Government-Owned Inventions; Availability for Licensing, 4921-4923 [2011-1671]
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Patent Status: U.S. Provisional
Application No. 61/455,261 filed 14 Oct
2010 (HHS Reference No. E–197–2010/
0–US–01).
Licensing Status: Available for
licensing.
Licensing Contact: Peter A. Soukas,
J.D.; 301–435–4646;
soukasp@mail.nih.gov.
Method for Detection and
Quantification of PLK1 Expression and
Activity
Description of Technology: Polo-like
kinase 1 (Plk1) plays a role in the
regulation of the cell cycle and control
of cellular proliferation. Because Plk1 is
associated with neoplastic
transformation of human cells,
expression of this protein has been
proposed as a prognostic marker for
many types of malignancies. In
mammalian cells, four Plks exist, but
their expression patterns and functions
appear to be distinct from each other.
Available for licensing is a Plk1 ELISA
assay using peptide substrates that are
specific for Plk1, in that they are
phosphorylated and bound by Plk1, but
not by the related polo kinases Plk2,
Plk3 and Plk4.
By exploiting a unique Plk1dependent phosphorylation and binding
property, an easy and reliable ELISA
assay has been developed to quantify
Plk1 expression levels and kinase
activity. With this highly sensitive
assay, Plk1 activity can be measured
with 2–20 microgram of total lysates
without immunoprecipitation or
purification steps. Since deregulated
Plk1 expression has been suggested as a
prognostic marker for a wide range of
human malignancies, this assay may
provide an innovative tool for assessing
the predisposition for cancer
development, monitoring cancer
progression, and estimating the
prognosis of various types of cancer
patients.
Applications:
• Optimized PBIP1 polypeptides, a
natural substrate of Plk1, with enhanced
specificity and sensitivity over the
native PBIP1 sequence.
• ELISA assay to quantify Plk1
expression and kinase activity.
Advantages:
• Rapid, highly sensitive assay that
requires lower amounts of starting
material than conventional
immunoprecipitation assays.
• Assay that is selective for Plk1.
Development Status: The technology
is currently in the pre-clinical stage of
development.
Market:
• Cancer is the second leading cause
of death in United States.
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• An estimated 1,529,560 new cancer
cases and 569,490 deaths from cancer
occurred in the United States in 2010.
• In vitro cancer diagnostic market
will be worth an estimated $8 billion by
the end of 2012.
Inventors: Kyung S. Lee and Jung-Eun
Park (NCI).
Publications:
1. JE Park et al. Direct quantification
of polo-like kinase 1 activity in cells and
tissues using a highly sensitive and
specific ELISA assay. Proc Natl Acad
Sci USA. 2009 Feb 10;106(6):1725–
1730. [PubMed: 19181852]
2. KS Lee et al. Mechanisms of
mammalian polo-like kinase 1 (Plk1)
localization: self-versus non-selfpriming. Cell Cycle 2008 Jan;7(2):141–
145. [PubMed: 18216497]
3. KS Lee et al. Self-regulated
mechanism of Plk1 localization to
kinetochores: lessons from the Plk1–
PBIP1 interaction. Cell Div. 2008 Jan
23;3:4. [PubMed: 18215321]
4. YH Kang et al. Self-regulated Plk1
recruitment to kinetochores by the
Plk1–PBIP1 interaction is critical for
proper chromosome segregation. Mol
Cell. 2006 Nov 3;24(3):409–422.
[PubMed: 17081991]
Patent Status: U.S. Patent Application
No. 12/992,887 filed 15 Nov 2010 (HHS
Reference No. E–091–2008/0–US–03).
Licensing Status: Available for
licensing.
Licensing Contact: Jennifer Wong;
301–435–4633; wongje@mail.nih.gov.
Collaborative Research Opportunity:
The National Cancer Institute,
Laboratory of Metabolism, is seeking
statements of capability or interest from
parties interested in collaborative
research to further develop, evaluate, or
commercialize the PLK1 ELISA assay
described above. Please contact John D.
Hewes, Ph.D. at 301–435–3121 or
hewesj@mail.nih.gov for more
information.
only the same unique Ig molecules.
Some (5–10%) of the Igs specifically
bind antigens.
Applications:
• To understand the underlying
process of neoplastic development.
• To identify the genes that control
tumor susceptibility and resistance.
• To investigate the antigen binding
activities of myeloma proteins.
• To study Ig synthesis.
• To classify the various different
classes of Igs (IgG, IgA, IgM).
• As a fusion partner to make
monoclonal antibodies.
Advantages: Provide an unlimited
source of pure monoclonal Ig molecules.
Inventor: Michael Potter (NCI).
Relevant Publications:
1. Potter M, Fahey JL, Pilgrim HI.
Abnormal serum protein and bone
destruction and transmissible mouse
plasma cell neoplasm (multiple
myeloma). Proc Soc Exp Biol Med. 1957
Feb;94(2):327–333.
2. Nathans D, Fahey JL, Potter M. The
formation of myeloma protein by a
mouse plasma cell tumor. J Exp Med.
1958 Jul 1;108(1):121–130. [PubMed:
13549645]
3. Potter M, Boyce CR. Induction of
plasma cell neoplasms in strain BALB/
c mice with mineral oil and mineral oil
adjuvants. Nature. 1962 Mar
17;193:1086–1087.
4. Andersen PN, Potter M. Induction
of plasma cell tumors in BALB/c mice
with 2,6,10,14-tetramethylpentadecane
(pristane). Nature. 1969 Jun
7;222(5197):994–995.
Patent Status: HHS Reference No. E–
277–2001/0—Research Material. Patent
protection is not being pursued for this
technology.
Licensing Status: Available for
biological materials licensing only.
Licensing Contact: Patrick P. McCue,
Ph.D.; 301–435–5560;
mccuepat@mail.nih.gov.
Immunoglobulin-Producing Mouse
Plasmacytomas
Description of Technology: Overall
cancer costs in the U.S. in 2006 are
estimated at $206.3 billion. The World
Health Organization predicts upwards
of 15 million new cancer cases globally
by 2020. There remains a significant
unmet need for new therapies to treat
cancer, as well as a need to further
understand the role of the immune
system in cancer susceptibility.
Available for licensing are isolated
immunoglobulin-producing mouse
plasmacytomas (PCTs). Each tumor
produces only one species of
monoclonal immunoglobulin (Ig). When
transplanted into mice, these plasma
cell tumors will continue to produce
Dated: January 19, 2011.
Richard U. Rodriguez,
Director, Division of Technology Development
and Transfer, Office of Technology Transfer,
National Institutes of Health.
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[FR Doc. 2011–1669 Filed 1–26–11; 8:45 am]
BILLING CODE 4140–01–P
DEPARTMENT OF HEALTH AND
HUMAN SERVICES
National Institutes of Health
Government-Owned Inventions;
Availability for Licensing
National Institutes of Health,
Public Health Service, HHS.
ACTION: Notice.
AGENCY:
E:\FR\FM\27JAN1.SGM
27JAN1
4922
Federal Register / Vol. 76, No. 18 / Thursday, January 27, 2011 / Notices
The inventions listed below
are owned by an agency of the U.S.
Government and are available for
licensing in the U.S. in accordance with
35 U.S.C. 207 to achieve expeditious
commercialization of results of
federally-funded research and
development. Foreign patent
applications are filed on selected
inventions to extend market coverage
for companies and may also be available
for licensing.
ADDRESSES: Licensing information and
copies of the U.S. patent applications
listed below may be obtained by writing
to the indicated licensing contact at the
Office of Technology Transfer, National
Institutes of Health, 6011 Executive
Boulevard, Suite 325, Rockville,
Maryland 20852–3804; telephone: 301/
496–7057; fax: 301/402–0220. A signed
Confidential Disclosure Agreement will
be required to receive copies of the
patent applications.
mstockstill on DSKH9S0YB1PROD with NOTICES
SUMMARY:
A Computer Program To Predict
Optimal Sites on Protein Sequences for
Production of Peptide-Directed
Antibodies (NHLBI AbDesigner)
Description of Technology: The
invention offered for licensing is a
computer program called ‘‘NHLBI
AbDesigner’’ that allows the user to
input a unique identifier for an
individual mammalian protein to be
analyzed in order to find out what short
peptides in its amino sequence would
most likely result in a strong
immunogenic response when injected
into a research animal. The software
displays standard predictors of
immunogenicity and antigenicity in
easy-to-view heat maps and also allows
users to choose peptides most likely to
elicit antibodies that are specific to said
protein. The computer code is written in
Java and would be made available in the
form of jar files.
For additional information please
refer to: https://dirweb.nhlbi.nih.gov/
labs/LKEM_G/LKEM/Pages/
Antibodydesignsoftware.aspx.
Applications:
• Design and production of
antibodies for research or therapeutic
purposes.
• Bioinformatic analysis of protein
structure and functions.
• Analysis and interpretation of
proteomic data.
Advantages: This program allows the
user to identify tradeoffs in the decision
making process by aligning various
types of information with the amino
acid sequence, constituting an
improvement over present ad hoc
methods of accumulating and relating
different type of information regarding
immunogenicity, uniqueness of
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Jkt 223001
sequences, conservation of sequences,
and presence of post-translational
modifications.
Development Status: Fully developed.
Inventors: Mark A. Knepper (NHLBI)
et al.
Patent Status: HHS Reference No.
E–251–2010/0—Software. Patent
protection is not being pursued for this
technology.
Licensing Status: Available for
licensing.
Licensing Contacts:
• Uri Reichman, PhD, MBA; 301–
435–4616; UR7a@nih.gov.
• Michael Shmilovich, Esq.; 301–
435–5019; shmilovm@mail.nih.gov.
Collaborative Research Opportunity:
The NHLBI is seeking statements of
capability or interest from parties
interested in collaborative research to
further develop, evaluate, or
commercialize this technology. Please
contact Brian Bailey, Ph.D. at 301–594–
4094 or bbailey@mail.nih.gov for more
information.
Nanoparticle Probes and Mid-Infrared
Chemical Imaging for DNA Microarray
Detection
Description of Technology: The
technology offered for licensing is a
faster, more flexible, cost-effective
microarray visualization. The invention
describes and claims the mid-infrared
chemical imaging (IRCI) to detect
nanostructure-based DNA microarrays,
which can be utilized in the life science
research arena to examine gene
expression and single nucleotide
polymorphisms (SNPs), as well as to
characterize entire genomes. The IRCI
improves the signal-to-noise ratio (SNR)
obtained for hybridized microarrayed
spots compared to the commonly used
fluorescence detection method. The
improved method of this invention
results in the sensitivity and precision
for detecting pathogenic bacterial genes
and can be utilized to detect lowexpressing genes which cannot be
identified by fluorescent-based DNA
microarrays. Furthermore, the
automated IRCI systems can also be
fabricated for the dedicated detection of
other (protein, tissue, biochemical, or
chemical) microarrays.
Applications: DNA microarrays can
be applied to the areas of environmental
sciences, agriculture research, biodefense, diagnostics, forensics,
pharmacogenomics and toxicogenomics.
Advantages: The invention provides a
cost-effective, faster, more flexible, and
less labor intensive microarray
technology.
Development Status:
• The invention is fully developed.
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• Need to develop a commercialized
kit and protocol.
Inventors: Magdi M. Mossoba, et al.
(FDA).
Patent Status: U.S. Provisional
Application No. 61/395,635 filed 15 Oct
2010 (HHS Reference No. E–127–2010/
0–US–01).
Licensing Status: Available for
licensing.
Licensing Contact: Susan Ano, PhD;
301–435–5515; anos@mail.nih.gov.
Fluoroquinolone Derivatives as
Inhibitors of Human Tyrosyl-DNA
Phosphodiesterase (Tdp1)
Description of Technology:
Chemotherapy can provide therapeutic
benefits in many cancer patients, but it
often ultimately fails to cure the disease
since cancer cells can become resistant
to the chemotherapeutic agent. To
overcome these limitations, additional
strategies are needed to restore or
amplify the effect of antitumor agents.
Tyrosyl-DNA phosphodiesterase 1
(Tdp1) is a DNA repair enzyme involved
in the repair of DNA lesions created
when the activity of the Topoisomerase
1 (Top1) is inhibited. Tdp1 has been
regarded as a potential therapeutic cotarget of Top1 in that it seemingly
counteracts the effects of Top1
inhibitors, such as camptothecin. By
reducing the repair of Top1-DNA
lesions, Tdp1 inhibitors have the
potential to augment the anticancer
activity of Top1 inhibitors.
The NIH investigators discovered
fluoroquinolone derivatives as specific
Tdp1 inhibitors that could potentiate
the pharmacological action of Top1
inhibitors, which are currently used in
cancer treatment. The instant invention
discloses a method of treating cancers
with a therapeutically effective amount
of a Top1 inhibitor, and a
fluoroquinolone derivative that inhibits
Tdp1 activity.
Applications and Market:
• This invention may provide a new
combination of drugs to target various
cancers for treatment.
• Cancer is the second leading cause
of death in the U.S. The National Cancer
Institute estimates the overall annual
costs for cancer in the U.S. at $107
billion; development of more effective
cancer therapies is always in high
demand.
Development Status: Pre-clinical stage
of development.
Inventors: Yves G. Pommier,
Christophe R. Marchand, Thomas S.
Dexheimer (NCI), et al.
Related Publications:
1. Dexheimer TS, Antony S,
Marchand C, Pommier Y. Tyrosyl-DNA
phosphodiesterase as a target for
E:\FR\FM\27JAN1.SGM
27JAN1
Federal Register / Vol. 76, No. 18 / Thursday, January 27, 2011 / Notices
mstockstill on DSKH9S0YB1PROD with NOTICES
anticancer therapy. Anticancer Agents
Med Chem. 2008 May;8(4):381–389.
[PubMed: 18473723]
2. Dexheimer TS, et al. 4-Pregnen-21ol-3,20-dione-21-(4bromobenzenesulfonate) and related
novel steroid derivatives as tyrosyl-DNA
phosphodiesterase (Tdp1) inhibitors. J
Med Chem. 2009 Nov 26;52(22):7122–
7131. [PubMed: 19883083]
3. Marchand C, et al. Identification of
phosphotyrosine mimetic inhibitors of
human tyrosyl-DNA phosphodiesterase
I by a novel AlphaScreen highthroughput assay. Mol Cancer Ther.
2009 Jan;8(1):240–248. [PubMed:
19139134]
Patent Status: U.S. Provisional
Application No. 61/407,325 filed 07 Oct
2010 (HHS Reference No. E–199–2010/
0–US–01).
Licensing Status: Available for
licensing.
Licensing Contact: Betty B. Tong,
PhD; 301–594–6565;
tongb@mail.nih.gov.
Collaborative Research Opportunity:
The Center for Cancer Research,
Laboratory of Molecular Pharmacology,
is seeking statements of capability or
interest from parties interested in
collaborative research to further
develop, evaluate, or commercialize
tyrosyl-DNA-phosphodiesterase
inhibitors. Please contact John Hewes,
PhD at 301–435–3121 or
hewesj@mail.nih.gov for more
information.
HMG3 for Detecting and Treating
Diabetes
Description of Technology: This
invention relates to the use of High
Mobility Group N 3 (HMGN3) as a
marker for detecting diabetes and as a
therapeutic agent for treating diabetes.
Diabetes is disabling largely because
commonly available anti-diabetic drugs
do not adequately control blood sugar
levels to completely prevent the
occurrence of high and low blood sugar
levels. Inappropriate blood sugar levels
can be toxic and can cause long-term
complications including renopathy,
retinopathy, neuropathy and peripheral
vascular disease. Those with diabetes
are also at risk for developing related
conditions such as obesity,
hypertension, heart disease and
hyperlipidemia.
This invention relates to the discovery
that reduced expression of HMGN3 (also
called TRIP7) gives rise to elevated
blood glucose levels, reduced serum
insulin levels and impaired glucose
tolerance.
Applications: Diagnostic and
therapeutic for diabetes.
Development Status: Early stage.
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Inventors: Michael Bustin et al. (NCI).
Related Publication: Ueda T,
Furusawa T, Kurahashi T, Tessarollo L,
Bustin M. The nucleosome binding
protein HMGN3 modulates the
transcription profile of pancreatic beta
cells and affects insulin secretion. Mol
Cell Biol. 2009 Oct;29(19):5264–5276.
[PubMed: 19651901]
Patent Status: PCT Application No.
PCT/US2009/039406 filed 03 Apr 2009
(HHS Reference No. E–338–2008/0–
PCT–01).
Licensing Status: Available for
licensing.
Licensing Contact: Fatima Sayyid,
M.H.P.M.; 301–435–4521;
Fatima.Sayyid@nih.hhs.gov.
Collaborative Research Opportunity:
The National Cancer Institute,
Laboratory of Metabolism, is seeking
statements of capability or interest from
parties interested in collaborative
research to further develop, evaluate, or
commercialize HMGN and related
chromatin-binding proteins in the
function of pancreatic islet cells. Please
contact John Hewes, PhD at 301–435–
3121 or hewesj@mail.nih.gov for more
information.
Molecular Motors Powered by Proteins
Description of Technology: The
technology available for licensing and
commercial development relates to
molecular motors powered by proteins.
Some implementations describe a
molecular motor in which multiple
concentric cylinders or nested cones
rotate around a common longitudinal
axis. Opposing complementary surfaces
of the cylinders or cones are coated with
complementary motor protein pairs,
such as actin and myosin. The actin and
myosin interact with one another in the
presence of ATP to rotate the cylinders
or cones relative to one another, and
this rotational energy is harnessed to
produce work. Speed of movement is
controlled by the concentration of ATP
and the number of nested cylinders or
cones. The length of the cylinders or
cones can also be used to control the
power generated by the motor.
Another configuration forms the
motor out of a set of stacked disks,
much like CDs on a spindle. The
advantage of this form is extreme
simplicity of construction compared to
the nested cylinders or cones. In yet
another configuration, which has
aspects of both of the previous forms,
the surfaces are broken into annular
rings in order to overcome that the inner
surfaces rotate at a different rate than
the outer surfaces. This belt form may
ultimately be used in molecular
manufacturing.
Applications:
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4923
• Supplying power to prosthetic
implants and other medical devices
without external power sources.
• Many other applications that could
use a motor in other biotechnological
areas, in addition to the medical
applications.
• The inventions can be implemented
on either a microscopic or macroscopic
scale.
Development Status: Very early stage
of development.
Inventors: Thomas D. Schneider and
Ilya G. Lyakhov (NCI).
Relevant Publications: ‘‘Molecular
Motor’’, Patent Publication Nos. WO
2001/009181 A1, published 02/08/2001;
CA 2380611A1, published 02/08/2001;
AU 6616600A, published 02/19/2001;
EP 1204680A1, published 05/15/2002;
and U.S. 20020083710, published 07/
04/2002.
Patent Status:
• HHS Reference No. E–018–1999/
0—International Application Number
PCT/US 2000/20925 filed 31 Jul 2000;
granted Application AU 2002/18688 B2,
and the corresponding European and
Canadian applications being prosecuted,
all entitled ‘‘Molecular Motor’’
• HHS Reference No. E–018–1999/
1—U.S. Patent No. 7,349,834 issued 25
Mar 2008, and U.S. Patent Application
No. 12/011,239 filed 24 Jan 2008, both
entitled ‘‘Molecular Motor’’
Licensing Status: Available for
licensing.
Licensing Contact: Susan Ano, PhD;
301–435–5515; anos@mail.nih.gov.
Collaborative Research Opportunity:
The National Cancer Institute, Center for
Cancer Research Nanobiology Program
is seeking statements of capability or
interest from parties interested in
collaborative research to further
develop, evaluate, or commercialize the
Molecular Rotation Engine. Please
contact John D. Hewes, PhD at 301–435–
3121 or hewesj@mail.nih.gov for more
information.
Dated: January 19, 2011.
Richard U. Rodriguez,
Director, Division of Technology Development
and Transfer, Office of Technology Transfer,
National Institutes of Health.
[FR Doc. 2011–1671 Filed 1–26–11; 8:45 am]
BILLING CODE 4140–01–P
DEPARTMENT OF HEALTH AND
HUMAN SERVICES
National Institutes of Health
Center for Scientific Review; Notice of
Closed Meeting
Pursuant to section 10(d) of the
Federal Advisory Committee Act, as
E:\FR\FM\27JAN1.SGM
27JAN1
Agencies
[Federal Register Volume 76, Number 18 (Thursday, January 27, 2011)]
[Notices]
[Pages 4921-4923]
From the Federal Register Online via the Government Printing Office [www.gpo.gov]
[FR Doc No: 2011-1671]
-----------------------------------------------------------------------
DEPARTMENT OF HEALTH AND HUMAN SERVICES
National Institutes of Health
Government-Owned Inventions; Availability for Licensing
AGENCY: National Institutes of Health, Public Health Service, HHS.
ACTION: Notice.
-----------------------------------------------------------------------
[[Page 4922]]
SUMMARY: The inventions listed below are owned by an agency of the U.S.
Government and are available for licensing in the U.S. in accordance
with 35 U.S.C. 207 to achieve expeditious commercialization of results
of federally-funded research and development. Foreign patent
applications are filed on selected inventions to extend market coverage
for companies and may also be available for licensing.
ADDRESSES: Licensing information and copies of the U.S. patent
applications listed below may be obtained by writing to the indicated
licensing contact at the Office of Technology Transfer, National
Institutes of Health, 6011 Executive Boulevard, Suite 325, Rockville,
Maryland 20852-3804; telephone: 301/496-7057; fax: 301/402-0220. A
signed Confidential Disclosure Agreement will be required to receive
copies of the patent applications.
A Computer Program To Predict Optimal Sites on Protein Sequences for
Production of Peptide-Directed Antibodies (NHLBI AbDesigner)
Description of Technology: The invention offered for licensing is a
computer program called ``NHLBI AbDesigner'' that allows the user to
input a unique identifier for an individual mammalian protein to be
analyzed in order to find out what short peptides in its amino sequence
would most likely result in a strong immunogenic response when injected
into a research animal. The software displays standard predictors of
immunogenicity and antigenicity in easy-to-view heat maps and also
allows users to choose peptides most likely to elicit antibodies that
are specific to said protein. The computer code is written in Java and
would be made available in the form of jar files.
For additional information please refer to: https://dirweb.nhlbi.nih.gov/labs/LKEM_G/LKEM/Pages/Antibodydesignsoftware.aspx.
Applications:
Design and production of antibodies for research or
therapeutic purposes.
Bioinformatic analysis of protein structure and functions.
Analysis and interpretation of proteomic data.
Advantages: This program allows the user to identify tradeoffs in
the decision making process by aligning various types of information
with the amino acid sequence, constituting an improvement over present
ad hoc methods of accumulating and relating different type of
information regarding immunogenicity, uniqueness of sequences,
conservation of sequences, and presence of post-translational
modifications.
Development Status: Fully developed.
Inventors: Mark A. Knepper (NHLBI) et al.
Patent Status: HHS Reference No. E-251-2010/0--Software. Patent
protection is not being pursued for this technology.
Licensing Status: Available for licensing.
Licensing Contacts:
Uri Reichman, PhD, MBA; 301-435-4616; UR7a@nih.gov.
Michael Shmilovich, Esq.; 301-435-5019;
shmilovm@mail.nih.gov.
Collaborative Research Opportunity: The NHLBI is seeking statements
of capability or interest from parties interested in collaborative
research to further develop, evaluate, or commercialize this
technology. Please contact Brian Bailey, Ph.D. at 301-594-4094 or
bbailey@mail.nih.gov for more information.
Nanoparticle Probes and Mid-Infrared Chemical Imaging for DNA
Microarray Detection
Description of Technology: The technology offered for licensing is
a faster, more flexible, cost-effective microarray visualization. The
invention describes and claims the mid-infrared chemical imaging (IRCI)
to detect nanostructure-based DNA microarrays, which can be utilized in
the life science research arena to examine gene expression and single
nucleotide polymorphisms (SNPs), as well as to characterize entire
genomes. The IRCI improves the signal-to-noise ratio (SNR) obtained for
hybridized microarrayed spots compared to the commonly used
fluorescence detection method. The improved method of this invention
results in the sensitivity and precision for detecting pathogenic
bacterial genes and can be utilized to detect low-expressing genes
which cannot be identified by fluorescent-based DNA microarrays.
Furthermore, the automated IRCI systems can also be fabricated for the
dedicated detection of other (protein, tissue, biochemical, or
chemical) microarrays.
Applications: DNA microarrays can be applied to the areas of
environmental sciences, agriculture research, bio-defense, diagnostics,
forensics, pharmacogenomics and toxicogenomics.
Advantages: The invention provides a cost-effective, faster, more
flexible, and less labor intensive microarray technology.
Development Status:
The invention is fully developed.
Need to develop a commercialized kit and protocol.
Inventors: Magdi M. Mossoba, et al. (FDA).
Patent Status: U.S. Provisional Application No. 61/395,635 filed 15
Oct 2010 (HHS Reference No. E-127-2010/0-US-01).
Licensing Status: Available for licensing.
Licensing Contact: Susan Ano, PhD; 301-435-5515; anos@mail.nih.gov.
Fluoroquinolone Derivatives as Inhibitors of Human Tyrosyl-DNA
Phosphodiesterase (Tdp1)
Description of Technology: Chemotherapy can provide therapeutic
benefits in many cancer patients, but it often ultimately fails to cure
the disease since cancer cells can become resistant to the
chemotherapeutic agent. To overcome these limitations, additional
strategies are needed to restore or amplify the effect of antitumor
agents. Tyrosyl-DNA phosphodiesterase 1 (Tdp1) is a DNA repair enzyme
involved in the repair of DNA lesions created when the activity of the
Topoisomerase 1 (Top1) is inhibited. Tdp1 has been regarded as a
potential therapeutic co-target of Top1 in that it seemingly
counteracts the effects of Top1 inhibitors, such as camptothecin. By
reducing the repair of Top1-DNA lesions, Tdp1 inhibitors have the
potential to augment the anticancer activity of Top1 inhibitors.
The NIH investigators discovered fluoroquinolone derivatives as
specific Tdp1 inhibitors that could potentiate the pharmacological
action of Top1 inhibitors, which are currently used in cancer
treatment. The instant invention discloses a method of treating cancers
with a therapeutically effective amount of a Top1 inhibitor, and a
fluoroquinolone derivative that inhibits Tdp1 activity.
Applications and Market:
This invention may provide a new combination of drugs to
target various cancers for treatment.
Cancer is the second leading cause of death in the U.S.
The National Cancer Institute estimates the overall annual costs for
cancer in the U.S. at $107 billion; development of more effective
cancer therapies is always in high demand.
Development Status: Pre-clinical stage of development.
Inventors: Yves G. Pommier, Christophe R. Marchand, Thomas S.
Dexheimer (NCI), et al.
Related Publications:
1. Dexheimer TS, Antony S, Marchand C, Pommier Y. Tyrosyl-DNA
phosphodiesterase as a target for
[[Page 4923]]
anticancer therapy. Anticancer Agents Med Chem. 2008 May;8(4):381-389.
[PubMed: 18473723]
2. Dexheimer TS, et al. 4-Pregnen-21-ol-3,20-dione-21-(4-
bromobenzenesulfonate) and related novel steroid derivatives as
tyrosyl-DNA phosphodiesterase (Tdp1) inhibitors. J Med Chem. 2009 Nov
26;52(22):7122-7131. [PubMed: 19883083]
3. Marchand C, et al. Identification of phosphotyrosine mimetic
inhibitors of human tyrosyl-DNA phosphodiesterase I by a novel
AlphaScreen high-throughput assay. Mol Cancer Ther. 2009 Jan;8(1):240-
248. [PubMed: 19139134]
Patent Status: U.S. Provisional Application No. 61/407,325 filed 07
Oct 2010 (HHS Reference No. E-199-2010/0-US-01).
Licensing Status: Available for licensing.
Licensing Contact: Betty B. Tong, PhD; 301-594-6565;
tongb@mail.nih.gov.
Collaborative Research Opportunity: The Center for Cancer Research,
Laboratory of Molecular Pharmacology, is seeking statements of
capability or interest from parties interested in collaborative
research to further develop, evaluate, or commercialize tyrosyl-DNA-
phosphodiesterase inhibitors. Please contact John Hewes, PhD at 301-
435-3121 or hewesj@mail.nih.gov for more information.
HMG3 for Detecting and Treating Diabetes
Description of Technology: This invention relates to the use of
High Mobility Group N 3 (HMGN3) as a marker for detecting diabetes and
as a therapeutic agent for treating diabetes.
Diabetes is disabling largely because commonly available anti-
diabetic drugs do not adequately control blood sugar levels to
completely prevent the occurrence of high and low blood sugar levels.
Inappropriate blood sugar levels can be toxic and can cause long-term
complications including renopathy, retinopathy, neuropathy and
peripheral vascular disease. Those with diabetes are also at risk for
developing related conditions such as obesity, hypertension, heart
disease and hyperlipidemia.
This invention relates to the discovery that reduced expression of
HMGN3 (also called TRIP7) gives rise to elevated blood glucose levels,
reduced serum insulin levels and impaired glucose tolerance.
Applications: Diagnostic and therapeutic for diabetes.
Development Status: Early stage.
Inventors: Michael Bustin et al. (NCI).
Related Publication: Ueda T, Furusawa T, Kurahashi T, Tessarollo L,
Bustin M. The nucleosome binding protein HMGN3 modulates the
transcription profile of pancreatic beta cells and affects insulin
secretion. Mol Cell Biol. 2009 Oct;29(19):5264-5276. [PubMed: 19651901]
Patent Status: PCT Application No. PCT/US2009/039406 filed 03 Apr
2009 (HHS Reference No. E-338-2008/0-PCT-01).
Licensing Status: Available for licensing.
Licensing Contact: Fatima Sayyid, M.H.P.M.; 301-435-4521;
Fatima.Sayyid@nih.hhs.gov.
Collaborative Research Opportunity: The National Cancer Institute,
Laboratory of Metabolism, is seeking statements of capability or
interest from parties interested in collaborative research to further
develop, evaluate, or commercialize HMGN and related chromatin-binding
proteins in the function of pancreatic islet cells. Please contact John
Hewes, PhD at 301-435-3121 or hewesj@mail.nih.gov for more information.
Molecular Motors Powered by Proteins
Description of Technology: The technology available for licensing
and commercial development relates to molecular motors powered by
proteins. Some implementations describe a molecular motor in which
multiple concentric cylinders or nested cones rotate around a common
longitudinal axis. Opposing complementary surfaces of the cylinders or
cones are coated with complementary motor protein pairs, such as actin
and myosin. The actin and myosin interact with one another in the
presence of ATP to rotate the cylinders or cones relative to one
another, and this rotational energy is harnessed to produce work. Speed
of movement is controlled by the concentration of ATP and the number of
nested cylinders or cones. The length of the cylinders or cones can
also be used to control the power generated by the motor.
Another configuration forms the motor out of a set of stacked
disks, much like CDs on a spindle. The advantage of this form is
extreme simplicity of construction compared to the nested cylinders or
cones. In yet another configuration, which has aspects of both of the
previous forms, the surfaces are broken into annular rings in order to
overcome that the inner surfaces rotate at a different rate than the
outer surfaces. This belt form may ultimately be used in molecular
manufacturing.
Applications:
Supplying power to prosthetic implants and other medical
devices without external power sources.
Many other applications that could use a motor in other
biotechnological areas, in addition to the medical applications.
The inventions can be implemented on either a microscopic
or macroscopic scale.
Development Status: Very early stage of development.
Inventors: Thomas D. Schneider and Ilya G. Lyakhov (NCI).
Relevant Publications: ``Molecular Motor'', Patent Publication Nos.
WO 2001/009181 A1, published 02/08/2001; CA 2380611A1, published 02/08/
2001; AU 6616600A, published 02/19/2001; EP 1204680A1, published 05/15/
2002; and U.S. 20020083710, published 07/04/2002.
Patent Status:
HHS Reference No. E-018-1999/0--International Application
Number PCT/US 2000/20925 filed 31 Jul 2000; granted Application AU
2002/18688 B2, and the corresponding European and Canadian applications
being prosecuted, all entitled ``Molecular Motor''
HHS Reference No. E-018-1999/1--U.S. Patent No. 7,349,834
issued 25 Mar 2008, and U.S. Patent Application No. 12/011,239 filed 24
Jan 2008, both entitled ``Molecular Motor''
Licensing Status: Available for licensing.
Licensing Contact: Susan Ano, PhD; 301-435-5515; anos@mail.nih.gov.
Collaborative Research Opportunity: The National Cancer Institute,
Center for Cancer Research Nanobiology Program is seeking statements of
capability or interest from parties interested in collaborative
research to further develop, evaluate, or commercialize the Molecular
Rotation Engine. Please contact John D. Hewes, PhD at 301-435-3121 or
hewesj@mail.nih.gov for more information.
Dated: January 19, 2011.
Richard U. Rodriguez,
Director, Division of Technology Development and Transfer, Office of
Technology Transfer, National Institutes of Health.
[FR Doc. 2011-1671 Filed 1-26-11; 8:45 am]
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