Draft Guidance for Industry on Pathologic Complete Response in Neoadjuvant Treatment of High-Risk Early-Stage Breast Cancer: Use as an Endpoint To Support Accelerated Approval; Availability, 31858-31859 [2012-12928]
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Federal Register / Vol. 77, No. 104 / Wednesday, May 30, 2012 / Notices
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[FR Doc. 2012–13029 Filed 5–29–12; 8:45 am]
BILLING CODE 4184–01–P
DEPARTMENT OF HEALTH AND
HUMAN SERVICES
Food and Drug Administration
[Docket No. FDA–2012–D–0432]
Draft Guidance for Industry on
Pathologic Complete Response in
Neoadjuvant Treatment of High-Risk
Early-Stage Breast Cancer: Use as an
Endpoint To Support Accelerated
Approval; Availability
AGENCY:
Food and Drug Administration,
HHS.
ACTION:
Notice.
The Food and Drug
Administration (FDA) is announcing the
availability of a draft guidance for
industry entitled ‘‘Pathologic Complete
Response in Neoadjuvant Treatment of
High-Risk Early-Stage Breast Cancer:
Use as an Endpoint to Support
Accelerated Approval.’’ FDA’s
accelerated approval regulations permit
approval of a new drug to treat a serious
disease on the basis of an effect on a
surrogate endpoint reasonably likely to
predict the clinical benefit of the drug.
This draft guidance is intended to assist
applicants in designing trials to support
marketing approval of drugs to treat
breast cancer in the neoadjuvant
(preoperative) setting using pathologic
complete response (pCR) as a surrogate
endpoint that could support approval
under the accelerated approval
regulations. Despite advances in
systemic therapy of early-stage breast
srobinson on DSK4SPTVN1PROD with NOTICES
SUMMARY:
VerDate Mar<15>2010
17:58 May 29, 2012
Jkt 226001
cancer over the past few decades, there
remains a significant unmet medical
need for certain high-risk or poor
prognosis populations of early-stage
breast cancer patients. This guidance is
intended to encourage industry
innovation and expedite the
development of breakthrough therapies
to treat high-risk early-stage breast
cancer.
DATES: Although you can comment on
any guidance at any time (see 21 CFR
10.115(g)(5)), to ensure that the Agency
considers your comment on this draft
guidance before it begins work on the
final version of the guidance, submit
either electronic or written comments
on the draft guidance by July 30, 2012.
ADDRESSES: Submit written requests for
single copies of the draft guidance to the
Division of Drug Information, Center for
Drug Evaluation and Research, Food
and Drug Administration, 10903 New
Hampshire Ave., Bldg. 51, Rm. 2201,
Silver Spring, MD 20993–0002. Send
one self-addressed adhesive label to
assist that office in processing your
requests. See the SUPPLEMENTARY
INFORMATION section for electronic
access to the draft guidance document.
Submit electronic comments on the
draft guidance to https://
www.regulations.gov. Submit written
comments to the Division of Dockets
Management (HFA–305), Food and Drug
Administration, 5630 Fishers Lane, Rm.
1061, Rockville, MD 20852.
FOR FURTHER INFORMATION CONTACT:
Tatiana Prowell, Center for Drug
Evaluation and Research, Food and
Drug Administration, 10903 New
Hampshire Ave., Bldg. 22, Rm. 5249,
Silver Spring, MD 20993–0002, 301–
796–2330.
SUPPLEMENTARY INFORMATION:
I. Background
FDA is announcing the availability of
a draft guidance for industry entitled
‘‘Pathologic Complete Response in
Neoadjuvant Treatment of High-Risk
Early-Stage Breast Cancer: Use as an
Endpoint to Support Accelerated
Approval.’’ Under the accelerated
approval regulations (21 CFR part 314,
subpart H, and 21 CFR part 601, subpart
E), FDA may grant marketing approval
for a new drug on the basis of adequate
and well-controlled trials establishing
that the drug has an effect on a surrogate
endpoint that is reasonably likely to
predict clinical benefit (e.g., an effect on
survival or irreversible morbidity),
provided that the applicant conducts
additional trials after approval to verify
and describe the predicted clinical
benefit. This draft guidance is intended
to assist applicants in designing trials to
PO 00000
Frm 00031
Fmt 4703
Sfmt 4703
support marketing approval of drugs to
treat breast cancer in the neoadjuvant
(preoperative) setting using pCR as a
surrogate endpoint that could support
approval under the accelerated approval
regulations. The guidance proposes a
uniform definition of pCR for regulatory
purposes. The guidance also advises on
appropriate patient populations for
inclusion and on the trial designs
intended to verify the predicted clinical
benefit associated with pCR to support
conversion to full approval.
FDA recognizes that despite advances
in adjuvant systemic therapy of breast
cancer over the past few decades, there
remains a significant unmet medical
need for certain high-risk or poor
prognosis populations of early-stage
breast cancer patients. Developing
highly effective new drugs for these
populations is an FDA priority. In
providing guidance on the use of pCR as
a surrogate endpoint that could support
accelerated approval in the neoadjuvant
setting, FDA hopes to encourage
industry innovation and expedite the
development of breakthrough therapies
to treat high-risk early-stage breast
cancer.
This draft guidance is being issued
consistent with FDA’s good guidance
practices regulation (21 CFR 10.115).
The draft guidance, when finalized, will
represent the Agency’s current thinking
on the use of pCR in neoadjuvant
treatment of high-risk early-stage breast
cancer as an endpoint to support
accelerated approval. It does not create
or confer any rights for or on any person
and does not operate to bind FDA or the
public. An alternative approach may be
used if such approach satisfies the
requirements of the applicable statutes
and regulations.
II. The Paperwork Reduction Act of
1995
This guidance refers to previously
approved collections of information that
are subject to review by the Office of
Management and Budget (OMB) under
the Paperwork Reduction Act of 1995
(44 U.S.C. 3501–3520). The collections
of information in 21 CFR parts 312 and
314 have been approved under OMB
control numbers 0910–0014 and 0910–
0001, respectively. The collections of
information for special protocol
assessments have been approved under
OMB control number 0910–0470.
III. Comments
Interested persons may submit to the
Division of Dockets Management (see
ADDRESSES) either electronic or written
comments regarding this document. It is
only necessary to send one set of
comments. Identify comments with the
E:\FR\FM\30MYN1.SGM
30MYN1
Federal Register / Vol. 77, No. 104 / Wednesday, May 30, 2012 / Notices
docket number found in brackets in the
heading of this document. Received
comments may be seen in the Division
of Dockets Management between 9 a.m.
and 4 p.m., Monday through Friday.
IV. Electronic Access
Persons with access to the Internet
may obtain the document at either
https://www.fda.gov/Drugs/
GuidanceComplianceRegulatory
Information/Guidances/default.htm or
https://www.regulations.gov.
Dated: May 15, 2012.
Leslie Kux,
Assistant Commissioner for Policy.
[FR Doc. 2012–12928 Filed 5–29–12; 8:45 am]
BILLING CODE 4160–01–P
DEPARTMENT OF HEALTH AND
HUMAN SERVICES
National Institutes of Health
Government-Owned Inventions;
Availability for Licensing
National Institutes of Health,
Public Health Service, HHS.
ACTION: Notice.
AGENCY:
The inventions listed below
are owned by an agency of the U.S.
Government and are available for
licensing in the U.S. in accordance with
35 U.S.C. 207 to achieve expeditious
commercialization of results of
federally-funded research and
development. Foreign patent
applications are filed on selected
inventions to extend market coverage
for companies and may also be available
for licensing.
FOR FURTHER INFORMATION CONTACT:
Licensing information and copies of the
U.S. patent applications listed below
may be obtained by writing to the
indicated licensing contact at the Office
of Technology Transfer, National
Institutes of Health, 6011 Executive
Boulevard, Suite 325, Rockville,
Maryland 20852–3804; telephone: 301–
496–7057; fax: 301–402–0220. A signed
Confidential Disclosure Agreement will
be required to receive copies of the
patent applications.
srobinson on DSK4SPTVN1PROD with NOTICES
SUMMARY:
Rabbit Polyclonal Antibody To Detect a
Pro-Peptide Fragment of NSAIDActivated Gene (NAG–1)/GDF15, a
Protein Associated With Cancer
Description of Technology: Chronic
inflammation is clearly associated with
an increase in the risk of cancer. Nonsteroidal anti-inflammatory drugs
(NSAIDs) are well documented as agents
that inhibit tumor growth and with
long-term use can prevent tumor
VerDate Mar<15>2010
17:58 May 29, 2012
Jkt 226001
development. NSAID-activated gene
(NAG–1), a unique member of the TGFbeta superfamily, is highly induced by
NSAIDs and numerous drugs and
chemicals with anti-tumorigenic
activities.
The protein product of NAG–1 is first
formed into an immature peptide dimer
that must be cut at a specific site before
it can be secreted as a mature protein.
Currently available antibodies can only
detect either the immature form of
NAG–1 or the secreted mature protein,
but do not recognize the peptide
fragment that remains when the
immature dimer is cut to form the
mature protein. Now available for the
first time, the present new antibody
recognizes this NAG–1 pro-peptide
fragment.
Potential Commercial Applications:
As a research tool to detect expression
of the NAG–1/GDF15 cleavage fragment
in cells and media from cultured cells.
Competitive Advantages: No other
antibody is currently available to detect
the NAG–1/GDF15 pro-peptide
fragment.
Development Stage: In vitro data
available
Inventor: Thomas Eling (NIEHS)
Intellectual Property: HHS Reference
No. E–177–2012/0—Research Tool.
Patent protection is not being pursued
for this technology.
Related Technology: HHS Reference
No. E–093–2011/0—Transgenic mice
expressing human GDF15/Nag-1/Mic-1
Licensing Contact: Patrick McCue,
Ph.D.; 301–435–5560;
mccuepat@mail.nih.gov.
Collaborative Research Opportunity:
The NIEHS is seeking statements of
capability or interest from parties
interested in collaborative research to
further develop, evaluate or
commercialize this antibody. For
collaboration opportunities, please
contact Elizabeth M. Denholm, Ph.D. at
denholme@niehs.nih.gov.
Software for Automated Determination
of Macromolecular Structure Using
Cryo-Electron Microscopy
Description of Technology: Available
for licensing is software for automated
generation of density maps of
macromolecular structures from series
of 2D digital micrographs of frozen
hydrated specimens collected using an
electron microscope equipped with an
ultra-cooled computerized stage. Series
of images of biological specimens
collected at different tilt angles relative
to the electron beam are aligned to
compensate for mechanical errors of the
stage and combined to obtain 3D images
(tomograms). Sub volumes containing a
single macromolecular complex can be
PO 00000
Frm 00032
Fmt 4703
Sfmt 4703
31859
extracted from the 3D image of a protein
solution, or suspension of viruses or
cells. These individual sub-volumes of
identical structures are aligned and
averaged together to generate a density
map of the macromolecular complex of
interest.
Potential Commercial Applications:
• Macromolecular imaging
• Molecular interaction
• Molecular structure and reactivity
Competitive Advantages:
• Noise processing
• Algorithmic averaging
Development Stage: Prototype
Inventors: Mario Juan Borgnia,
Alberto Bartesaghi, Sriram
Subramaniam (all of NCI)
Publications:
1. Amat F, et al. Markov random field
based automatic image alignment for
electron tomography. J Struct Biol. 2008
Mar;161(3):260–75. [PMID 17855124]
2. Kremer JR, et al. Computer
visualization of three-dimensional
image data using IMOD. J Struct Biol.
1996 Jan–Feb;116(1):71–76. [PMID
8742726]
3. Mastronarde DN. Dual-axis
tomography: an approach with
alignment methods that preserve
resolution. J Struct Biol. 1997
Dec;120(3):343–52. [PMID 9441937]
4. Bartesaghi A, et al. An energy-based
three-dimensional segmentation
approach for the quantitative
interpretation of electron tomograms.
IEEE Trans Image Process. 2005
Sep;14(9):1314–23. [PMID 16190467]
Intellectual Property: HHS Reference
No. E–162–2012/0—Research Tool.
Patent protection is not being pursued
for this technology.
Licensing Contact: Michael
Shmilovich; 301–435–5019;
mish@codon.nih.gov.
Collaborative Research Opportunity:
The NCI Laboratory of Cell Biology is
seeking statements of capability or
interest from parties interested in
collaborative research to further
develop, evaluate or commercialize this
technology. For collaboration
opportunities, please contact John
Hewes, Ph.D. at hewesj@mail.nih.gov.
Chimeric Antigen Receptors That
Recognize Mesothelin for Cancer
Immunotherapy
Description of Technology: Scientists
at the National Institutes of Health (NIH)
have developed chimeric antigen
receptors (CARs) with high affinity for
mesothelin to use as a promising
immunotherapy to treat cancers, such as
pancreatic cancer, ovarian cancer, and
mesothelioma. Mesothelin is a protein
cancer antigen with limited expression
on normal cells that is overexpressed by
E:\FR\FM\30MYN1.SGM
30MYN1
Agencies
[Federal Register Volume 77, Number 104 (Wednesday, May 30, 2012)]
[Notices]
[Pages 31858-31859]
From the Federal Register Online via the Government Printing Office [www.gpo.gov]
[FR Doc No: 2012-12928]
-----------------------------------------------------------------------
DEPARTMENT OF HEALTH AND HUMAN SERVICES
Food and Drug Administration
[Docket No. FDA-2012-D-0432]
Draft Guidance for Industry on Pathologic Complete Response in
Neoadjuvant Treatment of High-Risk Early-Stage Breast Cancer: Use as an
Endpoint To Support Accelerated Approval; Availability
AGENCY: Food and Drug Administration, HHS.
ACTION: Notice.
-----------------------------------------------------------------------
SUMMARY: The Food and Drug Administration (FDA) is announcing the
availability of a draft guidance for industry entitled ``Pathologic
Complete Response in Neoadjuvant Treatment of High-Risk Early-Stage
Breast Cancer: Use as an Endpoint to Support Accelerated Approval.''
FDA's accelerated approval regulations permit approval of a new drug to
treat a serious disease on the basis of an effect on a surrogate
endpoint reasonably likely to predict the clinical benefit of the drug.
This draft guidance is intended to assist applicants in designing
trials to support marketing approval of drugs to treat breast cancer in
the neoadjuvant (preoperative) setting using pathologic complete
response (pCR) as a surrogate endpoint that could support approval
under the accelerated approval regulations. Despite advances in
systemic therapy of early-stage breast cancer over the past few
decades, there remains a significant unmet medical need for certain
high-risk or poor prognosis populations of early-stage breast cancer
patients. This guidance is intended to encourage industry innovation
and expedite the development of breakthrough therapies to treat high-
risk early-stage breast cancer.
DATES: Although you can comment on any guidance at any time (see 21 CFR
10.115(g)(5)), to ensure that the Agency considers your comment on this
draft guidance before it begins work on the final version of the
guidance, submit either electronic or written comments on the draft
guidance by July 30, 2012.
ADDRESSES: Submit written requests for single copies of the draft
guidance to the Division of Drug Information, Center for Drug
Evaluation and Research, Food and Drug Administration, 10903 New
Hampshire Ave., Bldg. 51, Rm. 2201, Silver Spring, MD 20993-0002. Send
one self-addressed adhesive label to assist that office in processing
your requests. See the SUPPLEMENTARY INFORMATION section for electronic
access to the draft guidance document.
Submit electronic comments on the draft guidance to https://www.regulations.gov. Submit written comments to the Division of Dockets
Management (HFA-305), Food and Drug Administration, 5630 Fishers Lane,
Rm. 1061, Rockville, MD 20852.
FOR FURTHER INFORMATION CONTACT: Tatiana Prowell, Center for Drug
Evaluation and Research, Food and Drug Administration, 10903 New
Hampshire Ave., Bldg. 22, Rm. 5249, Silver Spring, MD 20993-0002, 301-
796-2330.
SUPPLEMENTARY INFORMATION:
I. Background
FDA is announcing the availability of a draft guidance for industry
entitled ``Pathologic Complete Response in Neoadjuvant Treatment of
High-Risk Early-Stage Breast Cancer: Use as an Endpoint to Support
Accelerated Approval.'' Under the accelerated approval regulations (21
CFR part 314, subpart H, and 21 CFR part 601, subpart E), FDA may grant
marketing approval for a new drug on the basis of adequate and well-
controlled trials establishing that the drug has an effect on a
surrogate endpoint that is reasonably likely to predict clinical
benefit (e.g., an effect on survival or irreversible morbidity),
provided that the applicant conducts additional trials after approval
to verify and describe the predicted clinical benefit. This draft
guidance is intended to assist applicants in designing trials to
support marketing approval of drugs to treat breast cancer in the
neoadjuvant (preoperative) setting using pCR as a surrogate endpoint
that could support approval under the accelerated approval regulations.
The guidance proposes a uniform definition of pCR for regulatory
purposes. The guidance also advises on appropriate patient populations
for inclusion and on the trial designs intended to verify the predicted
clinical benefit associated with pCR to support conversion to full
approval.
FDA recognizes that despite advances in adjuvant systemic therapy
of breast cancer over the past few decades, there remains a significant
unmet medical need for certain high-risk or poor prognosis populations
of early-stage breast cancer patients. Developing highly effective new
drugs for these populations is an FDA priority. In providing guidance
on the use of pCR as a surrogate endpoint that could support
accelerated approval in the neoadjuvant setting, FDA hopes to encourage
industry innovation and expedite the development of breakthrough
therapies to treat high-risk early-stage breast cancer.
This draft guidance is being issued consistent with FDA's good
guidance practices regulation (21 CFR 10.115). The draft guidance, when
finalized, will represent the Agency's current thinking on the use of
pCR in neoadjuvant treatment of high-risk early-stage breast cancer as
an endpoint to support accelerated approval. It does not create or
confer any rights for or on any person and does not operate to bind FDA
or the public. An alternative approach may be used if such approach
satisfies the requirements of the applicable statutes and regulations.
II. The Paperwork Reduction Act of 1995
This guidance refers to previously approved collections of
information that are subject to review by the Office of Management and
Budget (OMB) under the Paperwork Reduction Act of 1995 (44 U.S.C. 3501-
3520). The collections of information in 21 CFR parts 312 and 314 have
been approved under OMB control numbers 0910-0014 and 0910-0001,
respectively. The collections of information for special protocol
assessments have been approved under OMB control number 0910-0470.
III. Comments
Interested persons may submit to the Division of Dockets Management
(see ADDRESSES) either electronic or written comments regarding this
document. It is only necessary to send one set of comments. Identify
comments with the
[[Page 31859]]
docket number found in brackets in the heading of this document.
Received comments may be seen in the Division of Dockets Management
between 9 a.m. and 4 p.m., Monday through Friday.
IV. Electronic Access
Persons with access to the Internet may obtain the document at
either https://www.fda.gov/Drugs/GuidanceComplianceRegulatoryInformation/Guidances/default.htm or https://www.regulations.gov.
Dated: May 15, 2012.
Leslie Kux,
Assistant Commissioner for Policy.
[FR Doc. 2012-12928 Filed 5-29-12; 8:45 am]
BILLING CODE 4160-01-P