: Tedd Fenn; 301-435-5031; Tedd.Fenn@nih.gov
Collaborative Research Opportunity: The NIH Clinical Center is
seeking statements of capability or interest from parties interested in
collaborative research to further develop, evaluate or commercialize
Cardiac CT, Cardiac CTA, myocardial scar, myocardial fibrosis, coronary
artery disease imaging. For collaboration opportunities, please contact
Ken Rose, Ph.D. at rosek@mail.nih.gov.
Quantitative in Vivo Methods To Estimate the Conduction Time of Nerve
Impulses in the Brain
Description of Technology: The axon diameter distribution (ADD) is
an important anatomical feature of nerve fascicles both in normal and
abnormal development. Axon diameter directly affects nerve function. It
is well known that in myelinated axons, the conduction velocity is
directly proportional to axon diameter. Moreover, it is hypothesized
that in amyotrophic lateral sclerosis (ALS) large diameter axons are
damaged selectively, while in autism, small-diameter axons are over-
expressed. Despite its importance, the ADD within nerve fascicles has
not been measurable in-vivo, and currently can only be assessed by
invasive histological means. Previously, the NICHD inventors developed
magnetic resonance imaging (MRI) methods to measure the ADD within
nerve fascicles (e.g., by AxCaliber MRI). This invention extends from
the inventor's prior work to AxCaliber MRI along with the non-invasive
measurement of the arc-length of a nerve pathway (e.g., using DTI
tractography), to estimate the mean conduction time of nerve impulses
along that pathway, as well as other statistical moments of the
conduction time distribution. This method could be used to diagnose
abnormalities in nerve conduction in brain regions and providing a
neuroanatomical basis for many cognitive and behavior disorders.
Potential Commercial Applications:
Used to diagnose abnormalities in nerve conduction in
brain regions
Provides a neuroanatomical basis for many cognitive and
behavior disorders
A basic tool in neuroscience research to explore the
dynamic functioning of the brain
Competitive Advantages:
Diagnose a number of cognitive and behavioral
abnormalities, disease and disorders [currently only assessed using
psychological or psychiatric testing].
A new quantitative imaging biomarker
Used to understand and follow brain changes during normal
aging and in Alzheimer's disease.
Used to explain motor deficits in ALS disease.
[[Page 31861]]
Provides way of classifying and understanding various
neurological and neuropsychiatric conditions according to conduction
delays.
Development Stage:
Prototype
Clinical
In vivo data available (animal)
In vivo data available (human)
Inventor: Peter J. Basser (NICHD)
Publications:
1. Assaf Y, et al. Ax-Caliber: a method for measuring axon diameter
distribution from diffusion MRI. Magn Reson Med. 2008 Jun;59(6):1347-
54. [PMID 18506799]
2. Barazany D, et al. In vivo measurement of axon diameter
distribution in the corpus callosum of rat brain. Brain 2009 May;132(Pt
5):1210-20. [PMID 19403788]
Intellectual Property: HHS Reference No. E-226-2010/0--U.S.
Provisional Application No. 61/535,851 filed 16 Sep 2011
Related Technology: HHS Reference No. E-079-2003/1--U.S. Patent
Application No. 12/114,713 filed 02 May 2008
Licensing Contact: John Stansberry, Ph.D.; 301-435-5236;
stansbej@mail.nih.gov.
Collaborative Research Opportunity: The NICHD is seeking statements
of capability or interest from parties interested in collaborative
research to further develop, evaluate or commercialize methods to
estimate conduction time of nerve impulses in brain. For collaboration
opportunities, please contact Charlotte McGuinness at
mcguinnc@mail.nih.gov.
Simple, Quantitative Sensitive High-Throughput Antibody Detection for
Lyme Disease
Description of Technology: This technology is for compositions and
methods for diagnosis of Lyme disease. Currently, Lyme disease is
diagnosed by clinical exam and a history of exposure to endemic
regions. Although, laboratory tests may aid diagnosis, the best tests
currently available are slow and labor intensive and require
understanding of the test, and infection stage. A two-step antibody
based test process is currently the recommended laboratory test. The
first step is either an enzyme immunoassay (EIA), or an indirect
immunofluorescence assay (IFA). If the first step is positive, a
``Western blot'' test is then performed. Because early intervention is
critical to prevent neurological, rheumatological and cardiac damage
from advanced infection, more sensitive, specific, simpler, high-
throughput format laboratory diagnostics are needed. This technology
uses a novel synthetic gene (VOVO) in a highly sensitive, specific and
high-throughput Luciferase Immunoprecipitation Systems (LIPS) format.
LIPS screening using VOVO offers an efficient and qualitative approach
for serological screening of antibodies in Lyme disease in human and
veterinary applications.
Potential Commercial Applications: Diagnostic for Lyme disease in
human and veterinary applications.
Competitive Advantages: Higher efficiencies, High-throughput Format
Qualitative
Development Stage:
Early-stage
Pre-clinical
Inventors: Peter D. Burbelo (NIDCR), Michael J. Iadarola (NIDCR),
Adriana Marques (NIAID)
Publication: Burbelo PD, et al. Simple, quantitative, and highly
sensitive antibody detection for Lyme disease. Clin Vaccine Immunol.
2010 Jun;17(6):904-9. [PMID: 20392886]
Intellectual Property: HHS Reference No. E-036-2010/1--PCT
application PCT/US2011/027888 filed 10 Mar 2011
Licensing Contact: Tedd Fenn; 301-435-5031; Tedd.Fenn@nih.gov
Collaborative Research Opportunity: The NIDCR, Laboratory of
Sensory Biology, Neurobiology and Pain Therapeutics Section, is seeking
statements of capability or interest from parties interested in
collaborative research to further develop, evaluate or commercialize
this technology. For collaboration opportunities, please contact David
Bradley, Ph.D. at bradleyda@nidcr.nih.gov.
Dated: May 23, 2012.
Richard U. Rodriguez,
Director, Division of Technology Development and Transfer, Office of
Technology Transfer, National Institutes of Health.
[FR Doc. 2012-13007 Filed 5-29-12; 8:45 am]
BILLING CODE 4140-01-P